Alternative-NHEJ is a mechanistically distinct pathway of mammalian chromosome break repair - Test2 - elhamkhani - TriplyDB

Alternative-NHEJ is a mechanistically distinct pathway of mammalian chromosome break repair

Alternative-NHEJ is a mechanistically distinct pathway of mammalian chromosome break repair

http://www.wikidata.org/entity/Q21145054

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Alternative end-joining mechanisms: a historical perspective DNA ligase III promotes alternative nonhomologous end-joining during chromosomal translocation formation Ku regulates the non-homologous end joining pathway choice of DNA double-strand break repair in human somatic cells The proteasomal de-ubiquitinating enzyme POH1 promotes the double-strand DNA break response DNA ligase III and DNA ligase IV carry out genetically distinct forms of end joining in human somatic cells Homologous-recombination-deficient tumours are dependent on Polθ-mediated repair Mechanism of microhomology-mediated end-joining promoted by human DNA polymerase θ A dual role for UVRAG in maintaining chromosomal stability independent of autophagy PIAS3 promotes homology‑directed repair and distal non‑homologous end joining Telomere dysfunction and chromosome instability Involvement of nucleotide excision and mismatch repair mechanisms in double strand break repair Novel Biological Approaches for Testing the Contributions of Single DSBs and DSB Clusters to the Biological Effects of High LET Radiation Physiology of the read-write genome DNA double-strand break repair pathway choice and cancer Replication protein A prevents promiscuous annealing between short sequence homologies: Implications for genome integrity Involvement of p53 in the repair of DNA double strand breaks: multifaceted Roles of p53 in homologous recombination repair (HRR) and non-homologous end joining (NHEJ)Sources of DNA double-strand breaks and models of recombinational DNA repair Repair of strand breaks by homologous recombination DNA repair endonuclease ERCC1-XPF as a novel therapeutic target to overcome chemoresistance in cancer therapy BRG1 promotes the repair of DNA double-strand breaks by facilitating the replacement of RPA with RAD51.Human Ku70/80 protein blocks exonuclease 1-mediated DNA resection in the presence of human Mre11 or Mre11/Rad50 protein complex.Repair of DNA Double-Strand Breaks in Heterochromatin DNA polymerase θ (POLQ), double-strand break repair, and cancer Balancing self-renewal against genome preservation in stem cells: How do they manage to have the cake and eat it too?Replication fork instability and the consequences of fork collisions from rereplication Noncanonical views of homology-directed DNA repair Microhomology-Mediated End Joining: A Back-up Survival Mechanism or Dedicated Pathway?Cullin3-KLHL15 ubiquitin ligase mediates CtIP protein turnover to fine-tune DNA-end resection Homology and enzymatic requirements of microhomology-dependent alternative end joining miR-155 Overexpression Promotes Genomic Instability by Reducing High-fidelity Polymerase Delta Expression and Activating Error-Prone DSB Repair RAD52 inactivation is synthetically lethal with deficiencies in BRCA1 and PALB2 in addition to BRCA2 through RAD51-mediated homologous recombination ATM limits incorrect end utilization during non-homologous end joining of multiple chromosome breaks Mechanism of suppression of chromosomal instability by DNA polymerase POLQ The HSV-1 exonuclease, UL12, stimulates recombination by a single strand annealing mechanism High-Fidelity Reprogrammed Human IPSCs Have a High Efficacy of DNA Repair and Resemble hESCs in Their MYC Transcriptional Signature Double strand break repair functions of histone H2AX PARP inhibitor treatment in ovarian and breast cancer RIF1 is essential for 53BP1-dependent nonhomologous end joining and suppression of DNA double-strand break resection.MMEJ repair of double-strand breaks (director's cut): deleted sequences and alternative endings DNA fragile site breakage as a measure of chemical exposure and predictor of individual susceptibility to form oncogenic rearrangements.

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Alternative-NHEJ is a mechanistically distinct pathway of mammalian chromosome break repair

http://www.wikidata.org/entity/Q21145054

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2008 nî lūn-bûn

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2008年論文

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Alternative-NHEJ is a mechanis ...... malian chromosome break repair

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Anita Cheng

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Jeremy M Stark

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Nick Huang

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Nicole Bennardo

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P2860

Nuclear localization and cell cycle-specific expression of CtIP, a protein that associates with the BRCA1 tumor suppressor

Physical and functional interaction between the XPF/ERCC1 endonuclease and hRad52

Crystal structure and DNA binding functions of ERCC1, a subunit of the DNA structure-specific endonuclease XPF-ERCC1

XRCC3 promotes homology-directed repair of DNA damage in mammalian cells

Human CtIP promotes DNA end resection

DNA end resection, homologous recombination and DNA damage checkpoint activation require CDK1

Cancer predisposition caused by elevated mitotic recombination in Bloom mice

DNA double-strand breaks: signaling, repair and the cancer connection

Molecular views of recombination proteins and their control

IgH class switching and translocations use a robust non-classical end-joining pathway

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e1000110

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18584027

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double-strand break repair via alternative nonhomologous end joining

double-strand break repair via classical nonhomologous end joining

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2430616

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