about
A genome-wide gene-expression analysis and database in transgenic mice during development of amyloid or tau pathologyAge-dependent alterations of corticostriatal activity in the YAC128 mouse model of Huntington diseaseHippocampal circuit dysfunction in the Tc1 mouse model of Down syndrome.Forebrain striatal-specific expression of mutant huntingtin protein in vivo induces cell-autonomous age-dependent alterations in sensitivity to excitotoxicity and mitochondrial function.Large and Small Dendritic Spines Serve Different Interacting Functions in Hippocampal Synaptic Plasticity and Homeostasis.Bi-directional plasticity and age-dependent long-term depression at mouse CA3-CA1 hippocampal synapses.Dissecting the contribution of individual receptor subunits to the enhancement of N-methyl-d-aspartate currents by dopamine D1 receptor activation in striatumDynamic range of GSK3α not GSK3β is essential for bidirectional synaptic plasticity at hippocampal CA3-CA1 synapses.Dopamine modulation of excitatory currents in the striatum is dictated by the expression of D1 or D2 receptors and modified by endocannabinoids.Changes in Dopamine Signalling Do Not Underlie Aberrant Hippocampal Plasticity in a Mouse Model of Huntington's Disease.Early development of aberrant synaptic plasticity in a mouse model of Huntington's disease.Aberrant cortical synaptic plasticity and dopaminergic dysfunction in a mouse model of Huntington's disease.Progressive CAG expansion in the brain of a novel R6/1-89Q mouse model of Huntington's disease with delayed phenotypic onset.Dysfunctional Dopaminergic Neurones in Mouse Models of Huntington's Disease: A Role for SK3 Channels.Neuronal coupling via connexin36 contributes to spontaneous synaptic currents of striatal medium-sized spiny neurons.Neuronal and Peripheral Pentraxins Modify Glutamate Release and may Interact in Blood-Brain Barrier Failure.Effects of rising amyloidβ levels on hippocampal synaptic transmission, microglial response and cognition in APPSwe/PSEN1M146V transgenic miceImproving Mouse Models for Dementia. Are All the Effects in Tau Mouse Models Due to Overexpression?Genetic variability in response to amyloid beta deposition influences Alzheimer's disease riskDevelopmental origins of cortical hyperexcitability in Huntington's disease: Review and new observationsDementia associated with disorders of the basal ganglia
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description
Forscher
@de
investigador
@es
researcher
@en
ricercatore
@it
wetenschapper
@nl
հետազոտող
@hy
研究者
@zh
name
Damian M Cummings
@ast
Damian M Cummings
@en
Damian M Cummings
@es
Damian M Cummings
@nl
type
label
Damian M Cummings
@ast
Damian M Cummings
@en
Damian M Cummings
@es
Damian M Cummings
@nl
prefLabel
Damian M Cummings
@ast
Damian M Cummings
@en
Damian M Cummings
@es
Damian M Cummings
@nl
P106
P31
P496
0000-0001-5567-0907