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Expression of cassini, a murine gamma-satellite sequence conserved in evolution, is regulated in normal and malignant hematopoietic cells53BP1 inhibits homologous recombination in Brca1-deficient cells by blocking resection of DNA breaksRif1 prevents resection of DNA breaks and promotes immunoglobulin class switchingMolecular portraits of B cell lineage commitment.T lymphoid differentiation in human bone marrowA method for identification of HIV gp140 binding memory B cells in human blood.AID produces DNA double-strand breaks in non-Ig genes and mature B cell lymphomas with reciprocal chromosome translocations.53BP1 regulates DNA resection and the choice between classical and alternative end joining during class switch recombination.Deficiency of Bruton's tyrosine kinase in B cell precursor leukemia cells.The B cell mutator AID promotes B lymphoid blast crisis and drug resistance in chronic myeloid leukemia.RETRACTED: Silencing of B cell receptor signals in human naive B cells.Broad diversity of neutralizing antibodies isolated from memory B cells in HIV-infected individuals.Regulation of DNA end joining, resection, and immunoglobulin class switch recombination by 53BP1.Interference of BCR-ABL1 kinase activity with antigen receptor signaling in B cell precursor leukemia cells.Plasmodium Infection Promotes Genomic Instability and AID-Dependent B Cell Lymphoma.Environment-mediated drug resistance in Bcr/Abl-positive acute lymphoblastic leukemiaNilotinib treatment in mouse models of P190 Bcr/Abl lymphoblastic leukemia.The hSSB1 orthologue Obfc2b is essential for skeletogenesis but dispensable for the DNA damage response in vivoA new human somatic stem cell from placental cord blood with intrinsic pluripotent differentiation potential.Mimicry of a constitutively active pre-B cell receptor in acute lymphoblastic leukemia cellsThe BCR-ABL1 kinase bypasses selection for the expression of a pre-B cell receptor in pre-B acute lymphoblastic leukemia cells.53BP1 alters the landscape of DNA rearrangements and suppresses AID-induced B cell lymphoma.Lineage-Specific Genes Are Prominent DNA Damage Hotspots during Leukemic Transformation of B Cell Precursors.Activation-induced cytidine deaminase acts as a mutator in BCR-ABL1-transformed acute lymphoblastic leukemia cells.Tracing the pre-B to immature B cell transition in human leukemia cells reveals a coordinated sequence of primary and secondary IGK gene rearrangement, IGK deletion, and IGL gene rearrangement.The molecular requirements for LAT-mediated differentiation and the role of LAT in limiting pre-B cell expansion.Mutations in SNRPB, encoding components of the core splicing machinery, cause cerebro-costo-mandibular syndrome.Visceral Adipose Tissue Immune Homeostasis Is Regulated by the Crosstalk between Adipocytes and Dendritic Cell Subsets.B-lymphoid or myeloid lineage identity of cell lines derived from chronic myeloid leukemia blast crisis.Retraction
P50
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P50
name
Niklas Feldhahn
@en
type
label
Niklas Feldhahn
@en
prefLabel
Niklas Feldhahn
@en
P2038
Niklas_Feldhahn