Three dimensions of the amyloid hypothesis: time, space and 'wingmen' - Test2 - elhamkhani - TriplyDB

Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Stepping closer to treating Alzheimer's disease patients with BACE1 inhibitor drugs The Role of Amyloid-β Oligomers in Toxicity, Propagation, and Immunotherapy Growing up in a Bubble: Using Germ-Free Animals to Assess the Influence of the Gut Microbiota on Brain and Behavior Expanding the Repertoire of Biomarkers for Alzheimer's Disease: Targeted and Non-targeted Approaches Amyloid-β Receptors: The Good, the Bad, and the Prion Protein Aβ Clearance, "hub" of Multiple Deficiencies Leading to Alzheimer Disease β-Amyloid: the key peptide in the pathogenesis of Alzheimer's disease Impaired vascular-mediated clearance of brain amyloid beta in Alzheimer's disease: the role, regulation and restoration of LRP1 Towards Personalized Intervention for Alzheimer's Disease Tau-mediated synaptic damage in Alzheimer's disease Neutral Sphingomyelinase-2 Deficiency Ameliorates Alzheimer's Disease Pathology and Improves Cognition in the 5XFAD Mouse Anti-Viral Properties of Amyloid-β Peptides.Truncating mutations in APP cause a distinct neurological phenotype.Preparation of pure populations of covalently stabilized amyloid β-protein oligomers of specific sizes.Glial contributions to neurodegeneration in tauopathies.Pioglitazone ameliorates Aβ42 deposition in rats with diet-induced insulin resistance associated with AKT/GSK3β activation Role of Pro-Inflammatory Cytokines and Vitamin D in Probable Alzheimer's Disease with Depression.Interplay of histidine residues of the Alzheimer's disease Aβ peptide governs its Zn-induced oligomerization.Tau and Aβ imaging, CSF measures, and cognition in Alzheimer's disease Specific Binding of Cu(II) Ions to Amyloid-Beta Peptides Bound to Aggregation-Inhibiting Molecules or SDS Micelles Creates Complexes that Generate Radical Oxygen Species.Small molecule LX2343 ameliorates cognitive deficits in AD model mice by targeting both amyloid β production and clearance.Determining composition of micron-scale protein deposits in neurodegenerative disease by spatially targeted optical microproteomics.Metabolic and Non-Cognitive Manifestations of Alzheimer's Disease: The Hypothalamus as Both Culprit and Target of Pathology Magnetite-Amyloid-β deteriorates activity and functional organization in an in vitro model for Alzheimer's disease.A novel pathway for amyloids self-assembly in aggregates at nanomolar concentration mediated by the interaction with surfaces.Cerebrospinal fluid ratios with Aβ42 predict preclinical brain β-amyloid accumulation.Chronic Toxoplasma gondii infection enhances β-amyloid phagocytosis and clearance by recruited monocytes.The incubation period of Alzheimer's disease and the timing of tau versus amyloid misfolding and spreading within the brain.Adipocyte-derived factors in age-related dementia and their contribution to vascular and Alzheimer pathology APP Receptor? To Be or Not To Be.Leptin Dysfunction and Alzheimer's Disease: Evidence from Cellular, Animal, and Human Studies.Rapid in vivo measurement of β-amyloid reveals biphasic clearance kinetics in an Alzheimer's mouse model.Loss of presenilin function is associated with a selective gain of APP function.The New Criteria for Alzheimer's Disease - Implications for Geriatricians Immunization with Small Amyloid-β-derived Cyclopeptide Conjugates Diminishes Amyloid-β-Induced Neurodegeneration in Mice The ability of apolipoprotein E fragments to promote intraneuronal accumulation of amyloid beta peptide 42 is both isoform and size-specific.Changes in insulin and insulin signaling in Alzheimer's disease: cause or consequence?Genome-wide interaction study of brain beta-amyloid burden and cognitive impairment in Alzheimer's disease.Fluoxetine Prevents Aβ1-42-Induced Toxicity via a Paracrine Signaling Mediated by Transforming-Growth-Factor-β1.Improvement of Electroacupuncture on APP/PS1 Transgenic Mice in Spatial Learning and Memory Probably due to Expression of Aβ and LRP1 in Hippocampus.

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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description

2015 nî lūn-bûn

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2015 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2015 թվականի հունիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Nature Neuroscience

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Three dimensions of the amyloid hypothesis: time, space and 'wingmen'

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Erik S Musiek

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P2860

The importance of neuritic plaques and tangles to the development and evolution of AD

A mutation in APP protects against Alzheimer’s disease and age-related cognitive decline

The gamma -secretase-cleaved C-terminal fragment of amyloid precursor protein mediates signaling to the nucleus

Gene transfer of human Apoe isoforms results in differential modulation of amyloid deposition and neurotoxicity in mouse brain

Aging as an event of proteostasis collapse

Amyloid-beta dynamics are regulated by orexin and the sleep-wake cycle

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Neuropathological alterations in Alzheimer disease

Gene dose of apolipoprotein E type 4 allele and the risk of Alzheimer's disease in late onset families

Neuropathological stageing of Alzheimer-related changes

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800-6

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10.1038/NN.4018

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6

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David M. Holtzman

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