about
Mutations in the p16INK4/MTS1/CDKN2, p15INK4B/MTS2, and p18 genes in primary and metastatic lung cancerTumor microenvironment-based feed-forward regulation of NOS2 in breast cancer progression.Metabolic profiles are principally different between cancers of the liver, pancreas and breast.Molecular pathogenesis of human hepatocellular carcinoma.Macrophage-dependent nitric oxide expression regulates tumor cell detachment and metastasis after IL-2/anti-CD40 immunotherapy.The adaptive imbalance in base excision-repair enzymes generates microsatellite instability in chronic inflammation.Selective targeting of KRAS-mutant cells by miR-126 through repression of multiple genes essential for the survival of KRAS-mutant cellsTumor suppressor genes: at the crossroads of molecular carcinogenesis, molecular epidemiology and human risk assessment.Nitric oxide-induced cellular stress and p53 activation in chronic inflammationThe p53 tumor suppressor network is a key responder to microenvironmental components of chronic inflammatory stress.TP53 and liver carcinogenesis.Radical causes of cancer.Nitric oxide in cancer and chemoprevention.Nitric oxide and p53 in cancer-prone chronic inflammation and oxyradical overload disease.Mutations and altered expression of p16INK4 in human cancer.p53 biological network: at the crossroads of the cellular-stress response pathway and molecular carcinogenesis.Inflammation and cancer: an ancient link with novel potentials.A Novel MIF Signaling Pathway Drives the Malignant Character of Pancreatic Cancer by Targeting NR3C2.Integration of metabolomics and transcriptomics revealed a fatty acid network exerting growth inhibitory effects in human pancreatic cancerAutocrine IL-10 functions as a rheostat for M1 macrophage glycolytic commitment by tuning nitric oxide productionInducible nitric oxide synthase enhances disease aggressiveness in pancreatic cancer.Regulation of human nitric oxide synthase 2 expression by Wnt beta-catenin signaling.NO• and Pancreatic Cancer: A Complex Interaction with Therapeutic Potential.p53-induced up-regulation of MnSOD and GPx but not catalase increases oxidative stress and apoptosis.Regulation of cyclooxygenase-2 expression by the Wnt and ras pathways.Modulation of 7,12-dimethylbenz[a]anthracene-induced transmammary carcinogenesis by disulfiram and butylated hydroxyanisole in mice.p53 tumor suppressor gene: at the crossroads of molecular carcinogenesis, molecular epidemiology, and human risk assessment.Endothelial Nitric Oxide Synthase Traffic Inducer (NOSTRIN) is a Negative Regulator of Disease Aggressiveness in Pancreatic Cancer.Chronic inflammation promotes retinoblastoma protein hyperphosphorylation and E2F1 activation.Nitric oxide, a mediator of inflammation, suppresses tumorigenesis.Relationship between p53 mutations and inducible nitric oxide synthase expression in human colorectal cancer.p53 and vascular endothelial growth factor regulate tumor growth of NOS2-expressing human carcinoma cells.Mutagenesis of codon 248 of the human p53 tumor suppressor gene by N-ethyl-N-nitrosourea.Chemopreventive action of garlic on methylcholanthrene-induced carcinogenesis in the uterine cervix of mice.Chemopreventive action of mace (Myristica fragrans, Houtt) on DMBA-induced papillomagenesis in the skin of miceModulation of methylcholanthrene-induced carcinogenesis in the uterine cervix of mouse by indomethacinOxy-radical induced mutagenesis of hotspot codons 248 and 249 of the human p53 geneMolecular epidemiology of human cancerMolecular epidemiology of human cancer: contribution of mutation spectra studies of tumor suppressor genes
P50
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P50
description
Forscher
@de
chercheur
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investigador
@es
researcher
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ricercatore
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研究者
@zh
name
S P Hussain
@ast
S P Hussain
@en
S P Hussain
@es
S P Hussain
@nl
type
label
S P Hussain
@ast
S P Hussain
@en
S P Hussain
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S P Hussain
@nl
prefLabel
S P Hussain
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S P Hussain
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S P Hussain
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S P Hussain
@nl
P108
P31
P496
0000-0002-2294-522X