Rac is a dominant regulator of cadherin-directed actin assembly that is activated by adhesive ligation independently of Tiam1.
about
The Tiam1 PDZ domain couples to Syndecan1 and promotes cell-matrix adhesionComparative analysis of mRNA targets for human PUF-family proteins suggests extensive interaction with the miRNA regulatory systemDistinct PTPmu-associated signaling molecules differentially regulate neurite outgrowth on E-, N-, and R-cadherinGuidance of Signaling Activations by Cadherins and Integrins in Epithelial Ovarian Cancer CellsJunctional actin assembly is mediated by Formin-like 2 downstream of Rac1.Neogenin recruitment of the WAVE regulatory complex maintains adherens junction stability and tension.Chemical genomic-based pathway analyses for epidermal growth factor-mediated signaling in migrating cancer cellsLoss of Par3 promotes breast cancer metastasis by compromising cell-cell cohesion.The serine/threonine kinase Par1b regulates epithelial lumen polarity via IRSp53-mediated cell-ECM signaling.Dynamic assessment of fibroblast mechanical activity during Rac-induced cell spreading in 3-D culture.Immediate-early signaling induced by E-cadherin engagement and adhesion.Rac1 and Rho contribute to the migratory and invasive phenotype associated with somatic E-cadherin mutation.The role of reactive oxygen species in microvascular remodeling.Polarization of migrating cortical neurons by Rap1 and N-cadherin: Revisiting the model for the Reelin signaling pathway.Crossroads of integrins and cadherins in epithelia and stroma remodeling.A WAVE2-Arp2/3 actin nucleator apparatus supports junctional tension at the epithelial zonula adherens.Small GTPase protein Rac-1 is activated with maturation and regulates cell morphology and function in chondrocytesTissue organization by cadherin adhesion molecules: dynamic molecular and cellular mechanisms of morphogenetic regulationKIF17 regulates RhoA-dependent actin remodeling at epithelial cell-cell adhesions.Mechanical stretch decreases migration of alveolar epithelial cells through mechanisms involving Rac1 and Tiam1.Cortactin is a functional target of E-cadherin-activated Src family kinases in MCF7 epithelial monolayersPhosphoinositide 3-kinases in cell migration.Cortical F-actin stabilization generates apical-lateral patterns of junctional contractility that integrate cells into epithelia.THSD1 preserves vascular integrity and protects against intraplaque haemorrhaging in ApoE-/- mice.
P2860
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P2860
Rac is a dominant regulator of cadherin-directed actin assembly that is activated by adhesive ligation independently of Tiam1.
description
2006 nî lūn-bûn
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name
Rac is a dominant regulator of ...... gation independently of Tiam1.
@en
Rac is a dominant regulator of ...... gation independently of Tiam1.
@nl
type
label
Rac is a dominant regulator of ...... gation independently of Tiam1.
@en
Rac is a dominant regulator of ...... gation independently of Tiam1.
@nl
prefLabel
Rac is a dominant regulator of ...... gation independently of Tiam1.
@en
Rac is a dominant regulator of ...... gation independently of Tiam1.
@nl
P2093
P2860
P1476
Rac is a dominant regulator of ...... gation independently of Tiam1.
@en
P2093
Astrid Kraemer
Marita Goodwin
Radiya G Ali
Suzie Verma
P2860
P304
P356
10.1152/AJPCELL.00073.2006
P50
P577
2006-10-04T00:00:00Z