Regulation of NMDA receptor trafficking and function by striatal-enriched tyrosine phosphatase (STEP).
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Abeta-mediated NMDA receptor endocytosis in Alzheimer's disease involves ubiquitination of the tyrosine phosphatase STEP61Protein phosphatases and Alzheimer's diseaseRole of Striatal-Enriched Tyrosine Phosphatase in Neuronal FunctionGenetic reduction of striatal-enriched tyrosine phosphatase (STEP) reverses cognitive and cellular deficits in an Alzheimer's disease mouse modelStriatal-enriched protein tyrosine phosphatase in Alzheimer's disease.Role of NMDA Receptor-Mediated Glutamatergic Signaling in Chronic and Acute NeuropathologiesStriatal-enriched protein tyrosine phosphatase expression and activity in Huntington's disease: a STEP in the resistance to excitotoxicityCalpain and STriatal-Enriched protein tyrosine phosphatase (STEP) activation contribute to extrasynaptic NMDA receptor localization in a Huntington's disease mouse modelStriatal-enriched protein tyrosine phosphatase-STEPs toward understanding chronic stress-induced activation of corticotrophin releasing factor neurons in the rat bed nucleus of the stria terminalis.Inhibition of striatal-enriched tyrosine phosphatase 61 in the dorsomedial striatum is sufficient to increased ethanol consumptionRegulation of tyrosine phosphatase STEP61 by protein kinase A during motor skill learning in miceSegregation and crosstalk of D1 receptor-mediated activation of ERK in striatal medium spiny neurons upon acute administration of psychostimulantsA substrate trapping mutant form of striatal-enriched protein tyrosine phosphatase prevents amphetamine-induced stereotypies and long-term potentiation in the striatumActivation of dopamine D1 receptors blocks phencyclidine-induced neurotoxicity by enhancing N-methyl-D-aspartate receptor-mediated synaptic strengthBlockade of lysosomal acid ceramidase induces GluN2B-dependent Tau phosphorylation in rat hippocampal slices.Therapeutic implications for striatal-enriched protein tyrosine phosphatase (STEP) in neuropsychiatric disorders.The NMDA receptor as a target for cognitive enhancementNR2B-NMDA receptor-mediated increases in intracellular Ca2+ concentration regulate the tyrosine phosphatase, STEP, and ERK MAP kinase signalingSTEP regulation of seizure thresholds in the hippocampus.Striatal-enriched protein tyrosine phosphatase (STEP) knockout mice have enhanced hippocampal memory.STEP levels are unchanged in pre-frontal cortex and associative striatum in post-mortem human brain samples from subjects with schizophrenia, bipolar disorder and major depressive disorderDopaminergic modulation of synaptic transmission in cortex and striatumSTEP signaling pathway mediates psychomotor stimulation and morphine withdrawal symptoms, but not for reward, analgesia and toleranceIn vitro stretch injury induces time- and severity-dependent alterations of STEP phosphorylation and proteolysis in neurons.Glutamate system, amyloid ß peptides and tau protein: functional interrelationships and relevance to Alzheimer disease pathologySeizure-Induced Regulations of Amyloid-β, STEP61, and STEP61 Substrates Involved in Hippocampal Synaptic Plasticity.The tyrosine phosphatase STEP mediates AMPA receptor endocytosis after metabotropic glutamate receptor stimulation.PSD-95 stabilizes NMDA receptors by inducing the degradation of STEP61.Short and long access to cocaine self-administration activates tyrosine phosphatase STEP and attenuates GluN expression but differentially regulates GluA expression in the prefrontal cortex.Knockout of striatal enriched protein tyrosine phosphatase in mice results in increased ERK1/2 phosphorylation.Expression and function of striatal enriched protein tyrosine phosphatase is profoundly altered in cerebral ischemia.STEP activation by Gαq coupled GPCRs opposes Src regulation of NMDA receptors containing the GluN2A subunit.Taking STEPs forward to understand fragile X syndrome.A STEP forward in neural function and degeneration.The Role of Striatal-Enriched Protein Tyrosine Phosphatase (STEP) in Cognition.Hypothesis review: are clathrin-mediated endocytosis and clathrin-dependent membrane and protein trafficking core pathophysiological processes in schizophrenia and bipolar disorder?Cocaine self-administration causes signaling deficits in corticostriatal circuitry that are reversed by BDNF in early withdrawal.Developmental localization of NMDA receptors, Src and MAP kinases in mouse brain.Intra-prelimbic cortical inhibition of striatal-enriched tyrosine phosphatase suppresses cocaine seeking in rats.Glutamatergic neurotransmission in the prefrontal cortex mediates the suppressive effect of intra-prelimbic cortical infusion of BDNF on cocaine-seeking.
P2860
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P2860
Regulation of NMDA receptor trafficking and function by striatal-enriched tyrosine phosphatase (STEP).
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
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2006年學術文章
@zh-hant
name
Regulation of NMDA receptor tr ...... d tyrosine phosphatase (STEP).
@en
Regulation of NMDA receptor tr ...... -enriched tyrosine phosphatase
@nl
type
label
Regulation of NMDA receptor tr ...... d tyrosine phosphatase (STEP).
@en
Regulation of NMDA receptor tr ...... -enriched tyrosine phosphatase
@nl
prefLabel
Regulation of NMDA receptor tr ...... d tyrosine phosphatase (STEP).
@en
Regulation of NMDA receptor tr ...... -enriched tyrosine phosphatase
@nl
P2093
P1476
Regulation of NMDA receptor tr ...... d tyrosine phosphatase (STEP).
@en
P2093
Adrian Nava
Brett Masterson
Donna Oksenberg
John Leung
Karoly Nikolich
Michael Adkisson
Roman Urfer
Steven P Braithwaite
P304
P356
10.1111/J.1460-9568.2006.04837.X
P407
P577
2006-06-01T00:00:00Z