Glucose metabolism attenuates p53 and Puma-dependent cell death upon growth factor deprivation - Test2 - elhamkhani - TriplyDB

Glucose metabolism attenuates p53 and Puma-dependent cell death upon growth factor deprivation

Glucose metabolism attenuates p53 and Puma-dependent cell death upon growth factor deprivation

http://www.wikidata.org/entity/Q24643422

about

The Warburg effect revisited--lesson from the Sertoli cell Death on the slopes. Symposium on Cell Death Pathways.Myeloid progenitor cells lacking p53 exhibit delayed up-regulation of Puma and prolonged survival after cytokine deprivation.PUMA decreases the growth of prostate cancer PC-3 cells independent of p53.Akt and c-Myc differentially activate cellular metabolic programs and prime cells to bioenergetic inhibition.Modulation of glucose transporter 1 (GLUT1) expression levels alters mouse mammary tumor cell growth in vitro and in vivo.A low-carb diet kills tumor cells with a mutant p53 tumor suppressor gene: the Atkins diet suppresses tumor growth Metabolic regulation of Drosophila apoptosis through inhibitory phosphorylation of Dronc.p53-Dependent transcriptional responses to interleukin-3 signaling High glucose dephosphorylates serine 46 and inhibits p53 apoptotic activity IL-7 is essential for homeostatic control of T cell metabolism in vivo The apoptotic function analysis of p53, Apaf1, Caspase3 and Caspase7 during the spermatogenesis of the Chinese fire-bellied newt Cynops orientalis.Cell metabolism: an essential link between cell growth and apoptosis Akt requires glucose metabolism to suppress puma expression and prevent apoptosis of leukemic T cells.Phosphorylation of Tip60 by GSK-3 determines the induction of PUMA and apoptosis by p53 Roles of glucose in photoreceptor survival.Inhibition of an NAD⁺ salvage pathway provides efficient and selective toxicity to human pluripotent stem cells.Cancer metabolism: current perspectives and future directions.Ribavirin Inhibits the Activity of mTOR/eIF4E, ERK/Mnk1/eIF4E Signaling Pathway and Synergizes with Tyrosine Kinase Inhibitor Imatinib to Impair Bcr-Abl Mediated Proliferation and Apoptosis in Ph+ Leukemia.Genetic variants of a BH3-only pro-apoptotic gene, PUMA, and risk of HPV16-associated squamous cell carcinoma of the head and neck Metabolic stress in autophagy and cell death pathways Minor cell-death defects but reduced tumor latency in mice lacking the BH3-only proteins Bad and Bmf.Matched and mismatched metabolic fuels in lymphocyte function.PUMA and BIM are required for oncogene inactivation-induced apoptosis.Akt1 is the principal Akt isoform regulating apoptosis in limiting cytokine concentrations.Activated lymphocytes as a metabolic model for carcinogenesis.ZnCl2 sustains the adriamycin-induced cell death inhibited by high glucose.New insights into the regulation and function of serine/threonine kinases in T lymphocytes.The metabolic life and times of a T-cell Sugar-free approaches to cancer cell killing.The Role of p53 in Metabolic Regulation Metabolic approaches to overcoming chemoresistance in ovarian cancer.The GM-CSF receptor family: mechanism of activation and implications for disease.BCL-2 family of proteins and mammary cellular fate.The wonders of 2-deoxy-D-glucose.Metabolic control of the proteotoxic stress response: implications in diabetes mellitus and neurodegenerative disorders.Glucose Deprivation Induces ATF4-Mediated Apoptosis through TRAIL Death Receptors.Glucose transporter 1-mediated glucose uptake is limiting for B-cell acute lymphoblastic leukemia anabolic metabolism and resistance to apoptosis.Metabolic Regulation of Apoptosis in Cancer.Cytokine receptor signaling activates an IKK-dependent phosphorylation of PUMA to prevent cell death.

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Glucose metabolism attenuates p53 and Puma-dependent cell death upon growth factor deprivation

http://www.wikidata.org/entity/Q24643422

description

2008 nî lūn-bûn

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2008 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Glucose metabolism attenuates ...... upon growth factor deprivation

@ast

Glucose metabolism attenuates ...... upon growth factor deprivation

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Glucose metabolism attenuates ...... upon growth factor deprivation

@nl

type

label

Glucose metabolism attenuates ...... upon growth factor deprivation

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Glucose metabolism attenuates ...... upon growth factor deprivation

@en

Glucose metabolism attenuates ...... upon growth factor deprivation

@nl

prefLabel

Glucose metabolism attenuates ...... upon growth factor deprivation

@ast

Glucose metabolism attenuates ...... upon growth factor deprivation

@en

Glucose metabolism attenuates ...... upon growth factor deprivation

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Journal of Biological Chemistry

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Glucose metabolism attenuates ...... upon growth factor deprivation

@en

P2093

Emily C Ferguson

http://www.w3.org/2001/XMLSchema#string

Jeffrey C Rathmell

http://www.w3.org/2001/XMLSchema#string

Jonathan L Coloff

http://www.w3.org/2001/XMLSchema#string

Kai Cui

http://www.w3.org/2001/XMLSchema#string

Sarah R Jacobs

http://www.w3.org/2001/XMLSchema#string

Yuxing Zhao

http://www.w3.org/2001/XMLSchema#string

P2860

PUMA induces the rapid apoptosis of colorectal cancer cells

PUMA, a novel proapoptotic gene, is induced by p53

Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function

Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis

Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1

Growth factors can influence cell growth and survival through effects on glucose metabolism

The BH3-only protein Puma plays an essential role in cytokine deprivation induced apoptosis of mast cells

FOXO3a-dependent regulation of Puma in response to cytokine/growth factor withdrawal

Puma cooperates with Bim, the rate-limiting BH3-only protein in cell death during lymphocyte development, in apoptosis induction

On the Origin of Cancer Cells

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36344-53

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scholarly article

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4717755

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10.1074/JBC.M803580200

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52

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283

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2008-12-26T00:00:00Z

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18990690

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2606014

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