A novel ATP-dependent inward rectifier potassium channel expressed predominantly in glial cells.
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Association analysis of chromosome 1 migraine candidate genesCloning and characterization of two K+ inward rectifier (Kir) 1.1 potassium channel homologs from human kidney (Kir1.2 and Kir1.3)Cloning and characterization of a novel human inwardly rectifying potassium channel predominantly expressed in small intestineSpecific localization of an inwardly rectifying K(+) channel, Kir4.1, at the apical membrane of rat gastric parietal cells; its possible involvement in K(+) recycling for the H(+)-K(+)-pumpExpression of a functional Kir4 family inward rectifier K+ channel from a gene cloned from mouse liverThe astrocyte odysseyEAST syndrome: Clinical, pathophysiological, and genetic aspects of mutations in KCNJ10The Role of MicroRNAs in the Regulation of K(+) Channels in Epithelial TissueActivity-Dependent Plasticity of Astroglial Potassium and Glutamate ClearanceNeuronal and Cardiovascular Potassium Channels as Therapeutic Drug Targets: Promise and PitfallsVascular inward rectifier K+ channels as external K+ sensors in the control of cerebral blood flowGain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy.Expression of an inwardly rectifying K(+) channel, Kir4.1, in satellite cells of rat cochlear gangliaGenetic and functional linkage of Kir5.1 and Kir2.1 channel subunitsClustering and enhanced activity of an inwardly rectifying potassium channel, Kir4.1, by an anchoring protein, PSD-95/SAP90Gene identification in 1.6-Mb region of the Down syndrome region on chromosome 21Functional Kir7.1 channels localized at the root of apical processes in rat retinal pigment epitheliumExpression and permeation properties of the K(+) channel Kir7.1 in the retinal pigment epitheliumControl of rectification and permeation by two distinct sites after the second transmembrane region in Kir2.1 K+ channelKCNJ10 gene mutations causing EAST syndrome (epilepsy, ataxia, sensorineural deafness, and tubulopathy) disrupt channel functionCommon variants of KCNJ10 are associated with susceptibility and anti-epileptic drug resistance in Chinese genetic generalized epilepsiesIn vivo formation of a proton-sensitive K+ channel by heteromeric subunit assembly of Kir5.1 with Kir4.1Kir4.1 expression by astrocytes and oligodendrocytes in CNS white matter: a developmental study in the rat optic nerveSilencing the Kir4.1 potassium channel subunit in satellite glial cells of the rat trigeminal ganglion results in pain-like behavior in the absence of nerve injuryGenetic inactivation of an inwardly rectifying potassium channel (Kir4.1 subunit) in mice: phenotypic impact in retinaThe role of an inwardly rectifying K(+) channel (Kir4.1) in the inner ear and hearing loss.K(+) channelepsy: progress in the neurobiology of potassium channels and epilepsy.Mapping a barbiturate withdrawal locus to a 0.44 Mb interval and analysis of a novel null mutant identify a role for Kcnj9 (GIRK3) in withdrawal from pentobarbital, zolpidem, and ethanol.A hyperprostaglandin E syndrome mutation in Kir1.1 (renal outer medullary potassium) channels reveals a crucial residue for channel function in Kir1.3 channels.A novel junction-like membrane complex in the optic nerve astrocyte of the Japanese macaque with a possible relation to a potassium ion channel.TbIRK is a signature sequence free potassium channel from Trypanosoma brucei locating to acidocalcisomes.Molecular diversity and regulation of renal potassium channels.Potassium buffering in the central nervous systemMolecular aspects of structure, gating, and physiology of pH-sensitive background K2P and Kir K+-transport channels.Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome.Ablation of Kcnj10 expression in retinal explants revealed pivotal roles for Kcnj10 in the proliferation and development of Müller glia.Potassium channel activity and glutamate uptake are impaired in astrocytes of seizure-susceptible DBA/2 mice.Genetic defects in the hotspot of inwardly rectifying K(+) (Kir) channels and their metabolic consequences: a reviewAstrocyte calcium signaling and epilepsy.The role of astroglia in the epileptic brain.
P2860
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P2860
A novel ATP-dependent inward rectifier potassium channel expressed predominantly in glial cells.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
A novel ATP-dependent inward r ...... predominantly in glial cells.
@en
A novel ATP-dependent inward r ...... predominantly in glial cells.
@nl
type
label
A novel ATP-dependent inward r ...... predominantly in glial cells.
@en
A novel ATP-dependent inward r ...... predominantly in glial cells.
@nl
prefLabel
A novel ATP-dependent inward r ...... predominantly in glial cells.
@en
A novel ATP-dependent inward r ...... predominantly in glial cells.
@nl
P2093
P2860
P356
P1476
A novel ATP-dependent inward r ...... d predominantly in glial cells
@en
P2093
K Morishige
N Takahashi
S Nakanishi
T Yamashita
P2860
P304
16339-16346
P356
10.1074/JBC.270.27.16339
P407
P577
1995-07-01T00:00:00Z