RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunction - Wikidata - awgldk - TriplyDB

RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunction

RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunction

http://www.wikidata.org/entity/Q24315809

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Autonomic neuropathy in diabetes mellitus Receptor for advanced glycation end products and its involvement in inflammatory diseases The toxicity of amyloid β oligomers RAGE Expression and ROS Generation in Neurons: Differentiation versus Damage Amyloid-β Receptors: The Good, the Bad, and the Prion Protein Neuronal response in Alzheimer's and Parkinson's disease: the effect of toxic proteins on intracellular pathways Role of pro-inflammatory cytokines released from microglia in Alzheimer's disease Insight of brain degenerative protein modifications in the pathology of neurodegeneration and dementia by proteomic profiling Analyzing dendritic spine pathology in Alzheimer's disease: problems and opportunities A multimodal RAGE-specific inhibitor reduces amyloid β-mediated brain disorder in a mouse model of Alzheimer disease Geniposide Protects Primary Cortical Neurons against Oligomeric Aβ1-42-Induced Neurotoxicity through a Mitochondrial Pathway An aqueous orally active vaccine targeted against a RAGE/AB complex as a novel therapeutic for Alzheimer's disease Dolphin genome provides evidence for adaptive evolution of nervous system genes and a molecular rate slowdown Mitochondria-specific accumulation of amyloid β induces mitochondrial dysfunction leading to apoptotic cell death Microglial receptor for advanced glycation end product-dependent signal pathway drives beta-amyloid-induced synaptic depression and long-term depression impairment in entorhinal cortex.Models of membrane-bound Alzheimer's Abeta peptide assemblies The Role of Unfolded Protein Response and Mitogen-Activated Protein Kinase Signaling in Neurodegenerative Diseases with Special Focus on Prion Diseases Phagocytic clearance in neurodegeneration.Tunneling-nanotube direction determination in neurons and astrocytes Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans Neuronal p38α mediates synaptic and cognitive dysfunction in an Alzheimer's mouse model by controlling β-amyloid production.Unlocking the Door to Neuronal Woes in Alzheimer's Disease: Aβ and Mitochondrial Permeability Transition Pore The nuclear inclusion a (NIa) protease of turnip mosaic virus (TuMV) cleaves amyloid-β Association of the RAGE G82S polymorphism with Alzheimer's disease.S100B and APP promote a gliocentric shift and impaired neurogenesis in Down syndrome neural progenitors.Alzheimer's disease: diverse aspects of mitochondrial malfunctioning.Decreased proteolytic activity of the mitochondrial amyloid-β degrading enzyme, PreP peptidasome, in Alzheimer's disease brain mitochondria Early deficits in synaptic mitochondria in an Alzheimer's disease mouse model Pinocembrin protects against β-amyloid-induced toxicity in neurons through inhibiting receptor for advanced glycation end products (RAGE)-independent signaling pathways and regulating mitochondrion-mediated apoptosis.Cyclophilin D deficiency rescues axonal mitochondrial transport in Alzheimer's neurons Amyloid-β as a modulator of synaptic plasticity Role of amyloid peptides in vascular dysfunction and platelet dysregulation in Alzheimer's disease Identification of human presequence protease (hPreP) agonists for the treatment of Alzheimer's disease.Neurovascular defects and faulty amyloid-β vascular clearance in Alzheimer's disease Determination of small molecule ABAD inhibitors crossing blood-brain barrier and pharmacokinetics Mitochondrial dysfunction and accumulation of the β-secretase-cleaved C-terminal fragment of APP in Alzheimer's disease transgenic mice Involvement of Intracellular and Mitochondrial Aβ in the Ameliorative Effects of Huperzine A against Oligomeric Aβ42-Induced Injury in Primary Rat Neurons.IgG Conformer's Binding to Amyloidogenic Aggregates.A specific RAGE-binding peptide biopanning from phage display random peptide library that ameliorates symptoms in amyloid β peptide-mediated neuronal disorder.Genomics of Dementia: APOE- and CYP2D6-Related Pharmacogenetics.

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RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunction

http://www.wikidata.org/entity/Q24315809

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2009年の論文

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RAGE-mediated signaling contri ...... -beta and neuronal dysfunction

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RAGE-mediated signaling contri ...... -beta and neuronal dysfunction

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RAGE-mediated signaling contri ...... -beta and neuronal dysfunction

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Alexander Sosunov

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Daisuke Ibi

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David M Stern

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Emiko Fukuzaki

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Fang Fang

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Guy McKhann

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Heng Du

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Hiroyuki Mizoguchi

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Kazuhiro Takuma

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Kiyofumi Yamada

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P2860

Cloning and expression of a cell surface receptor for advanced glycosylation end products of proteins

Degradation of the amyloid beta-protein by the novel mitochondrial peptidasome, PreP

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

Endophilin I expression is increased in the brains of Alzheimer disease patients

Interaction of amyloid binding alcohol dehydrogenase/Abeta mediates up-regulation of peroxiredoxin II in the brains of Alzheimer's disease patients and a transgenic Alzheimer's disease mouse model

Regulation of NMDA receptor trafficking by amyloid-beta

Intracellular amyloid-beta in Alzheimer's disease

Altered intracellular signaling and reduced viability of Alzheimer's disease neuronal cybrids is reproduced by beta-amyloid peptide acting through receptor for advanced glycation end products (RAGE).

Evidence that neurones accumulating amyloid can undergo lysis to form amyloid plaques in Alzheimer's disease.

The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses.

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19901339

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Advanced glycosylation end product-specific receptor

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2785285

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