Neuronal Abelson helper integration site-1 (Ahi1) deficiency in mice alters TrkB signaling with a depressive phenotype.
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The Joubert syndrome-associated missense mutation (V443D) in the Abelson-helper integration site 1 (AHI1) protein alters its localization and protein-protein interactionsHuntingtin-associated protein 1 regulates postnatal neurogenesis and neurotrophin receptor sortingLoss of Ahi1 impairs neurotransmitter release and causes depressive behaviors in miceLoss of Ahi1 affects early development by impairing BM88/Cend1-mediated neuronal differentiationNeural mechanisms underlying stress resilience in Ahi1 knockout mice: relevance to neuropsychiatric disordersNeuroinformatic analyses of common and distinct genetic components associated with major neuropsychiatric disordersAHI-1: a novel signaling protein and potential therapeutic target in human leukemia and brain disorders.Alterations in the expression of a neurodevelopmental gene exert long-lasting effects on cognitive-emotional phenotypes and functional brain networks: translational evidence from the stress-resilient Ahi1 knockout mouse.Avoiding mouse traps in schizophrenia genetics: lessons and promises from current and emerging mouse models.Abelson helper integration site-1 gene variants on major depressive disorder and bipolar disorderThe influence of AHI1 variants on the diagnosis and treatment outcome in schizophreniaPostnatal loss of hap1 reduces hippocampal neurogenesis and causes adult depressive-like behavior in mice.Low-Level Laser Irradiation Improves Depression-Like Behaviors in Mice.Progress in multiple sclerosis genetics.Endosomal system genetics and autism spectrum disorders: A literature review.Zfp462 deficiency causes anxiety-like behaviors with excessive self-grooming in mice.Fisetin provides antidepressant effects by activating the tropomyosin receptor kinase B signal pathway in mice.Fibroblast growth factor 4 is required but not sufficient for the astrocyte dedifferentiation.Tyrosine hydroxylase down-regulation after loss of Abelson helper integration site 1 (AHI1) promotes depression via the circadian clock pathway in mice.The multiple sclerosis risk allele within the AHI1 gene is associated with relapses in children and adults.Loss of Tctn3 causes neuronal apoptosis and neural tube defects in mice.
P2860
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P2860
Neuronal Abelson helper integration site-1 (Ahi1) deficiency in mice alters TrkB signaling with a depressive phenotype.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Neuronal Abelson helper integr ...... ng with a depressive phenotype
@nl
Neuronal Abelson helper integr ...... g with a depressive phenotype.
@ast
Neuronal Abelson helper integr ...... g with a depressive phenotype.
@en
type
label
Neuronal Abelson helper integr ...... ng with a depressive phenotype
@nl
Neuronal Abelson helper integr ...... g with a depressive phenotype.
@ast
Neuronal Abelson helper integr ...... g with a depressive phenotype.
@en
prefLabel
Neuronal Abelson helper integr ...... ng with a depressive phenotype
@nl
Neuronal Abelson helper integr ...... g with a depressive phenotype.
@ast
Neuronal Abelson helper integr ...... g with a depressive phenotype.
@en
P2093
P2860
P921
P356
P1476
Neuronal Abelson helper integr ...... ng with a depressive phenotype
@en
P2093
Austin Cape
Xiao-Jiang Li
Yung-Feng Lin
P2860
P304
19126-19131
P356
10.1073/PNAS.1013032107
P407
P577
2010-10-18T00:00:00Z