Role of Interleukin-10 in the Lung Response to Silica in Mice - Wikidata - awgldk - TriplyDB

Role of Interleukin-10 in the Lung Response to Silica in Mice

Role of Interleukin-10 in the Lung Response to Silica in Mice

http://www.wikidata.org/entity/Q56834786

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The sources of inflammatory mediators in the lung after silica exposure Time course of gene expression of inflammatory mediators in rat lung after diesel exhaust particle exposure Mechanistically identified suitable biomarkers of exposure, effect, and susceptibility for silicosis and coal-worker's pneumoconiosis: A comprehensive review The importance of balanced pro-inflammatory and anti-inflammatory mechanisms in diffuse lung disease The role of pro- and anti-inflammatory responses in silica-induced lung fibrosis.Silica, Silicosis, and Autoimmunity Fibrosis Related Inflammatory Mediators: Role of the IL-10 Cytokine Family Co-localization of iron binding on silica with p62/sequestosome1 (SQSTM1) in lung granulomas of mice with acute silicosis Uncoupling between inflammatory and fibrotic responses to silica: evidence from MyD88 knockout mice Overexpression of apolipoprotein A1 in the lung abrogates fibrosis in experimental silicosis Molecular mechanisms of pulmonary response progression in crystalline silica exposed rats Suppressive oligodeoxynucleotides reduce lung cancer susceptibility in mice with silicosis.Innate immune processes are sufficient for driving silicosis in mice.IL-9 and lung fibrosis: a Th2 good guy?Protective role of interleukin-10 in ozone-induced pulmonary inflammation Cytokine gene polymorphisms and serum cytokine levels in patients with idiopathic pulmonary fibrosis.Phenotypic switching of Cryptococcus neoformans occurs in vivo and influences the outcome of infection Genome-wide Gene-Asbestos Exposure Interaction Association Study Identifies a Common Susceptibility Variant on 22q13.31 Associated with Lung Cancer Risk.Idiopathic pulmonary fibrosis: a decade of progress.The role of inflammation in the pathogenesis of idiopathic pulmonary fibrosis.Effect of IL-10 on neutrophil recruitment and survival after Pseudomonas aeruginosa challenge.Cigarette smoke, inflammation, and lung injury: a mechanistic perspective.The Witschi Hypothesis revisited after 35 years: genetic proof from SP-C BRICHOS domain mutations A mechanistic review of silica-induced inhalation toxicity.Radiation induced pulmonary fibrosis as a model of progressive fibrosis: Contributions of DNA damage, inflammatory response and cellular senescence genes.Tissue remodelling in pulmonary fibrosis.Introduction of the interleukin-10 gene into mice inhibited bleomycin-induced lung injury in vivo.Platelet-derived growth factor-producing CD4+ Foxp3+ regulatory T lymphocytes promote lung fibrosis.IL-6 receptor-mediated lung Th2 cytokine networking in silica-induced pulmonary fibrosis.LRBA Gene Polymorphisms and Risk of Coal Workers' Pneumoconiosis: A Case-Control Study from China.Interleukin-9 reduces lung fibrosis and type 2 immune polarization induced by silica particles in a murine model.Overexpression of tumor necrosis factor-alpha diminishes pulmonary fibrosis induced by bleomycin or transforming growth factor-beta.Anti-monocyte chemoattractant protein-1 gene therapy attenuates pulmonary fibrosis in mice.Characterization of the effect of interleukin-10 on silica-induced lung fibrosis in mice.Pulmonary toxicity following acute coexposures to diesel particulate matter and α-quartz crystalline silica in the Sprague-Dawley rat.Pulmonary overexpression of IL-10 augments lung fibrosis and Th2 responses induced by silica particles.Lipopolysaccharide-induced tumor necrosis factor-alpha and IL-10 production by lung macrophages from preterm and term neonates.Protection against bleomycin-induced lung injury by IL-18 in mice.Surfactant replacement therapy reduces acute lung injury and collapse induration-related lung remodeling in the bleomycin model.Transgenic overexpression of interleukin (IL)-10 in the lung causes mucus metaplasia, tissue inflammation, and airway remodeling via IL-13-dependent and -independent pathways.

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Role of Interleukin-10 in the Lung Response to Silica in Mice

http://www.wikidata.org/entity/Q56834786

description

article

@en

im Januar 1998 veröffentlichter wissenschaftlicher Artikel

@de

wetenschappelijk artikel

@nl

наукова стаття, опублікована в січні 1998

@uk

name

Role of Interleukin-10 in the Lung Response to Silica in Mice

@en

Role of Interleukin-10 in the Lung Response to Silica in Mice

@nl

type

label

Role of Interleukin-10 in the Lung Response to Silica in Mice

@en

Role of Interleukin-10 in the Lung Response to Silica in Mice

@nl

prefLabel

Role of Interleukin-10 in the Lung Response to Silica in Mice

@en

Role of Interleukin-10 in the Lung Response to Silica in Mice

@nl

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AJRCMB.18.1.2911

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American Journal of Respiratory Cell and Molecular Biology

P1476

Role of Interleukin-10 in the Lung Response to Silica in Mice

@en

P2093

Barry Hudspith

http://www.w3.org/2001/XMLSchema#string

Clive Meredith

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François Huaux

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Jamila Louahed

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Jean-Christophe Renauld

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Monique Delos

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P2860

Characterization of alveolar macrophage eicosanoid production in a non-human primate model of mineral dust exposure.

Interleukin 10 is a potent growth and differentiation factor for activated human B lymphocytes

Interleukin-10-deficient mice develop chronic enterocolitis

Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes

Two types of mouse T helper cell. IV. Th2 clones secrete a factor that inhibits cytokine production by Th1 clones

Immune complexes inhibit antimicrobial responses through interleukin-10 production. Effects in severe combined immunodeficient mice during Listeria infection

Interleukin-10 inhibits antigen-induced cellular recruitment into the airways of sensitized mice.

Molecular mechanisms of antifibrotic effect of interferon gamma in bleomycin-mouse model of lung fibrosis: downregulation of TGF-beta and procollagen I and III gene expression.

Silica-stimulated monocytes release fibroblast proliferation factors identical to interleukin 1. A potential role for interleukin 1 in the pathogenesis of silicosis.

Raised immunoglobulin concentrations in bronchoalveolar lavage fluid of healthy granite workers.

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51-59

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scholarly article

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10.1165/AJRCMB.18.1.2911

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1

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18

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Dominique Lison

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1998-01-01T00:00:00Z

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9448045

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