Maternal diabetes adversely affects AMP-activated protein kinase activity and cellular metabolism in murine oocytes. - Wikidata - awgldk - TriplyDB

Maternal diabetes adversely affects AMP-activated protein kinase activity and cellular metabolism in murine oocytes.

Maternal diabetes adversely affects AMP-activated protein kinase activity and cellular metabolism in murine oocytes.

http://www.wikidata.org/entity/Q48786022

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Maternal diabetes causes abnormal dynamic changes of endoplasmic reticulum during mouse oocyte maturation and early embryo development.Mitochondrial dysfunction and apoptosis in cumulus cells of type I diabetic mice Minireview: Metabolism of female reproduction: regulatory mechanisms and clinical implications Dietary sugar in healthy female primates perturbs oocyte maturation and in vitro preimplantation embryo development A requirement for fatty acid oxidation in the hormone-induced meiotic maturation of mouse oocytes.Diet-induced obesity model: abnormal oocytes and persistent growth abnormalities in the offspring.Fertility treatment in women with polycystic ovary syndrome: a decision analysis of different oral ovulation induction agents.Maternal insulin resistance causes oxidative stress and mitochondrial dysfunction in mouse oocytes.High fat diet induced developmental defects in the mouse: oocyte meiotic aneuploidy and fetal growth retardation/brain defects.Enriched environment-induced maternal weight loss reprograms metabolic gene expression in mouse offspring.Specific deletion of AMP-activated protein kinase (α1AMPK) in murine oocytes alters junctional protein expression and mitochondrial physiology Preimplantation exposure of mouse embryos to palmitic acid results in fetal growth restriction followed by catch-up growth in the offspring.Metabolic control of oocyte development: linking maternal nutrition and reproductive outcomes AMPK: a master energy regulator for gonadal function.Live imaging reveals the link between decreased glucose uptake in ovarian cumulus cells and impaired oocyte quality in female diabetic mice Slc2a8 deficiency in mice results in reproductive and growth impairments Polycystic ovary syndrome and maternal obesity affect oocyte size in in vitro fertilization/intracytoplasmic sperm injection cycles Crosstalk between the AMP-activated kinase and insulin signaling pathways rescues murine blastocyst cells from insulin resistance.Maternal obesity, infertility and mitochondrial dysfunction: potential mechanisms emerging from mouse model systems.Current knowledge of obesity's effects in the pre- and periconceptional periods and avenues for future research Decreased oocyte-granulosa cell gap junction communication and connexin expression in a type 1 diabetic mouse model Maternal diabetes and oocyte quality A differential autophagic response to hyperglycemia in the developing murine embryo.DHEA-mediated inhibition of the pentose phosphate pathway alters oocyte lipid metabolism in mice.Maternal diabetes causes mitochondrial dysfunction and meiotic defects in murine oocytes Glucose transporters in gametes and preimplantation embryos.Fatty acid oxidation and meiotic resumption in mouse oocytes Regulation of the G2/M transition in rodent oocytes The effect of peri-conception hyperglycaemia and the involvement of the hexosamine biosynthesis pathway in mediating oocyte and embryo developmental competence.Dicarbonyl stress and glyoxalases in ovarian function.Maternal diabetes mellitus and the origin of non-communicable diseases in offspring: the role of epigenetics.Commonly used fertility drugs, a diet supplement, and stress force AMPK-dependent block of stemness and development in cultured mammalian embryos.Acetyl CoA carboxylase inactivation and meiotic maturation in mouse oocytes.Sirt3-dependent deacetylation of SOD2 plays a protective role against oxidative stress in oocytes from diabetic mice.Treatment with AICAR inhibits blastocyst development, trophectoderm differentiation and tight junction formation and function in mice.CoQ10 increases mitochondrial mass and polarization, ATP and Oct4 potency levels, and bovine oocyte MII during IVM while decreasing AMPK activity and oocyte death.Maternal diabetes causes alterations of DNA methylation statuses of some imprinted genes in murine oocytes.The effect of leptin on maturing porcine oocytes is dependent on glucose concentration.Re: Hadh2 and 3-hydroxyacyl-CoA dehydrogenase.Why AMPK agonists not known to be stressors may surprisingly contribute to miscarriage or hinder IVF/ART.

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P2860

Maternal diabetes adversely affects AMP-activated protein kinase activity and cellular metabolism in murine oocytes.

http://www.wikidata.org/entity/Q48786022

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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name

Maternal diabetes adversely af ...... metabolism in murine oocytes.

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Maternal diabetes adversely af ...... metabolism in murine oocytes.

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Maternal diabetes adversely af ...... metabolism in murine oocytes.

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Maternal diabetes adversely af ...... metabolism in murine oocytes.

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Maternal diabetes adversely af ...... metabolism in murine oocytes.

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AJPENDO.00097.2007

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American Journal of Physiology - Endocrinology and Metabolism

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Maternal diabetes adversely af ...... metabolism in murine oocytes.

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Aimee S Chang

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Ann M Ratchford

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Kelle H Moley

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Maggie M-Y Chi

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Rachael Sheridan

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P2860

Role of AMP-activated protein kinase in the regulation by glucose of islet beta cell gene expression

Biochemistry and molecular cell biology of diabetic complications

The AMP-activated protein kinase cascade--a unifying system for energy control

Glycogen debranching enzyme association with beta-subunit regulates AMP-activated protein kinase activity

Characterization of the role of gamma2 R531G mutation in AMP-activated protein kinase in cardiac hypertrophy and Wolff-Parkinson-White syndrome.

Diabetes and preimplantation events of embryogenesis.

A novel domain in AMP-activated protein kinase causes glycogen storage bodies similar to those seen in hereditary cardiac arrhythmias.

AMPK beta subunit targets metabolic stress sensing to glycogen.

Effects of in-vivo and in-vitro environments on the metabolism of the cumulus-oocyte complex and its influence on oocyte developmental capacity.

The pattern of energy metabolism in the mouse oöcyte and zygote

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10.1152/AJPENDO.00097.2007

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