Early alterations of brain cellular energy homeostasis in Huntington disease models - Test2 - elhamkhani - TriplyDB

Early alterations of brain cellular energy homeostasis in Huntington disease models

Early alterations of brain cellular energy homeostasis in Huntington disease models

http://www.wikidata.org/entity/Q23912810

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Mouse models of polyglutamine diseases: review and data table. Part I PGC-1α, mitochondrial dysfunction, and Huntington's disease Modeling Huntington's disease with induced pluripotent stem cells Possible involvement of self-defense mechanisms in the preferential vulnerability of the striatum in Huntington's disease Shaping the role of mitochondria in the pathogenesis of Huntington's disease Role of oxidative DNA damage in mitochondrial dysfunction and Huntington's disease pathogenesis HdhQ111 Mice Exhibit Tissue Specific Metabolite Profiles that Include Striatal Lipid Accumulation N-Acetylcysteine improves mitochondrial function and ameliorates behavioral deficits in the R6/1 mouse model of Huntington's disease Mitochondrial dysfunction in neurodegenerative diseases Regulation of neuronal bioenergy homeostasis by glutamate Triheptanoin improves brain energy metabolism in patients with Huntington disease A novel manganese-dependent ATM-p53 signaling pathway is selectively impaired in patient-based neuroprogenitor and murine striatal models of Huntington's disease Enhanced neuronal glucose transporter expression reveals metabolic choice in a HD Drosophila model Energy failure: does it contribute to neurodegeneration?Homeostatic adaptations in brain energy metabolism in mouse models of Huntington disease A broad phenotypic screen identifies novel phenotypes driven by a single mutant allele in Huntington's disease CAG knock-in mice.In vivo neurometabolic profiling in patients with spinocerebellar ataxia types 1, 2, 3, and 7 Gender differences in the effect of tobacco use on brain phosphocreatine levels in methamphetamine-dependent subjects Pre-symptomatic activation of antioxidant responses and alterations in glucose and pyruvate metabolism in Niemann-Pick Type C1-deficient murine brain Compensation in Preclinical Huntington's Disease: Evidence From the Track-On HD Study A dual role for AMP-activated protein kinase (AMPK) during neonatal hypoxic-ischaemic brain injury in mice Combinatorial assessments of brain tissue metabolomics and histopathology in rodent models of human immunodeficiency virus infection Metabolism in HD: still a relevant mechanism?Inhibition of mitochondrial protein import by mutant huntingtin Conditioned medium of human adipose-derived mesenchymal stem cells mediates protection in neurons following glutamate excitotoxicity by regulating energy metabolism and GAP-43 expression Oxygen consumption deficit in Huntington disease mouse brain under metabolic stress Huntington’s disease blood and brain show a common gene expression pattern and share an immune signature with Alzheimer’s disease Energy dysfunction in Huntington's disease: insights from PGC-1α, AMPK, and CKB.AMP-activated protein kinase: new regulation, new roles?Genetic modifiers of Huntington's disease.Huntington's Disease: Pathogenic Mechanisms and Therapeutic Targets.Imaging and spectroscopic approaches to probe brain energy metabolism dysregulation in neurodegenerative diseases.AMPK in Neurodegenerative Diseases.Localized changes to glycogen synthase kinase-3 and collapsin response mediator protein-2 in the Huntington's disease affected brain.Triheptanoin for the treatment of brain energy deficit: A 14-year experience.Co-administration of creatine plus pyruvate prevents the effects of phenylalanine administration to female rats during pregnancy and lactation on enzymes activity of energy metabolism in cerebral cortex and hippocampus of the offspring.Early down-regulation of PKCδ as a pro-survival mechanism in Huntington's disease.Abnormal response to cortical activation in early stages of Huntington disease.A study of molecular changes relating to energy metabolism and cellular stress in people with Huntington's disease: looking for biomarkers.Oxidizing effects of exogenous stressors in Huntington's disease knock-in striatal cells--protective effect of cystamine and creatine.

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P2860

Early alterations of brain cellular energy homeostasis in Huntington disease models

http://www.wikidata.org/entity/Q23912810

description

2012 nî lūn-bûn

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2012 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2012 թվականի հունվարին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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JBC.M111.309849

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Journal of Biological Chemistry

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Early alterations of brain cellular energy homeostasis in Huntington disease models

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Fanny Mochel

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Hua Yu

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Raphael Schiffmann

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Sandrine Humbert

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Vanessa C. Wheeler

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Xingli Meng

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P2860

Huntingtin interacting proteins are genetic modifiers of neurodegeneration

Focused microwave irradiation of the brain preserves in vivo protein phosphorylation: comparison with other methods of sacrifice and analysis of multiple phosphoproteins

AMP-activated protein kinase phosphorylation in brain is dependent on method of killing and tissue preparation

Creatine synthesis and transport during rat embryogenesis: spatiotemporal expression of AGAT, GAMT and CT1.

Measurement of protein using bicinchoninic acid

A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

Impaired brain creatine kinase activity in Huntington's disease.

The power of automated high-resolution behavior analysis revealed by its application to mouse models of Huntington's and prion diseases.

Dysregulated brain creatine kinase is associated with hearing impairment in mouse models of Huntington disease

Early energy deficit in Huntington disease: identification of a plasma biomarker traceable during disease progression.

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scholarly article

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10.1074/JBC.M111.309849

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2

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287

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Emmanuel Brouillet

James P. O'Callaghan

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2012-01-06T00:00:00Z

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22123819

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National Institute for Occupational Safety and Health

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Huntington disease

human behaviour

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3256882

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