Hepatitis C virus subgenomic replicons induce endoplasmic reticulum stress activating an intracellular signaling pathway
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Stress responses in flavivirus-infected cells: activation of unfolded protein response and autophagyProtein synthesis and endoplasmic reticulum stress can be modulated by the hepatitis C virus envelope protein E2 through the eukaryotic initiation factor 2alpha kinase PERKInvolvement of PKR and RNase L in translational control and induction of apoptosis after Hepatitis C polyprotein expression from a vaccinia virus recombinantHepatitis C Virus Infection Induces Autophagy as a Prosurvival Mechanism to Alleviate Hepatic ER-Stress ResponseInflammatory and oxidative stress in rotavirus infectionChaperones in hepatitis C virus infectionSignificance of hepatitis virus infection in the oncogenic initiation of hepatocellular carcinomaHepatitis C virus comes for dinner: How the hepatitis C virus interferes with autophagyInterplay between Inflammation and Cellular Stress Triggered by Flaviviridae VirusesHCV and Oxidative Stress: Implications for HCV Life Cycle and HCV-Associated PathogenesisHuman cytomegalovirus infection activates and regulates the unfolded protein response.Hepatitis C Virus Stimulates the Expression of Cyclooxygenase-2 via Oxidative Stress: Role of Prostaglandin E2 in RNA ReplicationCell-free replication of the hepatitis C virus subgenomic replicon.Two-step affinity purification of the hepatitis C virus ribonucleoprotein complex.Flavivirus Infection Activates the XBP1 Pathway of the Unfolded Protein Response To Cope with Endoplasmic Reticulum StressReplication and IRES-Dependent Translation Are Both Affected by Core Coding Sequences in Subgenomic GB Virus B RepliconsHepatitis C Virus Induces Proteolytic Cleavage of Sterol Regulatory Element Binding Proteins and Stimulates Their Phosphorylation via Oxidative StressWest Nile Virus Infection Activates the Unfolded Protein Response, Leading to CHOP Induction and ApoptosisDengue virus serotype infection specifies the activation of the unfolded protein responseCre-estrogen receptor-mediated hepatitis C virus structural protein expression in miceHepatitis C Virus Genotype 1a Growth and Induction of AutophagyCoronavirus Infection Modulates the Unfolded Protein Response and Mediates Sustained Translational RepressionCell Surface Expression of Major Histocompatibility Complex Class I Molecules Is Reduced in Hepatitis C Virus Subgenomic Replicon-Expressing CellsSubcellular forms and biochemical events triggered in human cells by HCV polyprotein expression from a viral vectorEndoplasmic Reticulum Stress Is a Determinant of Retrovirus-Induced Spongiform NeurodegenerationArsenic Trioxide Inhibits Hepatitis C Virus RNA Replication through Modulation of the Glutathione Redox System and Oxidative StressHepatitis C Virus Proteins Induce Lipogenesis and Defective Triglyceride Secretion in Transgenic MiceDengue Virus Infection Induces Upregulation of GRP78, Which Acts To Chaperone Viral Antigen ProductionTemporal Proteome and Lipidome Profiles Reveal Hepatitis C Virus-Associated Reprogramming of Hepatocellular Metabolism and BioenergeticsCellular stress response and innate immune signaling: integrating pathways in host defense and inflammation.Endoplasmic reticulum stress activates the hepatic activator protein 1 complex via mitogen activated protein kinase-dependent signaling pathwaysHCV causes chronic endoplasmic reticulum stress leading to adaptation and interference with the unfolded protein responseHepatitis C virus induces the mitochondrial translocation of Parkin and subsequent mitophagyER stress affects processing of MHC class I-associated peptides.The SARS Coronavirus 3a protein causes endoplasmic reticulum stress and induces ligand-independent downregulation of the type 1 interferon receptor.Homocysteine supplementation attenuates the unfolded protein response in a murine nutritional model of steatohepatitis.Hepatitis C virus triggers mitochondrial fission and attenuates apoptosis to promote viral persistencePatterns of host genome-wide gene transcript abundance in the peripheral blood of patients with acute dengue hemorrhagic fever.Hepatitis C virus (HCV) constitutively activates STAT-3 via oxidative stress: role of STAT-3 in HCV replicationER stress, autophagy, and RNA viruses.
P2860
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P2860
Hepatitis C virus subgenomic replicons induce endoplasmic reticulum stress activating an intracellular signaling pathway
description
2002 nî lūn-bûn
@nan
2002 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@ast
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@en
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@nl
type
label
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@ast
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@en
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@nl
altLabel
Hepatitis C Virus Subgenomic R ...... ntracellular Signaling Pathway
@en
prefLabel
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@ast
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@en
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@nl
P2093
P2860
P3181
P1433
P1476
Hepatitis C virus subgenomic r ...... ntracellular signaling pathway
@en
P2093
Aleem Siddiqui
Kazutoshi Mori
Keith D Tardif
P2860
P304
P3181
P356
10.1128/JVI.76.15.7453-7459.2002
P407
P577
2002-08-01T00:00:00Z