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Elevated ventricular wall stress disrupts cardiomyocyte t-tubule structure and calcium homeostasisMutual antagonism between IP(3)RII and miRNA-133a regulates calcium signals and cardiac hypertrophyDeletion of mouse Alkbh7 leads to obesity.A semiautomatic method for rapid segmentation of velocity-encoded myocardial magnetic resonance imaging data.Toll-Like Receptor 9 Promotes Survival in SERCA2a KO Heart Failure Mice.Genome-wide profiling of DNA 5-hydroxymethylcytosine during rat Sertoli cell maturation.Compensatory and decompensatory alterations in cardiomyocyte Ca2+ dynamics in hearts with diastolic dysfunction following aortic banding.Attenuated fatigue in slow twitch skeletal muscle during isotonic exercise in rats with chronic heart failure.Differential regulation of β2 -adrenoceptor-mediated inotropic and lusitropic response by PDE3 and PDE4 in failing and non-failing rat cardiac ventricle.Molecular basis of calpain cleavage and inactivation of the sodium-calcium exchanger 1 in heart failure.Pentosan polysulfate decreases myocardial expression of the extracellular matrix enzyme ADAMTS4 and improves cardiac function in vivo in rats subjected to pressure overload by aortic banding.Agents increasing cyclic GMP amplify 5-HT4-elicited positive inotropic response in failing rat cardiac ventricle.Sustained Toll-Like Receptor 9 Activation Promotes Systemic and Cardiac Inflammation, and Aggravates Diastolic Heart Failure in SERCA2a KO MiceThree-Directional Evaluation of Mitral Flow in the Rat Heart by Phase-Contrast Cardiovascular Magnetic Resonance.Beta-Adrenoceptor Stimulation Reveals Ca2+ Waves and Sarcoplasmic Reticulum Ca2+ Depletion in Left Ventricular Cardiomyocytes from Post-Infarction Rats with and without Heart Failure.Protein Phosphatase 1c Associated with the Cardiac Sodium Calcium Exchanger 1 Regulates Its Activity by Dephosphorylating Serine 68-phosphorylated Phospholemman.Exercise training increases protein O-GlcNAcylation in rat skeletal muscleSERCA2 activity is involved in the CNP-mediated functional responses in failing rat myocardium.Novel insight into the detailed myocardial motion and deformation of the rodent heart using high-resolution phase contrast cardiovascular magnetic resonance.The H3K9 dimethyltransferases EHMT1/2 protect against pathological cardiac hypertrophyCardiac sodium transport and excitation-contraction coupling.Regional Dysfunction After Myocardial Infarction in Rats.Cardiomyocyte Ca2+ dynamics: clinical perspectives.Noninvasive stratification of postinfarction rats based on the degree of cardiac dysfunction using magnetic resonance imaging and echocardiography.Exhausting treadmill running causes dephosphorylation of sMLC2 and reduced level of myofilament MLCK2 in slow twitch rat soleus muscleShedding of syndecan-4 promotes immune cell recruitment and mitigates cardiac dysfunction after lipopolysaccharide challenge in mice.5-HT4-elicited positive inotropic response is mediated by cAMP and regulated by PDE3 in failing rat and human cardiac ventricles.Hypokalaemia induces Ca(2+) overload and Ca(2+) waves in ventricular myocytes by reducing Na(+),K(+)-ATPase α2 activity.Beta-adrenergic stimulation maintains cardiac function in Serca2 knockout mice.Sodium accumulation in SERCA knockout-induced heart failure.Cyclic AMP-dependent inotropic effects are differentially regulated by muscarinic G(i)-dependent constitutive inhibition of adenylyl cyclase in failing rat ventricleThe functional activity of inhibitory G protein (G(i)) is not increased in failing heart ventricle.Cre-loxP DNA recombination is possible with only minimal unspecific transcriptional changes and without cardiomyopathy in Tg(alphaMHC-MerCreMer) mice.Cyclic GMP generated by natriuretic peptide receptor B enhances β1-adrenoceptor signaling in normal and failing hearts.The mechanism of CNP-induced negative inotropic and positive lusitropic responses are dependent on SERCA activity in failing rat ventricle.Temporary fatigue and altered extracellular matrix in skeletal muscle during progression of heart failure in rats.Cardiomyocytes from postinfarction failing rat hearts have improved ischemia tolerance.Enhancement of cAMP-mediated inotropic responses by CNP is regulated differently by PDE2 in normal and failing hearts.Improved MR phase-contrast velocimetry using a novel nine-point balanced motion-encoding scheme with increased robustness to eddy current effects.Altered Na+/Ca2+-exchanger activity due to downregulation of Na+/K+-ATPase alpha2-isoform in heart failure.
P50
Q27302066-7C55A911-C4C6-45CE-886C-E60D169895F2Q28771365-A6B3F92B-D7F9-407C-A0B4-2BB716535AE2Q29347294-D5AA6B60-48DF-4215-A635-CA3BE6D9F7BAQ31134625-AB8A46BA-37AB-4D3E-8384-B13E95A25A22Q33604761-165E3894-188B-48BD-AAA5-A2B85050E776Q33651917-F3A861EB-2C2E-4F21-92CD-8EB44C5C8C8AQ33800047-8DDF0238-7DAF-4DBF-B90D-3D4D932D5F78Q33974952-8672874A-B780-44F7-AC9D-2BF1A727F427Q34449709-F4089028-47FC-4550-B07B-C741F18D659BQ34634178-3E58382D-298F-4E88-A952-6097D8F0B610Q35111270-A6D3F32A-E719-4754-97F0-F34BDEABF2F8Q35563508-0AE03EF1-33CF-499F-9488-23B03DA4B563Q35804806-0EE12E35-4504-49EC-AD99-892DFAF7C5EFQ35940745-44B22B6D-B8A2-4584-B5D2-247D53F4CB87Q35994821-BC5CD7C8-BD1A-46E9-9746-740D535D65B9Q36744537-7C24E670-7307-40EF-9218-8A8BD6A62524Q37286799-62FE9801-F0A8-461E-A556-8193F2B55D97Q37327400-E7D8EA2D-B43E-4ACC-8EA1-F695528C4225Q37360072-20000A08-4BF5-4070-A4F1-C450EAF512A1Q37544984-C3D2D69D-4C24-4ED4-90C7-AF00D1F3157CQ38115058-8888084F-2207-4144-90AB-D497ED8FF50FQ38609492-CE1655D3-45EB-4E3B-8EA0-C9E773C4FA5DQ38686480-1AEFC0DC-0CF9-4E97-9DD0-FB2868E272EFQ38966607-9B2822DF-B1C3-49F6-9AF0-4A47857F0A07Q39509213-9C9B0D98-0C8F-407F-ACA9-E19A9EF76FD4Q40451749-3FB94A07-3886-4804-B736-B64CB12A7E41Q41139399-0A384FD5-70BE-479F-A686-A733C7FDA4C3Q41220726-140E0970-AE91-4512-B228-688AEA54331AQ41841957-634EE16E-9698-4FDD-A8D3-7D5D05939691Q41954642-0B159953-4960-4BBD-AE21-1B79C66874ACQ42058871-3173D2EA-829A-46EE-9707-6D33E4D677A4Q42517602-F97271E3-54E8-46FE-AFD8-D6B8B000F428Q42923082-E14B3080-7993-44AD-817E-92A09FAC828DQ45928769-5D80A6E9-C155-4C79-8693-19304C141DC1Q45928791-8850DB22-0FEA-4F9B-BCA1-0FC5A9FB770FQ46064882-42FABB88-63BE-46B5-8EC1-EC73AEE1EB65Q46167724-739B5B7B-40F3-4244-BE1C-0229B3CFEB6EQ46307122-A3546E6E-12FE-4DBC-BA2D-A74890F4B44FQ46398265-A24FE460-586A-44C1-8F59-C3C8CCBF4A03Q46802227-79D6F5B8-F6CF-4084-B6E0-A43FFB8D49B2
P50
description
Noors onderzoeker
@nl
Norwegian researcher
@en
norsk forsker
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taighdeoir Ioruach
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name
Jan Magnus Aronsen
@ast
Jan Magnus Aronsen
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Jan Magnus Aronsen
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Jan Magnus Aronsen
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Jan Magnus Aronsen
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Jan Magnus Aronsen
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Jan Magnus Aronsen
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Jan Magnus Aronsen
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Jan Magnus Aronsen
@nl
Jan Magnus Aronsen
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type
label
Jan Magnus Aronsen
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Jan Magnus Aronsen
@da
Jan Magnus Aronsen
@de
Jan Magnus Aronsen
@en
Jan Magnus Aronsen
@es
Jan Magnus Aronsen
@fr
Jan Magnus Aronsen
@ga
Jan Magnus Aronsen
@nb
Jan Magnus Aronsen
@nl
Jan Magnus Aronsen
@nn
altLabel
Jan Aronsen
@nb
Jan M Aronsen
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Jan M. Aronsen
@nb
prefLabel
Jan Magnus Aronsen
@ast
Jan Magnus Aronsen
@da
Jan Magnus Aronsen
@de
Jan Magnus Aronsen
@en
Jan Magnus Aronsen
@es
Jan Magnus Aronsen
@fr
Jan Magnus Aronsen
@ga
Jan Magnus Aronsen
@nb
Jan Magnus Aronsen
@nl
Jan Magnus Aronsen
@nn
P214
P106
P21
P214
P2287
P27
P31
P569
1982-01-01T00:00:00Z
P734
P7859
viaf-228865906