Calcium/calmodulin-dependent kinase II and long-term potentiation enhance synaptic transmission by the same mechanism. - Test2 - elhamkhani - TriplyDB

Calcium/calmodulin-dependent kinase II and long-term potentiation enhance synaptic transmission by the same mechanism.

Calcium/calmodulin-dependent kinase II and long-term potentiation enhance synaptic transmission by the same mechanism.

http://www.wikidata.org/entity/Q33762318

about

Regulation of excitatory synapses and fearful memories by stress hormones Calcium input frequency, duration and amplitude differentially modulate the relative activation of calcineurin and CaMKII Long-Term Potentiation and Memory Calmodulin-kinases: modulators of neuronal development and plasticity Optical induction of plasticity at single synapses reveals input-specific accumulation of alphaCaMKII Characterization of a calmodulin kinase II inhibitor protein in brain Ca2+/calmodulin-kinase II enhances channel conductance of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate type glutamate receptors Adducin is an in vivo substrate for protein kinase C: phosphorylation in the MARCKS-related domain inhibits activity in promoting spectrin-actin complexes and occurs in many cells, including dendritic spines of neurons Long-term potentiation in bone--a role for glutamate in strain-induced cellular memory?The stability of a stochastic CaMKII switch: dependence on the number of enzyme molecules and protein turnover Barriers in the brain: resolving dendritic spine morphology and compartmentalization AMPA receptor regulation during synaptic plasticity in hippocampus and neocortex Mechanisms of CaMKII action in long-term potentiation Protein kinase Mzeta is necessary and sufficient for LTP maintenance Roles of CaMKII, PKA, and PKC in the induction and maintenance of LTP of C-fiber-evoked field potentials in rat spinal dorsal horn Translocation of autophosphorylated calcium/calmodulin-dependent protein kinase II to the postsynaptic density Synaptic plasticity: multiple forms, functions, and mechanisms Identification of the Ca2+/calmodulin-dependent protein kinase II regulatory phosphorylation site in the alpha-amino-3-hydroxyl-5-methyl-4-isoxazole-propionate-type glutamate receptor The effects of NR2 subunit-dependent NMDA receptor kinetics on synaptic transmission and CaMKII activation Alpha-synuclein involvement in hippocampal synaptic plasticity: role of NO, cGMP, cGK and CaMKII Severely impaired learning and altered neuronal morphology in mice lacking NMDA receptors in medium spiny neurons.Stratum radiatum giant cells: a type of principal cell in the rat hippocampus.Progress in understanding the factors regulating reversibility of long-term potentiation.The missing piece in the 'use it or lose it' puzzle: is inhibition regulated by activity or does it act on its own accord?Silent synapses and the emergence of a postsynaptic mechanism for LTP.The dense core vesicle protein IA-2, but not IA-2β, is required for active avoidance learning.Lobe specific Ca2+-calmodulin nano-domain in neuronal spines: a single molecule level analysis.Acute stress induces contrasting changes in AMPA receptor subunit phosphorylation within the prefrontal cortex, amygdala and hippocampus The effect of noise on CaMKII activation in a dendritic spine during LTP induction Synaptic plasticity and dynamic modulation of the postsynaptic membrane.Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylation Calcium/calmodulin-dependent protein kinase II mediates hippocampal glutamatergic plasticity during benzodiazepine withdrawal.CaMKII control of spine size and synaptic strength: role of phosphorylation states and nonenzymatic action.Neuronal SUMOylation: mechanisms, physiology, and roles in neuronal dysfunction.Genetic inhibition of CaMKII in dorsal striatal medium spiny neurons reduces functional excitatory synapses and enhances intrinsic excitability.Phosphorylation of the alpha-amino-3-hydroxy-5-methylisoxazole4-propionic acid receptor GluR1 subunit by calcium/calmodulin-dependent kinase II.Structure-function of the multifunctional Ca2+/calmodulin-dependent protein kinase II.Stimulation of the sigma-1 receptor by DHEA enhances synaptic efficacy and neurogenesis in the hippocampal dentate gyrus of olfactory bulbectomized mice Ca2+/calmodulin-dependent protein kinase II inhibitors disrupt AKAP79-dependent PKC signaling to GluA1 AMPA receptors.Glutamate receptor trafficking in synaptic plasticity.

P2860

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P2860

Calcium/calmodulin-dependent kinase II and long-term potentiation enhance synaptic transmission by the same mechanism.

http://www.wikidata.org/entity/Q33762318

description

1995 nî lūn-bûn

@nan

1995 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

1995 թվականի նոյեմբերին հրատարակված գիտական հոդված

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年论文

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name

Calcium/calmodulin-dependent k ...... mission by the same mechanism.

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Calcium/calmodulin-dependent k ...... mission by the same mechanism.

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Calcium/calmodulin-dependent k ...... mission by the same mechanism.

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Calcium/calmodulin-dependent k ...... mission by the same mechanism.

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prefLabel

Calcium/calmodulin-dependent k ...... mission by the same mechanism.

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Calcium/calmodulin-dependent k ...... mission by the same mechanism.

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PNAS.92.24.11175

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Calcium/calmodulin-dependent k ...... mission by the same mechanism.

@en

P2093

G O Hjelmstad

http://www.w3.org/2001/XMLSchema#string

P M Lledo

http://www.w3.org/2001/XMLSchema#string

R A Nicoll

http://www.w3.org/2001/XMLSchema#string

R C Malenka

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S Mukherji

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T R Soderling

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P2860

A synaptic model of memory: long-term potentiation in the hippocampus

Identification of a Ca2+/calmodulin-dependent protein kinase II regulatory phosphorylation site in non-N-methyl-D-aspartate glutamate receptors

Deficient hippocampal long-term potentiation in alpha-calcium-calmodulin kinase II mutant mice

The CaM kinase II hypothesis for the storage of synaptic memory

CaMKII regulates the frequency-response function of hippocampal synapses for the production of both LTD and LTP

Inhibition of postsynaptic PKC or CaMKII blocks induction but not expression of LTP

Evidence for silent synapses: implications for the expression of LTP.

Activation of postsynaptically silent synapses during pairing-induced LTP in CA1 region of hippocampal slice.

Long-term potentiation is associated with an increased activity of Ca2+/calmodulin-dependent protein kinase II.

Specificity of protein kinase inhibitor peptides and induction of long-term potentiation

P304

11175-11179

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scholarly article

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10.1073/PNAS.92.24.11175

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24

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92

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1995-11-01T00:00:00Z

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15719061

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7479960

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P932

40594

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