Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells - Test2 - elhamkhani - TriplyDB

Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

http://www.wikidata.org/entity/Q27000436

about

Inhibition of ENaC by endothelin-1 Pressure natriuresis and the renal control of arterial blood pressure Nanomechanics of the endothelial glycocalyx contribute to Na+-induced vascular inflammation.Blockade of ENaCs by amiloride induces c-Fos activation of the area postrema.Pathophysiology and treatment of resistant hypertension: the role of aldosterone and amiloride-sensitive sodium channels Functional Roles of Clusters of Hydrophobic and Polar Residues in the Epithelial Na+ Channel Knuckle Domain.Vasodysfunction That Involves Renal Vasodysfunction, Not Abnormally Increased Renal Retention of Sodium, Accounts for the Initiation of Salt-Induced Hypertension.Dipeptidyl Peptidase-4 Inhibitor Increases Vascular Leakage in Retina through VE-cadherin Phosphorylation.Hypertension: the missing WNKs.Involvement of Epithelial Na+ Channel in the Elevated Myogenic Response in Posterior Cerebral Arteries from Spontaneously Hypertensive Rats.Regulation of the Epithelial Na(+) Channel by Paraoxonase-2.Personalized Therapy of Hypertension: the Past and the Future.ISN Forefronts Symposium 2015: The Evolution of Hypertension-Old Genes, New Concepts.Actions of Quercetin, a Polyphenol, on Blood Pressure.Actions of quercetin, a flavonoid, on ion transporters: its physiological roles.Sodium renders endothelial cells sticky for red blood cells Feedforward activation of endothelial ENaC by high sodium.Association between pre hemodialysis serum sodium concentration and blood pressure: results from a retrospective analysis from the international monitoring dialysis outcomes (MONDO) initiative.Ouabagenin is a naturally occurring LXR ligand without causing hepatic steatosis as a side effect.Shear-mediated platelet activation in the free flow: Perspectives on the emerging spectrum of cell mechanobiological mechanisms mediating cardiovascular implant thrombosis.The Deletion of Endothelial Sodium Channel α (αENaC) Impairs Endothelium-Dependent Vasodilation and Endothelial Barrier Integrity in Endotoxemia in Vivo.

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

http://www.wikidata.org/entity/Q27000436

description

2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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Nature Reviews Nephrology

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Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells

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David G Warnock

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Hans Oberleithner

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Kristina Kusche-Vihrog

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Thomas R Kleyman

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P2860

Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S

COMMD1 downregulates the epithelial sodium channel through Nedd4-2

Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channel

COMMD1 regulates the delta epithelial sodium channel (δENaC) through trafficking and ubiquitination

Grb10 prevents Nedd4-mediated vascular endothelial growth factor receptor-2 degradation

Structure of the acid-sensing ion channel 1 in complex with the gating modifier Psalmotoxin 1

Regulation of stability and function of the epithelial Na+ channel (ENaC) by ubiquitination.

Phosphorylation of Nedd4-2 by Sgk1 regulates epithelial Na(+) channel cell surface expression

WW domains of Nedd4 bind to the proline-rich PY motifs in the epithelial Na+ channel deleted in Liddle's syndrome

Furin at the cutting edge: from protein traffic to embryogenesis and disease

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nrneph.2013.275

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146-157

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Frederic Jaisser

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