Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
about
A new PDAC mouse model originated from iPSCs-converted pancreatic cancer stem cells (CSCcm).Whole-blood RNA transcript-based models can predict clinical response in two large independent clinical studies of patients with advanced melanoma treated with the checkpoint inhibitor, tremelimumab.The Emerging Role of TRAF7 in Tumor Development.Differential impact of RB status on E2F1 reprogramming in human cancer.Identification of novel gene expression signature in lung adenocarcinoma by using next-generation sequencing data and bioinformatics analysis.Promoter methylation of tumor-related genes as a potential biomarker using blood samples for gastric cancer detection.Pathways from senescence to melanoma: focus on MITF sumoylation.Novel endogenous simian retroviral integrations in Vero cells: implications for quality control of a human vaccine cell substrate.MethCNA: a database for integrating genomic and epigenomic data in human cancer.Inhibition of enhancer of zeste homolog 2 increases the expression of p16 and suppresses the proliferation and migration of ovarian carcinoma cells in vitro and in vivo.A novel suicide gene therapy for the treatment of p16Ink4a-overexpressing tumors.Expression profiles analysis identifies a novel three-mRNA signature to predict overall survival in oral squamous cell carcinoma.Prognostic and clinicopathological value of p16 protein aberrant expression in colorectal cancer: A PRISMA-compliant Meta-analysis.Markers of Oral Lichen Planus Malignant Transformation.CDKN2A inhibits formation of homotypic cell-in-cell structures.Metabolism and Epigenetic Interplay in Cancer: Regulation and Putative Therapeutic TargetsMolecular Evolution of a Glioblastoma Controlled With Tumor Treating Fields and Concomitant TemozolomideThe transcriptome of human mammary epithelial cells infected with the HCMV-DB strain displays oncogenic traitsTranslation acrobatics: how cancer cells exploit alternate modes of translational initiationGenetic polymorphisms may influence the vertical growth rate of melanoma
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P2860
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
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2016 nî lūn-bûn
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2016 թուականի Յունիսին հրատարակուած գիտական յօդուած
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2016 թվականի հունիսին հրատարակված գիտական հոդված
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年學術文章
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name
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@ast
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@en
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@nl
type
label
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@ast
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@en
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@nl
prefLabel
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@ast
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@en
Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@nl
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P921
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Implications of Genetic and Epigenetic Alterations of CDKN2A (p16(INK4a)) in Cancer
@en
P2093
Ann M Bode
Bu Young Choi
Mee-Hyun Lee
Zigang Dong
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P3181
P356
10.1016/J.EBIOM.2016.04.017
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P577
2016-06-01T00:00:00Z