Hypoxia induces NO-dependent release of heparan sulfate in fibroblasts from the Alzheimer mouse Tg2576 by activation of nitrite reduction.

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Hypoxia induces NO-dependent release of heparan sulfate in fibroblasts from the Alzheimer mouse Tg2576 by activation of nitrite reduction.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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Hypoxia induces NO-dependent r ...... tivation of nitrite reduction.

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Hypoxia induces NO-dependent r ...... tivation of nitrite reduction.

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Item

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Hypoxia induces NO-dependent r ...... tivation of nitrite reduction.

@en

Hypoxia induces NO-dependent r ...... tivation of nitrite reduction.

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Hypoxia induces NO-dependent r ...... tivation of nitrite reduction.

@en

Hypoxia induces NO-dependent r ...... tivation of nitrite reduction.

@nl

P1476

Hypoxia induces NO-dependent r ...... ctivation of nitrite reduction

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P2093

Erika Bourseau-Guilmain

http://www.w3.org/2001/XMLSchema#string

Fang Cheng

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Katrin Mani

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Lars-Åke Fransson

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P2860

Prion, amyloid beta-derived Cu(II) ions, or free Zn(II) ions support S-nitroso-dependent autocleavage of glypican-1 heparan sulfate

Crystal Structure of N -Glycosylated Human Glypican-1 Core Protein

Copper-dependent autocleavage of glypican-1 heparan sulfate by nitric oxide derived from intrinsic nitrosothiols

Modulations of glypican-1 heparan sulfate structure by inhibition of endogenous polyamine synthesis. Mapping of spermine-binding sites and heparanase, heparin lyase, and nitric oxide/nitrite cleavage sites.

Nitric oxide-dependent processing of heparan sulfate in recycling S-nitrosylated glypican-1 takes place in caveolin-1-containing endosomes.

The amyloid precursor protein (APP) of Alzheimer disease and its paralog, APLP2, modulate the Cu/Zn-Nitric Oxide-catalyzed degradation of glypican-1 heparan sulfate in vivo.

Novel aspects of glypican glycobiology.

Nitrosothiol formation and protection against Fenton chemistry by nitric oxide-induced dinitrosyliron complex formation from anoxia-initiated cellular chelatable iron increase

Amyloid precursor protein (APP)/APP-like protein 2 (APLP2) expression is required to initiate endosome-nucleus-autophagosome trafficking of glypican-1-derived heparan sulfate

Biochemistry of nitric oxide and its redox-activated forms.

P304

623-634

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P31

scholarly article

P356

10.1093/GLYCOB/CWW007

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P433

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P478

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P50

Mattias Belting

P577

2016-01-20T00:00:00Z

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P698

26791445

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