Snail, Slug, and Smad-interacting protein 1 as novel parameters of disease aggressiveness in metastatic ovarian and breast carcinoma. - Wikidata - awgldk - TriplyDB

Snail, Slug, and Smad-interacting protein 1 as novel parameters of disease aggressiveness in metastatic ovarian and breast carcinoma.

Snail, Slug, and Smad-interacting protein 1 as novel parameters of disease aggressiveness in metastatic ovarian and breast carcinoma.

http://www.wikidata.org/entity/Q54677737

about

SCUBE3 is an endogenous TGF-β receptor ligand and regulates the epithelial-mesenchymal transition in lung cancer 14-3-3zeta Cooperates with ErbB2 to promote ductal carcinoma in situ progression to invasive breast cancer by inducing epithelial-mesenchymal transition Erk5 controls Slug expression and keratinocyte activation during wound healing The ZEB/miR-200 feedback loop--a motor of cellular plasticity in development and cancer?Regulation of miR-200 family microRNAs and ZEB transcription factors in ovarian cancer: evidence supporting a mesothelial-to-epithelial transition The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2 SIP1/ZEB2 induces EMT by repressing genes of different epithelial cell-cell junctions Epithelial to Mesenchymal Transition in a Clinical Perspective A periodic table for cancer Central role of Snail1 in the regulation of EMT and resistance in cancer: a target for therapeutic intervention Sheep, wolf, or werewolf: cancer stem cells and the epithelial-to-mesenchymal transition E-cadherin's dark side: possible role in tumor progression Pathophysiology of colorectal peritoneal carcinomatosis: Role of the peritoneum Prognostic significance of NBS1 and Snail expression in esophageal squamous cell carcinoma Anticancer effects of 15d-prostaglandin-J2 in wild-type and doxorubicin-resistant ovarian cancer cells: novel actions on SIRT1 and HDAC Tuberin regulates E-cadherin localization: implications in epithelial-mesenchymal transition Dedifferentiation of foetal CNS stem cells to mesendoderm-like cells through an EMT process The transcription factors Snail and Slug activate the transforming growth factor-beta signaling pathway in breast cancer Pancreatic cancers epigenetically silence SIP1 and hypomethylate and overexpress miR-200a/200b in association with elevated circulating miR-200a and miR-200b levels Mesenchymal gene program-expressing ovarian cancer spheroids exhibit enhanced mesothelial clearance Slug controls stem/progenitor cell growth dynamics during mammary gland morphogenesis Staurosporine augments EGF-mediated EMT in PMC42-LA cells through actin depolymerisation, focal contact size reduction and Snail1 induction - a model for cross-modulation Expression analysis of E-cadherin, Slug and GSK3beta in invasive ductal carcinoma of breast.MiR-101 suppresses the epithelial-to-mesenchymal transition by targeting ZEB1 and ZEB2 in ovarian carcinoma SNAI2/Slug promotes growth and invasion in human gliomas.Role of estrogen receptors and Src signaling in mechanisms of bone metastasis by estrogen receptor positive breast cancers.Yin yang 1 regulates the expression of snail through a distal enhancer.Phenotypic plasticity of neoplastic ovarian epithelium: unique cadherin profiles in tumor progression Ubiquitin ligase Cbl-b represses IGF-I-induced epithelial mesenchymal transition via ZEB2 and microRNA-200c regulation in gastric cancer cells Snail plays an oncogenic role in glioblastoma by promoting epithelial mesenchymal transition.SFRP1 reduction results in an increased sensitivity to TGF-β signaling.Dacomitinib, a pan-inhibitor of ErbB receptors, suppresses growth and invasive capacity of chemoresistant ovarian carcinoma cells Epithelial-mesenchymal transition in cancer metastasis through the lymphatic system.Involvement of members of the cadherin superfamily in cancer Epithelial-mesenchymal transition markers in pancreatic ductal adenocarcinoma SIP1 is downregulated in hepatocellular carcinoma by promoter hypermethylation The EMT (epithelial-mesenchymal-transition)-related protein expression indicates the metastatic status and prognosis in patients with ovarian cancer.Matrix metalloproteinase-9 cooperates with transcription factor Snail to induce epithelial-mesenchymal transition.Ovarian cancer development and metastasis.Notch3 overexpression is related to the recurrence of ovarian cancer and confers resistance to carboplatin.

P2860

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P2860

Snail, Slug, and Smad-interacting protein 1 as novel parameters of disease aggressiveness in metastatic ovarian and breast carcinoma.

http://www.wikidata.org/entity/Q54677737

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

@wuu

2005年学术文章

@zh

2005年学术文章

@zh-cn

2005年学术文章

@zh-hans

2005年学术文章

@zh-my

2005年学术文章

@zh-sg

2005年學術文章

@yue

2005年學術文章

@zh-hant

name

Snail, Slug, and Smad-interact ...... ovarian and breast carcinoma.

@en

Snail, Slug, and Smad-interact ...... ovarian and breast carcinoma.

@nl

type

label

Snail, Slug, and Smad-interact ...... ovarian and breast carcinoma.

@en

Snail, Slug, and Smad-interact ...... ovarian and breast carcinoma.

@nl

prefLabel

Snail, Slug, and Smad-interact ...... ovarian and breast carcinoma.

@en

Snail, Slug, and Smad-interact ...... ovarian and breast carcinoma.

@nl

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Snail, Slug, and Smad-interact ...... ovarian and breast carcinoma.

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P2093

Ben Davidson

http://www.w3.org/2001/XMLSchema#string

Gunnar Kvalheim

http://www.w3.org/2001/XMLSchema#string

Iris Goldberg

http://www.w3.org/2001/XMLSchema#string

Mari Bukholt Elstrand

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Reuven Reich

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Sivan Elloul

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P2860

The Hallmarks of Cancer

Snail and SIP1 increase cancer invasion by upregulating MMP family in hepatocellular carcinoma cells

The E-cadherin/catenin complex: an important gatekeeper in breast cancer tumorigenesis and malignant progression.

The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

Correlation of Snail expression with histological grade and lymph node status in breast carcinomas

The snail superfamily of zinc-finger transcription factors

The transcription factor snail controls epithelial-mesenchymal transitions by repressing E-cadherin expression

New functions for the matrix metalloproteinases in cancer progression

Cisplatin: mode of cytotoxic action and molecular basis of resistance

The two-handed E box binding zinc finger protein SIP1 downregulates E-cadherin and induces invasion

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1631-1643

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scholarly article

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10.1002/CNCR.20946

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8

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103

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Claes Göran Tropé

Jahn M. Nesland

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2005-04-01T00:00:00Z

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7992268

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15742334

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