Loss of HAI-2 in mice with decreased prostasin activity leads to an early-onset intestinal failure resembling congenital tufting enteropathy.

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Loss of HAI-2 in mice with decreased prostasin activity leads to an early-onset intestinal failure resembling congenital tufting enteropathy.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Loss of HAI-2 in mice with dec ...... ongenital tufting enteropathy.

@en

Loss of HAI-2 in mice with dec ...... ongenital tufting enteropathy.

@nl

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Item

label

Loss of HAI-2 in mice with dec ...... ongenital tufting enteropathy.

@en

Loss of HAI-2 in mice with dec ...... ongenital tufting enteropathy.

@nl

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Loss of HAI-2 in mice with dec ...... ongenital tufting enteropathy.

@en

Loss of HAI-2 in mice with dec ...... ongenital tufting enteropathy.

@nl

P1476

Loss of HAI-2 in mice with dec ...... ongenital tufting enteropathy.

@en

P2093

Roman Szabo

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P2860

Regulation of cell surface protease matriptase by HAI2 is essential for placental development, neural tube closure and embryonic survival in mice

Nuclear signalling by tumour-associated antigen EpCAM

Ichthyosis, follicular atrophoderma, and hypotrichosis caused by mutations in ST14 is associated with impaired profilaggrin processing

The epidermal barrier function is dependent on the serine protease CAP1/Prss8

Regulation of feto-maternal barrier by matriptase- and PAR-2-mediated signaling is required for placental morphogenesis and mouse embryonic survival

Reduced prostasin (CAP1/PRSS8) activity eliminates HAI-1 and HAI-2 deficiency-associated developmental defects by preventing matriptase activation

Inhibition of prostasin-induced ENaC activities by PN-1 and regulation of PN-1 expression by TGF-beta1 and aldosterone

Hepatocyte growth factor activator inhibitor type 1 (HAI-1) is required for branching morphogenesis in the chorioallantoic placenta

Hepatocyte growth factor activator inhibitor-1 (HAI-1) is essential for the integrity of basement membranes in the developing placental labyrinth

Mutations of the serine protease CAP1/Prss8 lead to reduced embryonic viability, skin defects, and decreased ENaC activity.

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e0194660

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scholarly article

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10.1371/JOURNAL.PONE.0194660

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P407

English

P433

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P478

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P50

Thomas H. Bugge

P577

2018-04-04T00:00:00Z

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P698

29617460

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P932

5884512

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