Intraneuronal beta-amyloid aggregates, neurodegeneration, and neuron loss in transgenic mice with five familial Alzheimer's disease mutations: potential factors in amyloid plaque formation.
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Orally administrated cinnamon extract reduces β-amyloid oligomerization and corrects cognitive impairment in Alzheimer's disease animal modelsRAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunctionSORLA-dependent and -independent functions for PACS1 in control of amyloidogenic processesAn epigenetic blockade of cognitive functions in the neurodegenerating brainNeural stem/progenitor cells in Alzheimer's diseaseOverview of Alzheimer's Disease and Some Therapeutic Approaches Targeting Aβ by Using Several Synthetic and Herbal CompoundsEpigenetic Alterations in Alzheimer's DiseaseConnectivity of Pathology: The Olfactory System as a Model for Network-Driven Mechanisms of Alzheimer's Disease PathogenesisAlzheimer's Disease: Mechanism and Approach to Cell TherapyNeuronal response in Alzheimer's and Parkinson's disease: the effect of toxic proteins on intracellular pathwaysβ-Amyloid: the key peptide in the pathogenesis of Alzheimer's diseaseAlzheimer's in 3D culture: challenges and perspectivesTDP-43: a new player on the AD field?The airbag problem-a potential culprit for bench-to-bedside translational efforts: relevance for Alzheimer's diseaseIs there a molecular logic that sustains neuronal functional integrity and survival? Lipid signaling is necessary for neuroprotective neuronal transcriptional programsIntraneuronal Aβ accumulation induces hippocampal neuron hyperexcitability through A-type K(+) current inhibition mediated by activation of caspases and GSK-3.Amyloid-Beta Induced Changes in Vesicular Transport of BDNF in Hippocampal NeuronsChronic adjunction of 1-deoxynojirimycin protects from age-related behavioral and biochemical changes in the SAMP8 mice.Abnormal cognition, sleep, EEG and brain metabolism in a novel knock-in Alzheimer mouse, PLB1IL-33 ameliorates Alzheimer's disease-like pathology and cognitive decline.HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer's disease.Transnasal delivery of human A-beta peptides elicits impaired learning and memory performance in wild type mice.Urine proteins identified by two-dimensional differential gel electrophoresis facilitate the differential diagnoses of scrapieMT5-MMP is a new pro-amyloidogenic proteinase that promotes amyloid pathology and cognitive decline in a transgenic mouse model of Alzheimer's disease.Endothelial LRP1 transports amyloid-β(1-42) across the blood-brain barrierA Redox-Active, Compact Molecule for Cross-Linking Amyloidogenic Peptides into Nontoxic, Off-Pathway Aggregates: In Vitro and In Vivo Efficacy and Molecular Mechanisms.Spines, plasticity, and cognition in Alzheimer's model miceAPP processing in Alzheimer's diseaseAPOE-modulated Aβ-induced neuroinflammation in Alzheimer's disease: current landscape, novel data, and future perspectiveAnalyzing dendritic spine pathology in Alzheimer's disease: problems and opportunitiesIn vivo imaging of microglial activation by positron emission tomography with [(11)C]PBR28 in the 5XFAD model of Alzheimer's diseaseLipid microdomain modification sustains neuronal viability in models of Alzheimer's diseaseApomorphine treatment in Alzheimer mice promoting amyloid-β degradationGalantamine slows down plaque formation and behavioral decline in the 5XFAD mouse model of Alzheimer's disease.Nanoscale Synaptic Membrane Mimetic Allows Unbiased High Throughput Screen That Targets Binding Sites for Alzheimer's-Associated Aβ Oligomers.Activity-dependent p25 generation regulates synaptic plasticity and Aβ-induced cognitive impairmentMechanisms underlying insulin deficiency-induced acceleration of β-amyloidosis in a mouse model of Alzheimer's diseaseStructure-mechanism-based engineering of chemical regulators targeting distinct pathological factors in Alzheimer's disease.Oral Triphenylmethane Food Dye Analog, Brilliant Blue G, Prevents Neuronal Loss in APPSwDI/NOS2-/- Mouse Model.Beneficial effects of a pyrroloquinolinequinone-containing dietary formulation on motor deficiency, cognitive decline and mitochondrial dysfunction in a mouse model of Alzheimer's disease.
P2860
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P2860
Intraneuronal beta-amyloid aggregates, neurodegeneration, and neuron loss in transgenic mice with five familial Alzheimer's disease mutations: potential factors in amyloid plaque formation.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Intraneuronal beta-amyloid agg ...... s in amyloid plaque formation.
@en
Intraneuronal beta-amyloid agg ...... s in amyloid plaque formation.
@nl
type
label
Intraneuronal beta-amyloid agg ...... s in amyloid plaque formation.
@en
Intraneuronal beta-amyloid agg ...... s in amyloid plaque formation.
@nl
prefLabel
Intraneuronal beta-amyloid agg ...... s in amyloid plaque formation.
@en
Intraneuronal beta-amyloid agg ...... s in amyloid plaque formation.
@nl
P2093
P1476
Intraneuronal beta-amyloid agg ...... rs in amyloid plaque formation
@en
P2093
Angela Guillozet-Bongaarts
Erika Maus
Holly Oakley
Jeffery Craft
Linda Van Eldik
Masuo Ohno
Robert Berry
Robert Vassar
Sarah L Cole
P304
10129-10140
P356
10.1523/JNEUROSCI.1202-06.2006
P407
P577
2006-10-01T00:00:00Z