Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
about
When Genome Maintenance Goes Badly Awry.The cell cycle restricts activation-induced cytidine deaminase activity to early G1.BRILIA: Integrated Tool for High-Throughput Annotation and Lineage Tree Assembly of B-Cell Repertoires.A transcriptional serenAID: the role of noncoding RNAs in class switch recombination.Functions and Malfunctions of Mammalian DNA-Cytosine DeaminasesR-Loop Depletion by Over-expressed RNase H1 in Mouse B Cells Increases Activation-Induced Deaminase Access to the Transcribed Strand without Altering Frequency of Isotype Switching.Recent advances in the study of immunodeficiency and DNA damage response.Malaria, Epstein-Barr virus infection and the pathogenesis of Burkitt's lymphoma.The IGH locus relocalizes to a "recombination compartment" in the perinucleolar region of differentiating B-lymphocytes.CCCTC-Binding Factor Locks Premature IgH Germline Transcription and Restrains Class Switch Recombination.The Complex Interplay between DNA Injury and Repair in Enzymatically Induced Mutagenesis and DNA Damage in B Lymphocytes.High-risk follicular lymphomas harbour more somatic mutations including those in the AID-motifLiving Organisms Author Their Read-Write Genomes in Evolution.B Cell Intrinsic Mechanisms Constraining IgE Memory.Enzyme cycling contributes to efficient induction of genome mutagenesis by the cytidine deaminase APOBEC3B.Phosphorylation promotes activation-induced cytidine deaminase activity at the Myc oncogene.Efficient Induction of Ig Gene Hypermutation in Ex Vivo-Activated Primary B Cells.BRCT-domain protein BRIT1 influences class switch recombination.AICDA drives epigenetic heterogeneity and accelerates germinal center-derived lymphomagenesis.Predicting AID off-targets: A step forward.Robust immunoglobulin class switch recombination and end joining in Parp9-deficient mice.Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.The H2B deubiquitinase Usp22 promotes antibody class switch recombination by facilitating non-homologous end joining.Chronic Lymphocytic Leukemia B-Cell Normal Cellular Counterpart: Clues From a Functional Perspective.Human Immunodeficiency Virus Tat Protein Aids V Region Somatic Hypermutation in Human B Cells.A licensing step links AID to transcription elongation for mutagenesis in B cells.Transcription-associated events affecting genomic integrity.Differences in genomic abnormalities among African individuals with monoclonal gammopathies using calculated ancestryGenetic and mechanistic basis for APOBEC3H alternative splicing, retrovirus restriction, and counteraction by HIV-1 proteaseBeyond Hot Spots: Biases in Antibody Somatic Hypermutation and Implications for Vaccine Design
P2860
Q37138583-B8B811B6-125C-442B-A7BC-B4D5B574ACF3Q37549772-14868CC4-72E2-4421-AEC9-B8DDA107E6F9Q37588592-EF6E92BC-D9EA-4E17-AF7A-82CD002D9F17Q38680202-4C3A4A7D-AFD6-40C6-865B-20446634A95BQ38943944-035997D8-23A6-4362-B4A3-B5DDD5838355Q39033272-A8F99033-A1DC-4C9C-B910-A9A10DFBB25FQ39333618-FFDAD951-29D6-46C9-B264-4DB824AA4F0CQ40123116-B8C17957-4BD1-48CB-A335-280D845FED61Q41092829-C535B64E-B279-47F2-AFF0-FA1AB0BD1D6BQ41475833-888B38D4-D413-4802-ADE3-BC36BE14A7F6Q41661129-A1EF1FF5-E5A5-4739-823C-724B0EE50A7FQ42630555-D9271424-0642-462E-BF70-5B6FCB588A5AQ46246927-59E407ED-3D17-4C61-A4B5-BCC3C05BA87BQ46253139-40ADA245-F3E1-4D55-8FD4-539E21B78182Q47152216-65A62982-59AB-40EC-BBDE-4494E9346D10Q47419320-1B211FC9-399E-4216-A264-E99B489914B8Q47758514-133F80F0-F1D9-49FF-905C-0F827C358F95Q48099306-ED4EA17E-D5C3-4894-85C4-2109325B0664Q49870129-3DCFC513-5E23-44EA-80BF-CEFDBD87D98BQ49960852-47DFCFBE-6414-4C50-9841-44727BC19B1DQ51180033-6300F5FF-2C1F-455B-A1B6-6FA4B9733A1EQ52321974-5EDD4E69-4B9D-4049-BEEC-1C2A52E0589CQ52362335-02DEBFC1-2E01-4CCC-8C84-C50408578C75Q52579132-C2E3DE36-EC4C-45FC-AC1C-9551024A5F7EQ52582141-CC87985F-6D61-4311-984B-D8D8FA590737Q52723490-9869EF00-6CDA-4207-BA46-FCA35D2FE1EAQ53801893-58D38272-7C32-458E-8909-E88844FF3336Q57282816-71887E42-1D46-42BE-8777-66C5DDF97471Q57295397-F166922F-9721-4944-88FB-CC2B421707DAQ58781552-C8B3D6E9-CB5F-406A-AA47-C637623894C8
P2860
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
description
2016 nî lūn-bûn
@nan
2016 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի մարտին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@ast
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@en
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@nl
type
label
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@ast
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@en
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@nl
prefLabel
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@ast
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@en
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@nl
P2093
P2860
P356
P1476
Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity
@en
P2093
Jayanta Chaudhuri
Rafael Casellas
Uttiya Basu
William T Yewdell
P2860
P304
P356
10.1038/NRI.2016.2
P407
P577
2016-02-22T00:00:00Z