CaMKII regulates the frequency-response function of hippocampal synapses for the production of both LTD and LTP
about
Long-Term Potentiation and MemoryRegulation of the multifunctional Ca2+/calmodulin-dependent protein kinase II by the PP2C phosphatase PPM1F in fibroblastsInteractive cloning with the SH3 domain of N-src identifies a new brain specific ion channel protein, with homology to eag and cyclic nucleotide-gated channelsNatural patterns of activity and long-term synaptic plasticityRescuing impairment of long-term potentiation in fyn-deficient mice by introducing Fyn transgeneFunctional fluorescent Ca2+ indicator proteins in transgenic mice under TET controlThe Emergence of NMDA Receptor Metabotropic Function: Insights from ImagingNon-ionotropic signaling by the NMDA receptor: controversy and opportunityIn search of a recognition memory engramThe role of growth retardation in lasting effects of neonatal dexamethasone treatment on hippocampal synaptic functionGSK-3β dysregulation contributes to parkinson's-like pathophysiology with associated region-specific phosphorylation and accumulation of tau and α-synucleinIdentification of a phosphorylation site for calcium/calmodulindependent protein kinase II in the NR2B subunit of the N-methyl-D-aspartate receptorEssential function of alpha-calcium/calmodulin-dependent protein kinase II in neurotransmitter release at a glutamatergic central synapse.Chemogenetic inactivation of ventral hippocampal glutamatergic neurons disrupts consolidation of contextual fear memory.Reversible disruption of pre-pulse inhibition in hypomorphic-inducible and reversible CB1-/- mice.Age-associated changes in Ca(2+)-dependent processes: relation to hippocampal synaptic plasticity.Animal models of gene-environment interactions in schizophrenia.Transgenic up-regulation of alpha-CaMKII in forebrain leads to increased anxiety-like behaviors and aggression.Inducible and selective erasure of memories in the mouse brain via chemical-genetic manipulation.Modulation of presynaptic plasticity and learning by the H-ras/extracellular signal-regulated kinase/synapsin I signaling pathwayGenetic evidence for a role for protein kinase A in the maintenance of sleep and thalamocortical oscillations.Maturation of GABAergic inhibition promotes strengthening of temporally coherent inputs among convergent pathwaysViagra for your synapses: Enhancement of hippocampal long-term potentiation by activation of beta-adrenergic receptors.A role for prefrontal calcium-sensitive protein phosphatase and kinase activities in working memory.Calcium/calmodulin-dependent kinase II and long-term potentiation enhance synaptic transmission by the same mechanism.Heterosynaptic metaplasticity in the hippocampus in vivo: a BCM-like modifiable threshold for LTP.Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylationA mechanism for synaptic frequency detection through autophosphorylation of CaM kinase II.Cellular signaling through multifunctional Ca2+/calmodulin-dependent protein kinase II.The h current is a candidate mechanism for regulating the sliding modification threshold in a BCM-like synaptic learning rule.Tau filament formation and associative memory deficit in aged mice expressing mutant (R406W) human tau.Neurochondrin negatively regulates CaMKII phosphorylation, and nervous system-specific gene disruption results in epileptic seizure.Theory for normal and impaired experience-dependent plasticity in neocortex of adult ratsCre-Lox Neurogenetics: 20 Years of Versatile Applications in Brain Research and Counting…A fresh look at the role of CaMKII in hippocampal synaptic plasticity and memory.Structure-function of the multifunctional Ca2+/calmodulin-dependent protein kinase II.Calcium/calmodulin-dependent protein kinase II regulates Caenorhabditis elegans locomotion in concert with a G(o)/G(q) signaling network.Mice transgenically overexpressing sulfonylurea receptor 1 in forebrain resist seizure induction and excitotoxic neuron death.Long-Term Potentiation at CA3-CA1 Hippocampal Synapses with Special Emphasis on Aging, Disease, and Stress.CA1 long-term potentiation is diminished but present in hippocampal slices from alpha-CaMKII mutant mice
P2860
Q22337020-2F46CB6B-B2B5-4CCD-9E59-48271C40FF80Q24292890-E7AD2276-7D19-4337-8710-3DD0E0AD2008Q24315270-D07718B3-55B6-4286-ACF2-D4B3A6F9E486Q24616717-8BD075D3-BC86-4B7D-9269-D8BDC6080F6EQ24676918-7B3FB68A-98F9-4D9E-B5DB-F49A1A8E355EQ24793235-7BD92339-3DFE-4431-AFDC-31B9D8E11743Q26741815-FAC64411-DDEA-4446-952B-24F84B153FE4Q26745925-42BE6A75-25A9-4E59-80FA-80540CAF7EDAQ27010528-12AC429B-07BE-4926-A086-92DB9DE28CE1Q27320030-DD884F63-5EA4-4412-A240-DA92A902244FQ27331743-EAE90051-3602-4EBA-8138-F2A6E203031CQ28298139-E78436BB-16C2-492C-83D7-645665C37BA8Q28593667-F09C569B-BBBD-4582-BF07-9DB1BB0527F9Q30407997-8C6F8DDF-BBD4-4729-9870-ACEEAEE1E5B5Q30467877-D30860C4-BAE0-4C57-9106-6DA6F009D8C1Q30471604-600FC4D5-EABF-459D-A9EA-478FB5B51007Q30478272-56327EFF-544D-450B-9016-9A4DD1F95717Q30486646-E8B48DAB-1AE1-4F16-997E-031F5BE48FFCQ30497032-7FF0FCE9-C825-4A01-B0DF-5E99B219210DQ33573694-4E57C136-088B-4094-A1F9-B223E472E0BAQ33573836-48B081E3-C3F6-42C3-8F77-3C3ED351AFC0Q33598580-759C7249-C1E3-4D35-A410-61ED8A8970A1Q33678921-22FCC5D4-84E3-4553-8111-A8C7C27984C2Q33736098-13BEAE80-2CFC-480B-9F3B-60C62E2E977AQ33762318-62556B40-87B8-488F-B6D3-D50EA3D509B1Q33943960-87B8020B-BBCE-40A8-B499-A2611D419B70Q33991390-D9358189-488A-4023-A054-D4B66EFD321EQ34017834-B6B1E4B6-93DD-4A19-A251-44DB08439CC9Q34092385-682D6472-A3D1-4815-BC8C-D7E279D88E01Q34108299-6D4D016C-B246-4AE4-8FE9-CC937AE8FBCBQ34162119-DC703384-D245-4DA9-BD87-7CBB911019A9Q34406227-A789677C-833E-44B1-A2FD-915CC1B791B2Q34495610-1BCF8FE7-E870-44F8-8DC3-45937C5BDDCFQ34516151-5A5D9ABF-0557-4955-8EFC-C4356F815DD1Q34561492-71901219-EF95-4ED3-B22B-F1278045164FQ34586783-301F86E4-EFB2-4FB1-9C6C-EFCEE63E8474Q34610776-A4643467-77CA-4A98-B195-7D6385DD9B14Q34749138-3EED771E-066C-41B0-9D15-C6C589507D6AQ35007766-E0A5E393-EAF9-4C9B-9F4A-E8F73D102B4CQ35039016-7377A38A-7FBC-413C-B09B-07CAE10D8E94
P2860
CaMKII regulates the frequency-response function of hippocampal synapses for the production of both LTD and LTP
description
1995 nî lūn-bûn
@nan
1995 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
name
CaMKII regulates the frequency ...... production of both LTD and LTP
@ast
CaMKII regulates the frequency ...... production of both LTD and LTP
@en
CaMKII regulates the frequency ...... production of both LTD and LTP
@nl
type
label
CaMKII regulates the frequency ...... production of both LTD and LTP
@ast
CaMKII regulates the frequency ...... production of both LTD and LTP
@en
CaMKII regulates the frequency ...... production of both LTD and LTP
@nl
prefLabel
CaMKII regulates the frequency ...... production of both LTD and LTP
@ast
CaMKII regulates the frequency ...... production of both LTD and LTP
@en
CaMKII regulates the frequency ...... production of both LTD and LTP
@nl
P2093
P3181
P1433
P1476
CaMKII regulates the frequency ...... production of both LTD and LTP
@en
P2093
P304
P3181
P356
10.1016/0092-8674(95)90009-8
P407
P577
1995-06-16T00:00:00Z