Increased CD36 expression in middle-aged mice contributes to obesity-related cardiac hypertrophy in the absence of cardiac dysfunction.
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Sphingolipids in High Fat Diet and Obesity-Related DiseasesLeft ventricular mechanical dysfunction in diet-induced obese mice is exacerbated during inotropic stress: a cine DENSE cardiovascular magnetic resonance studyObesity reduces left ventricular strains, torsion, and synchrony in mouse models: a cine displacement encoding with stimulated echoes (DENSE) cardiovascular magnetic resonance study.Increased hepatic CD36 expression with age is associated with enhanced susceptibility to nonalcoholic fatty liver disease.Common variation neighbouring micro-RNA 22 is associated with increased left ventricular massPerspectives of Targeting mTORC1-S6K1 in Cardiovascular AgingCardiospecific CD36 suppression by lentivirus-mediated RNA interference prevents cardiac hypertrophy and systolic dysfunction in high-fat-diet induced obese mice.CD36 protein influences myocardial Ca2+ homeostasis and phospholipid metabolism: conduction anomalies in CD36-deficient mice during fasting.Normalization of cardiac substrate utilization and left ventricular hypertrophy precede functional recovery in heart failure regression.Mammalian target of rapamycin (mTOR) inhibition with rapamycin improves cardiac function in type 2 diabetic mice: potential role of attenuated oxidative stress and altered contractile protein expression.mTOR signalling: the molecular interface connecting metabolic stress, aging and cardiovascular diseases.LXRα improves myocardial glucose tolerance and reduces cardiac hypertrophy in a mouse model of obesity-induced type 2 diabetes.Fatty acid metabolism in pulmonary arterial hypertension: role in right ventricular dysfunction and hypertrophy.Mechanisms of Lipid Accumulation in the Bone Morphogenetic Protein Receptor Type 2 Mutant Right Ventricle.Resveratrol improves exercise performance and skeletal muscle oxidative capacity in heart failure.Alterations of specific lipid groups in serum of obese humans: a review.Cardiomyocyte-specific ablation of CD36 accelerates the progression from compensated cardiac hypertrophy to heart failure.Inhibition of serine palmitoyl transferase I reduces cardiac ceramide levels and increases glycolysis rates following diet-induced insulin resistance.Rheb is a critical regulator of autophagy during myocardial ischemia: pathophysiological implications in obesity and metabolic syndrome.Inhibition of mammalian target of rapamycin protects against reperfusion injury in diabetic heart through STAT3 signaling.Differential effects of Mas receptor deficiency on cardiac function and blood pressure in obese male and female mice.Cluster-determinant 36 (CD36) impacts on vitamin E postprandial response.Transgenic rescue of defective Cd36 enhances myocardial adenylyl cyclase signaling in spontaneously hypertensive rats.Emerging Role of mTOR Signaling-Related miRNAs in Cardiovascular Diseases
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P2860
Increased CD36 expression in middle-aged mice contributes to obesity-related cardiac hypertrophy in the absence of cardiac dysfunction.
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2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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name
Increased CD36 expression in m ...... bsence of cardiac dysfunction.
@en
Increased CD36 expression in m ...... bsence of cardiac dysfunction.
@nl
type
label
Increased CD36 expression in m ...... bsence of cardiac dysfunction.
@en
Increased CD36 expression in m ...... bsence of cardiac dysfunction.
@nl
prefLabel
Increased CD36 expression in m ...... bsence of cardiac dysfunction.
@en
Increased CD36 expression in m ...... bsence of cardiac dysfunction.
@nl
P2093
P2860
P1476
Increased CD36 expression in m ...... bsence of cardiac dysfunction.
@en
P2093
Carrie-Lynn M Soltys
Debby P Y Koonen
Jason R B Dyck
Maria Febbraio
Miranda M Y Sung
P2860
P2888
P304
P356
10.1007/S00109-010-0720-4
P577
2011-03-10T00:00:00Z