Absence of deficits in social behaviors and ultrasonic vocalizations in later generations of mice lacking neuroligin4.
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Mouse Genetic Models of Human Brain DisordersBehavioral phenotypes of genetic mouse models of autism.Sexual attractiveness of male chemicals and vocalizations in mice.Behavioral and Neuroanatomical Phenotypes in Mouse Models of AutismAdvancing the understanding of autism disease mechanisms through geneticsGABAB Receptor Agonist R-Baclofen Reverses Social Deficits and Reduces Repetitive Behavior in Two Mouse Models of AutismTranslational Mouse Models of Autism: Advancing Toward Pharmacological Therapeutics.Recording Mouse Ultrasonic Vocalizations to Evaluate Social Communication.Social communication in mice--are there optimal cage conditions?16p11.2 Deletion Syndrome Mice Display Sensory and Ultrasonic Vocalization Deficits During Social Interactions.Repetitive grooming and sensorimotor abnormalities in an ephrin-A knockout model for Autism Spectrum DisordersBehavioral abnormalities and circuit defects in the basal ganglia of a mouse model of 16p11.2 deletion syndrome.Replicable in vivo physiological and behavioral phenotypes of the Shank3B null mutant mouse model of autism.Overview of mouse models of autism spectrum disordersSynapse assembly and neurodevelopmental disordersSpatial sexual dimorphism of X and Y homolog gene expression in the human central nervous system during early male developmentMale mice emit distinct ultrasonic vocalizations when the female leaves the social interaction arena.Synaptic basis of social dysfunction: a focus on postsynaptic proteins linking group-I mGluRs with AMPARs and NMDARs.Behavioural Phenotypes and Neural Circuit Dysfunctions in Mouse Models of Autism Spectrum Disorder.Determining Ultrasonic Vocalization Preferences in Mice using a Two-choice Playback TestNeuronal overexpression of Ube3a isoform 2 causes behavioral impairments and neuroanatomical pathology relevant to 15q11.2-q13.3 duplication syndrome.Behavioral and neuroanatomical approaches in models of neurodevelopmental disorders: opportunities for translation.Tyro3/Axl/Mertk-deficient mice develop bone marrow edema which is an early pathological marker in rheumatoid arthritis
P2860
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P2860
Absence of deficits in social behaviors and ultrasonic vocalizations in later generations of mice lacking neuroligin4.
description
2012 nî lūn-bûn
@nan
2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Absence of deficits in social ...... ns of mice lacking neuroligin4
@nl
Absence of deficits in social ...... s of mice lacking neuroligin4.
@ast
Absence of deficits in social ...... s of mice lacking neuroligin4.
@en
type
label
Absence of deficits in social ...... ns of mice lacking neuroligin4
@nl
Absence of deficits in social ...... s of mice lacking neuroligin4.
@ast
Absence of deficits in social ...... s of mice lacking neuroligin4.
@en
prefLabel
Absence of deficits in social ...... ns of mice lacking neuroligin4
@nl
Absence of deficits in social ...... s of mice lacking neuroligin4.
@ast
Absence of deficits in social ...... s of mice lacking neuroligin4.
@en
P2093
P2860
P50
P3181
P1476
Absence of deficits in social ...... s of mice lacking neuroligin4.
@en
P2093
A-M Le Sourd
C S Leblond
J L Silverman
J N Crawley
L Woldeyohannes
P2860
P304
P3181
P356
10.1111/J.1601-183X.2012.00849.X
P407
P577
2012-10-10T00:00:00Z