Phosphorylation of MeCP2 at Serine 80 regulates its chromatin association and neurological function. - Test2 - elhamkhani - TriplyDB

Phosphorylation of MeCP2 at Serine 80 regulates its chromatin association and neurological function.

Phosphorylation of MeCP2 at Serine 80 regulates its chromatin association and neurological function.

http://www.wikidata.org/entity/Q30438464

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Phosphorylation of myofibrillogenesis regulator-1 activates the MAPK signaling pathway and induces proliferation and migration in human breast cancer MCF7 cells Methyl-CpG-binding protein 2 is phosphorylated by homeodomain-interacting protein kinase 2 and contributes to apoptosis MeCP2 and the enigmatic organization of brain chromatin. Implications for depression and cocaine addiction Epigenetic Mechanisms in Developmental Alcohol-Induced Neurobehavioral Deficits MeCP2 post-translational modifications: a mechanism to control its involvement in synaptic plasticity and homeostasis?The impact of MeCP2 loss- or gain-of-function on synaptic plasticity Transcriptional co-repressors and memory storage BDNF deregulation in Rett syndrome Role of mecp2 in experience-dependent epigenetic programming The histone H3K36 demethylase Rph1/KDM4 regulates the expression of the photoreactivation gene PHR1 Rett syndrome - biological pathways leading from MECP2 to disorder phenotypes A review of Rett syndrome (RTT) with induced pluripotent stem cells Crh and Oprm1 mediate anxiety-related behavior and social approach in a mouse model of MECP2 duplication syndrome What we know and would like to know about CDKL5 and its involvement in epileptic encephalopathy Exercise impacts brain-derived neurotrophic factor plasticity by engaging mechanisms of epigenetic regulation Epigenetic suppression of neuroligin 1 underlies amyloid-induced memory deficiency Calcium-dependent dephosphorylation of the histone chaperone DAXX regulates H3.3 loading and transcription upon neuronal activation MeCP2 deficiency results in robust Rett-like behavioural and motor deficits in male and female rats SUMOylation of MeCP2 is essential for transcriptional repression and hippocampal synapse development Methyl-CpG binding protein 2 (MeCP2) localizes at the centrosome and is required for proper mitotic spindle organization A model for neural development and treatment of Rett syndrome using human induced pluripotent stem cells Female Mecp2(+/-) mice display robust behavioral deficits on two different genetic backgrounds providing a framework for pre-clinical studies.Beyond Widespread Mecp2 Deletions to Model Rett Syndrome: Conditional Spatio-Temporal Knockout, Single-Point Mutations and Transgenic Rescue Mice MeCP2 is required for activity-dependent refinement of olfactory circuits.Crosstalk in inflammation: the interplay of glucocorticoid receptor-based mechanisms and kinases and phosphatases Cross talk between microRNA and epigenetic regulation in adult neurogenesis.Epigenetic regulation of BDNF expression via the scaffolding protein RACK1.Affinity-based enrichment strategies to assay methyl-CpG binding activity and DNA methylation in early Xenopus embryos.Quantification of O-glycosylation stoichiometry and dynamics using resolvable mass tags.Is X-linked methyl-CpG binding protein 2 a new target for the treatment of Parkinson's disease DNA methylation reader MECP2: cell type- and differentiation stage-specific protein distribution.MeCP2 mutation results in compartment-specific reductions in dendritic branching and spine density in layer 5 motor cortical neurons of YFP-H mice.Calcium signaling in synapse-to-nucleus communication Genome-wide analysis reveals methyl-CpG-binding protein 2-dependent regulation of microRNAs in a mouse model of Rett syndrome Elevated IKKα accelerates the differentiation of human neuronal progenitor cells and induces MeCP2-dependent BDNF expression.Acetyl-L-carnitine improves behavior and dendritic morphology in a mouse model of Rett syndrome.Fetal alcohol exposure alters proopiomelanocortin gene expression and hypothalamic-pituitary-adrenal axis function via increasing MeCP2 expression in the hypothalamus.Epigenetic mechanisms in cognition.Synaptic determinants of rett syndrome.Epigenetic programming of hypoxic-ischemic encephalopathy in response to fetal hypoxia.

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Phosphorylation of MeCP2 at Serine 80 regulates its chromatin association and neurological function.

http://www.wikidata.org/entity/Q30438464

description

2009 nî lūn-bûn

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2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2009 թվականի փետրվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Phosphorylation of MeCP2 at Se ...... ion and neurological function.

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Phosphorylation of MeCP2 at Se ...... ion and neurological function.

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type

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Phosphorylation of MeCP2 at Se ...... ion and neurological function.

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Phosphorylation of MeCP2 at Se ...... ion and neurological function.

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Phosphorylation of MeCP2 at Se ...... ion and neurological function.

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Phosphorylation of MeCP2 at Se ...... ion and neurological function.

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PNAS.0811648106

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Proceedings of the National Academy of Sciences of the United States of America

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Phosphorylation of MeCP2 at Se ...... tion and neurological function

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Carolyn Schanen

http://www.w3.org/2001/XMLSchema#string

Jifang Tao

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Qiang Chang

http://www.w3.org/2001/XMLSchema#string

Robert J Klose

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Rudolf Jaenisch

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Weidong Wang

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Yi Eve Sun

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P2860

Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2

Calmodulin-kinases: modulators of neuronal development and plasticity

Transcriptional repression by the methyl-CpG-binding protein MeCP2 involves a histone deacetylase complex

Reduced cortical activity due to a shift in the balance between excitation and inhibition in a mouse model of Rett syndrome

MeCP2, a key contributor to neurological disease, activates and represses transcription

CaM-kinases: modulators of synaptic plasticity

Chromatin compaction by human MeCP2. Assembly of novel secondary chromatin structures in the absence of DNA methylation

Alpha-CaMKII-dependent plasticity in the cortex is required for permanent memory

CBP histone acetyltransferase activity is a critical component of memory consolidation

A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome

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12

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106

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Nicholas E Sherman

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24023102

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phosphorylation

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