WRN regulates pathway choice between classical and alternative non-homologous end joining - Test2 - elhamkhani - TriplyDB

WRN regulates pathway choice between classical and alternative non-homologous end joining

WRN regulates pathway choice between classical and alternative non-homologous end joining

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Recent Advances in Understanding Werner Syndrome.Role of Protein Linked DNA Breaks in Cancer.Cell cycle-dependent phosphorylation regulates RECQL4 pathway choice and ubiquitination in DNA double-strand break repair.Preserving Salivary Gland Physiology against Genotoxic Damage - The Tousled Way.How cells ensure correct repair of DNA double-strand breaks.The MRE11-RAD50-NBS1 Complex Conducts the Orchestration of Damage Signaling and Outcomes to Stress in DNA Replication and Repair.RecQ helicases in the malaria parasite Plasmodium falciparum affect genome stability, gene expression patterns and DNA replication dynamics Rare Genetic Diseases with Defects in DNA Repair: Opportunities and Challenges in Orphan Drug Development for Targeted Cancer Therapy Cerebral Haemorrhage in a Young Patient With Atypical Werner Syndrome Due to Mutations in

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WRN regulates pathway choice between classical and alternative non-homologous end joining

http://www.wikidata.org/entity/Q30831557

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2016 nî lūn-bûn

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2016 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2016 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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name

WRN regulates pathway choice between classical and alternative non-homologous end joining

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WRN regulates pathway choice between classical and alternative non-homologous end joining

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type

label

WRN regulates pathway choice between classical and alternative non-homologous end joining

@ast

WRN regulates pathway choice between classical and alternative non-homologous end joining

@en

prefLabel

WRN regulates pathway choice between classical and alternative non-homologous end joining

@ast

WRN regulates pathway choice between classical and alternative non-homologous end joining

@en

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Nature Communications

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WRN regulates pathway choice between classical and alternative non-homologous end joining

@en

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Deborah L Croteau

http://www.w3.org/2001/XMLSchema#string

Huiming Lu

http://www.w3.org/2001/XMLSchema#string

Jane Tian

http://www.w3.org/2001/XMLSchema#string

Jessica K de Freitas

http://www.w3.org/2001/XMLSchema#string

Raghavendra A Shamanna

http://www.w3.org/2001/XMLSchema#string

Vilhelm A Bohr

http://www.w3.org/2001/XMLSchema#string

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Alternative-NHEJ is a mechanistically distinct pathway of mammalian chromosome break repair

Ku complex interacts with and stimulates the Werner protein

Telomere-binding protein TRF2 binds to and stimulates the Werner and Bloom syndrome helicases

WRN exonuclease structure and molecular mechanism imply an editing role in DNA end processing

Mutator phenotype of Werner syndrome is characterized by extensive deletions

Up-regulation of WRN and DNA ligase IIIalpha in chronic myeloid leukemia: consequences for the repair of DNA double-strand breaks

Human CtIP promotes DNA end resection

Biochemical evidence for Ku-independent backup pathways of NHEJ

The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations

Recruitment and activation of the ATM kinase in the absence of DNA-damage sensors.

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13785

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scholarly article

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10.1038/NCOMMS13785

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7

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2016-12-06T00:00:00Z

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1022536137

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27922005

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5150655

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