about
The Notch/Hes1 pathway sustains NF-κB activation through CYLD repression in T cell leukemia.Hes repressors are essential regulators of hematopoietic stem cell development downstream of Notch signaling.The Notch1 transcriptional activation domain is required for development and reveals a novel role for Notch1 signaling in fetal hematopoietic stem cells.The Notch pathway in hematopoietic stem cells.Hes1 expression and CYLD repression are essential events downstream of Notch1 in T-cell leukemia.β-Catenin is required for T-cell leukemia initiation and MYC transcription downstream of Notch1.Notch1 Pathway Activation Results from the Epigenetic Abrogation of Notch-Related MicroRNAs in Mycosis Fungoides.A new genetic tool to improve immune-compromised mouse models: Derivation and CRISPR/Cas9-mediated targeting of NRG embryonic stem cell linesMutant CEBPA directly drives the expression of the targetable tumor-promoting factor CD73 in AMLHeterozygous loss of Srp72 in mice is not associated with major hematological phenotypes
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description
researcher ORCID ID = 0000-0002-8146-5365
@en
wetenschapper
@nl
name
Teresa D'Altri
@ast
Teresa D'Altri
@en
Teresa D'Altri
@es
Teresa D'Altri
@nl
type
label
Teresa D'Altri
@ast
Teresa D'Altri
@en
Teresa D'Altri
@es
Teresa D'Altri
@nl
prefLabel
Teresa D'Altri
@ast
Teresa D'Altri
@en
Teresa D'Altri
@es
Teresa D'Altri
@nl
P106
P21
P31
P496
0000-0002-8146-5365