TIRAP (MAL) S180L polymorphism is a common protective factor against developing tuberculosis and systemic lupus erythematosus.
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Structural Insights into TIR Domain Specificity of the Bridging Adaptor Mal in TLR4 SignalingPolymorphic variation in TIRAP is not associated with susceptibility to childhood TB but may determine susceptibility to TBM in some ethnic groupsIn mice, tuberculosis progression is associated with intensive inflammatory response and the accumulation of Gr-1 cells in the lungs.Genetic variants in the TIRAP gene are associated with increased risk of sepsis-associated acute lung injuryShared pathways to infectious disease susceptibility?High-mobility group box 1 represents a potential marker of disease activity and novel therapeutic target in systemic lupus erythematosus.Genetic associations in type I interferon related pathways with autoimmunityIdentification of 34 novel proinflammatory proteins in a genome-wide macrophage functional screenCandidate gene identification for systemic lupus erythematosus using network centrality measures and gene ontology.Toll-like receptor polymorphisms and tuberculosis susceptibility: A comprehensive meta-analysis.Ecogenomics of respiratory diseases of public health significanceToll-like receptors and human disease: lessons from single nucleotide polymorphisms.A Common Variant in the Adaptor Mal Regulates Interferon Gamma Signaling.Toll-like receptors as therapeutic targets for autoimmune connective tissue diseasesInduction of inflammatory and immune responses by HMGB1-nucleosome complexes: implications for the pathogenesis of SLEToll-like receptor polymorphisms in malaria-endemic populations.MyD88 adapter-like (Mal)/TIRAP interaction with TRAF6 is critical for TLR2- and TLR4-mediated NF-kappaB proinflammatory responses.Toll-like receptors and NOD-like receptors in rheumatic diseases.Modulation of toll-like receptor function has therapeutic potential in autoimmune disease.Pattern recognition receptors in immune disorders affecting the skin.Mal, more than a bridge to MyD88.Toll-Like Receptor Pathways in Autoimmune Diseases.Association of TLR1, TLR2, TLR4, TLR6, and TIRAP polymorphisms with disease susceptibility.A critical role for the TLR signaling adapter Mal in alveolar macrophage-mediated protection against Bordetella pertussis.Healthy first-degree relatives of patients with type 1 diabetes exhibit significant differences in basal gene expression pattern of immunocompetent cells compared to controls: expression pattern as predeterminant of autoimmune diabetes.Evaluation of Toll-like receptor and adaptor molecule polymorphisms for susceptibility to tuberculosis in a Colombian population.Bioinformatics analysis of differentially expressed gene profiles associated with systemic lupus erythematosus.Application of Various Statistical Models to Explore Gene-Gene Interactions in Folate, Xenobiotic, Toll-Like Receptor and STAT4 Pathways that Modulate Susceptibility to Systemic Lupus Erythematosus.The Emerging Role of Pattern Recognition Receptors in the Pathogenesis of Malaria.
P2860
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P2860
TIRAP (MAL) S180L polymorphism is a common protective factor against developing tuberculosis and systemic lupus erythematosus.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
TIRAP (MAL) S180L polymorphism ...... systemic lupus erythematosus.
@en
TIRAP
@nl
type
label
TIRAP (MAL) S180L polymorphism ...... systemic lupus erythematosus.
@en
TIRAP
@nl
prefLabel
TIRAP (MAL) S180L polymorphism ...... systemic lupus erythematosus.
@en
TIRAP
@nl
P2093
P50
P1476
TIRAP (MAL) S180L polymorphism ...... d systemic lupus erythematosus
@en
P2093
Diana-Cristina Varela
John Castiblanco
María-Eugenia Hincapié
P304
P356
10.1016/J.MEEGID.2008.03.001
P50
P577
2008-03-12T00:00:00Z