Apoptosis in murine calvarial bone and suture development.
about
Intracellular retention, degradation, and signaling of glycosylation-deficient FGFR2 and craniosynostosis syndrome-associated FGFR2C278FFibroblast growth factor signaling in skeletal development and diseaseTgf-beta1, Tgf-beta2, Tgf-beta3 and Msx2 expression is elevated during frontonasal suture morphogenesis and during active postnatal facial growthOverexpression of Nell-1, a craniosynostosis-associated gene, induces apoptosis in osteoblasts during craniofacial developmentEarly onset of craniosynostosis in an Apert mouse model reveals critical features of this pathologyYY1 activates Msx2 gene independent of bone morphogenetic protein signaling.Therapeutic effect of nanogel-based delivery of soluble FGFR2 with S252W mutation on craniosynostosis.The BMP ligand Gdf6 prevents differentiation of coronal suture mesenchyme in early cranial development.Integration of multiple signaling regulates through apoptosis the differential osteogenic potential of neural crest-derived and mesoderm-derived OsteoblastsCytokine therapy for craniosynostosis.Molecular and cellular mechanisms of the anabolic effect of intermittent PTHSignaling by fibroblast growth factors (FGF) and fibroblast growth factor receptor 2 (FGFR2)-activating mutations blocks mineralization and induces apoptosis in osteoblasts.Integration of multiple signaling pathways determines differences in the osteogenic potential and tissue regeneration of neural crest-derived and mesoderm-derived calvarial bonesAugmentation of Smad-dependent BMP signaling in neural crest cells causes craniosynostosis in mice.Mesodermal expression of Fgfr2S252W is necessary and sufficient to induce craniosynostosis in a mouse model of Apert syndrome.Intermittent PTH stimulates periosteal bone formation by actions on post-mitotic preosteoblastsEphA4 as an effector of Twist1 in the guidance of osteogenic precursor cells during calvarial bone growth and in craniosynostosisThe role of NELL-1, a growth factor associated with craniosynostosis, in promoting bone regeneration.Potential role of PC-1 expression and pyrophosphate elaboration in the molecular etiology of the FGFR-associated craniosynostosis syndromes.BMP-4 induces a Smad-dependent apoptotic cell death of mouse embryonic stem cell-derived neural precursors.Sclerostin is a novel secreted osteoclast-derived bone morphogenetic protein antagonist with unique ligand specificity.Increased osteoblast apoptosis in apert craniosynostosis: role of protein kinase C and interleukin-1.Loss-of-Function of Gli3 in Mice Causes Abnormal Frontal Bone Morphology and Premature Synostosis of the Interfrontal Suture.Twist is required for establishment of the mouse coronal suture.Expression of stromelysin-1 (MMP-3), gelatinase B (MMP-9), and plasminogen activator system during fetal calvarial development.Cell mixing at a neural crest-mesoderm boundary and deficient ephrin-Eph signaling in the pathogenesis of craniosynostosis.
P2860
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P2860
Apoptosis in murine calvarial bone and suture development.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Apoptosis in murine calvarial bone and suture development.
@en
Apoptosis in murine calvarial bone and suture development.
@nl
type
label
Apoptosis in murine calvarial bone and suture development.
@en
Apoptosis in murine calvarial bone and suture development.
@nl
prefLabel
Apoptosis in murine calvarial bone and suture development.
@en
Apoptosis in murine calvarial bone and suture development.
@nl
P2093
P1476
Apoptosis in murine calvarial bone and suture development.
@en
P2093
P304
P356
10.1046/J.0909-8836.1999.EOS107406.X
P577
1999-08-01T00:00:00Z