N-methyl-D-aspartate-sensitive glutamate receptors induce calcium-mediated arachidonic acid release in primary cultures of cerebellar granule cells.
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Cyclothiazide selectively inhibits mGluR1 receptors interacting with a common allosteric site for non-competitive antagonistsMolecular aspects of glutamate dysregulation: implications for schizophrenia and its treatment.Cytosolic phospholipase A2 alpha inhibition prevents neuronal NMDA receptor-stimulated arachidonic acid mobilization and prostaglandin production but not subsequent cell death.Non-Coding RNAs as Potential Neuroprotectants against Ischemic Brain Injury.Antiinflammatory and neuroprotective actions of COX2 inhibitors in the injured brain.Role of Mitochondria in Neonatal Hypoxic-Ischemic Brain InjuryKynurenines, neurodegeneration and Alzheimer's disease.Molecular pathways of pannexin1-mediated neurotoxicity.4-Hydroxy-2-nonenal upregulates and phosphorylates cytosolic phospholipase A(2) in cultured Ra2 microglial cells via MAPK pathways.Breakdown of membrane phospholipids in Alzheimer disease. Involvement of excitatory amino acid receptors.Cyclooxygenase-2 inhibition protects cultured cerebellar granule neurons from glutamate-mediated cell death.Receptor-stimulated phospholipase A(2) liberates arachidonic acid and regulates neuronal excitability through protein kinase C.Protective effect of phenolic compounds isolated from the hooks and stems of Uncaria sinensis on glutamate-induced neuronal death.Potassium-evoked glutamate release liberates arachidonic acid from cortical neurons.Chronic lithium administration to rats selectively modifies 5-HT2A/2C receptor-mediated brain signaling via arachidonic acid.Chronic N-methyl-D-aspartate administration increases the turnover of arachidonic acid within brain phospholipids of the unanesthetized rat.Ca2+-dependent generation of mitochondrial reactive oxygen species serves as a signal for poly(ADP-ribose) polymerase-1 activation during glutamate excitotoxicity.Extract prepared from the bark of Cinnamomum cassia Blume prevents glutamate-induced neuronal death in cultured cerebellar granule cells.Evaluation of the protective effects of alkaloids isolated from the hooks and stems of Uncaria sinensis on glutamate-induced neuronal death in cultured cerebellar granule cells from rats.Chronic NMDA administration to rats up-regulates frontal cortex cytosolic phospholipase A2 and its transcription factor, activator protein-2.Production of platelet-activating factor by neuronal cells in the rat brain with cold injury.Mitochondrial dysfunction and lipid peroxidation in rat frontal cortex by chronic NMDA administration can be partially prevented by lithium treatment.Vasoactive intestinal peptide (VIP) and pituitary adenylate cyclase-activating polypeptide (PACAP) potentiate the glutamate-evoked release of arachidonic acid from mouse cortical neurons. Evidence for a cAMP-independent mechanism.Arachidonic Acid Inhibits a Purified and Reconstituted Glutamate Transporter Directly from the Water Phase and Not via the Phospholipid Membrane
P2860
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P2860
N-methyl-D-aspartate-sensitive glutamate receptors induce calcium-mediated arachidonic acid release in primary cultures of cerebellar granule cells.
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年学术文章
@wuu
1990年学术文章
@zh
1990年学术文章
@zh-cn
1990年学术文章
@zh-hans
1990年学术文章
@zh-my
1990年学术文章
@zh-sg
1990年學術文章
@yue
1990年學術文章
@zh-hant
name
N-methyl-D-aspartate-sensitive ...... s of cerebellar granule cells.
@en
N-methyl-D-aspartate-sensitive ...... s of cerebellar granule cells.
@nl
type
label
N-methyl-D-aspartate-sensitive ...... s of cerebellar granule cells.
@en
N-methyl-D-aspartate-sensitive ...... s of cerebellar granule cells.
@nl
prefLabel
N-methyl-D-aspartate-sensitive ...... s of cerebellar granule cells.
@en
N-methyl-D-aspartate-sensitive ...... s of cerebellar granule cells.
@nl
P2093
P2860
P1476
N-methyl-D-aspartate-sensitive ...... s of cerebellar granule cells.
@en
P2093
J T Wroblewski
J W Lazarewicz
P2860
P304
P356
10.1111/J.1471-4159.1990.TB05771.X
P407
P577
1990-12-01T00:00:00Z