NKCC2 surface expression in mammalian cells: down-regulation by novel interaction with aldolase B. - Wikidata - awgldk - TriplyDB

NKCC2 surface expression in mammalian cells: down-regulation by novel interaction with aldolase B.

NKCC2 surface expression in mammalian cells: down-regulation by novel interaction with aldolase B.

http://www.wikidata.org/entity/Q52582023

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Molecular regulation of NKCC2 in the thick ascending limb Thick ascending limb: the Na(+):K (+):2Cl (-) co-transporter, NKCC2, and the calcium-sensing receptor, CaSR Dynamin2, clathrin, and lipid rafts mediate endocytosis of the apical Na/K/2Cl cotransporter NKCC2 in thick ascending limbs.Secretory carrier membrane protein 2 regulates exocytic insertion of NKCC2 into the cell membrane.Molecular cloning and functional characterization of zebrafish Slc4a3/Ae3 anion exchanger High-throughput fluorescent-based NKCC functional assay in adherent epithelial cells.Rare mutations in the human Na-K-Cl cotransporter (NKCC2) associated with lower blood pressure exhibit impaired processing and transport function.High salt differentially regulates surface NKCC2 expression in thick ascending limbs of Dahl salt-sensitive and salt-resistant rats Functional expression of human NKCC1 from a synthetic cassette-based cDNA: introduction of extracellular epitope tags and removal of cysteines.OS9 Protein Interacts with Na-K-2Cl Co-transporter (NKCC2) and Targets Its Immature Form for the Endoplasmic Reticulum-associated Degradation Pathway.Impact of Hybrid and Complex N-Glycans on Cell Surface Targeting of the Endogenous Chloride Cotransporter Slc12a2.Functional expression of the Na-K-2Cl cotransporter NKCC2 in mammalian cells fails to confirm the dominant-negative effect of the AF splice variant.Na+-K+-2Cl- cotransporter type 2 trafficking and activity: the role of interacting proteins.Multiple evolutionarily conserved Di-leucine like motifs in the carboxyl terminus control the anterograde trafficking of NKCC2.Plasma Membrane Targeting of Endogenous NKCC2 in COS7 Cells Bypasses Functional Golgi Cisternae and Complex N-Glycosylation.SORLA/SORL1 functionally interacts with SPAK to control renal activation of Na(+)-K(+)-Cl(-) cotransporter 2.A highly conserved motif at the COOH terminus dictates endoplasmic reticulum exit and cell surface expression of NKCC2.Aldolase sequesters WASP and affects WASP/Arp2/3-stimulated actin dynamics.Identification of moesin as NKCC2-interacting protein and analysis of its functional role in the NKCC2 apical trafficking.

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P2860

NKCC2 surface expression in mammalian cells: down-regulation by novel interaction with aldolase B.

http://www.wikidata.org/entity/Q52582023

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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name

NKCC2 surface expression in ma ...... l interaction with aldolase B.

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NKCC2 surface expression in ma ...... l interaction with aldolase B.

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type

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NKCC2 surface expression in ma ...... l interaction with aldolase B.

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NKCC2 surface expression in ma ...... l interaction with aldolase B.

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prefLabel

NKCC2 surface expression in ma ...... l interaction with aldolase B.

@en

NKCC2 surface expression in ma ...... l interaction with aldolase B.

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NKCC2 surface expression in ma ...... el interaction with aldolase B

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Boubacar Benziane

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Christophe Klein

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Michel Paillard

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Nadia Defontaine

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Nancy Zaarour

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Pascal Houillier

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Soline Bourgeois

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Sylvie Demaretz

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P2860

Cloning and functional characterization of a cation-Cl- cotransporter-interacting protein

Interaction between aldolase and vacuolar H+-ATPase: evidence for direct coupling of glycolysis to the ATP-hydrolyzing proton pump

A Golgi-associated PDZ domain protein modulates cystic fibrosis transmembrane regulator plasma membrane expression

Primary structure, functional expression, and chromosomal localization of the bumetanide-sensitive Na-K-Cl cotransporter in human colon

A novel site for ubiquitination: the N-terminal residue, and not internal lysines of MyoD, is essential for conjugation and degradation of the protein.

An experimental renal acidification defect in patients with hereditary fructose intolerance. I. Its resemblance to renal tubular acidosis

Accessory protein facilitated CFTR-CFTR interaction, a molecular mechanism to potentiate the chloride channel activity

Phospholipase D2 directly interacts with aldolase via Its PH domain

Characterization of the interaction of the stress kinase SPAK with the Na+-K+-2Cl- cotransporter in the nervous system: evidence for a scaffolding role of the kinase

Functional properties of the apical Na+-K+-2Cl- cotransporter isoforms

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33817-33830

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scholarly article

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10.1074/JBC.M700195200

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46

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282

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2007-09-11T00:00:00Z

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17848580

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