Detailed immunohistochemical characterization of temporal and spatial progression of Alzheimer's disease-related pathologies in male triple-transgenic mice - Test2 - elhamkhani - TriplyDB

Detailed immunohistochemical characterization of temporal and spatial progression of Alzheimer's disease-related pathologies in male triple-transgenic mice

Detailed immunohistochemical characterization of temporal and spatial progression of Alzheimer's disease-related pathologies in male triple-transgenic mice

http://www.wikidata.org/entity/Q33359744

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Targeting microglial K(ATP) channels to treat neurodegenerative diseases: a mitochondrial issue The flavonoid quercetin ameliorates Alzheimer's disease pathology and protects cognitive and emotional function in aged triple transgenic Alzheimer's disease model mice IVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathology Age-related intraneuronal elevation of αII-spectrin breakdown product SBDP120 in rodent forebrain accelerates in 3×Tg-AD mice Oral Triphenylmethane Food Dye Analog, Brilliant Blue G, Prevents Neuronal Loss in APPSwDI/NOS2-/- Mouse Model.Autoimmune manifestations in the 3xTg-AD model of Alzheimer's disease.Intracerebroventricular streptozotocin exacerbates Alzheimer-like changes of 3xTg-AD mice Deficits in memory-guided limb movements impair obstacle avoidance locomotion in Alzheimer's disease mouse model Human neural stem cells alleviate Alzheimer-like pathology in a mouse model.Intranasal Insulin Prevents Anesthesia-Induced Cognitive Impairment and Chronic Neurobehavioral Changes.Brain gene expression of a sporadic (icv-STZ Mouse) and a familial mouse model (3xTg-AD mouse) of Alzheimer's disease.Abeta-directed single-chain antibody delivery via a serotype-1 AAV vector improves learning behavior and pathology in Alzheimer's disease mice.PAK inactivation impairs social recognition in 3xTg-AD Mice without increasing brain deposition of tau and Aβ Intranasal insulin prevents anesthesia-induced hyperphosphorylation of tau in 3xTg-AD mice Spatial frequency domain imaging of intrinsic optical property contrast in a mouse model of Alzheimer's disease Age-Dependent Modifications of AMPA Receptor Subunit Expression Levels and Related Cognitive Effects in 3xTg-AD Mice.Distinct temporal and anatomical distributions of amyloid-β and tau abnormalities following controlled cortical impact in transgenic mice Staging of Alzheimer's pathology in triple transgenic mice: a light and electron microscopic analysis.Early oligodendrocyte/myelin pathology in Alzheimer's disease mice constitutes a novel therapeutic target.Animal models of amyloid-beta-related pathologies in Alzheimer's disease.Tyrosine phosphorylation of tau accompanies disease progression in transgenic mouse models of tauopathy.GFAP isoforms in adult mouse brain with a focus on neurogenic astrocytes and reactive astrogliosis in mouse models of Alzheimer disease.Cholinotrophic basal forebrain system alterations in 3xTg-AD transgenic mice.The β-secretase-derived C-terminal fragment of βAPP, C99, but not Aβ, is a key contributor to early intraneuronal lesions in triple-transgenic mouse hippocampus.Chronic temporal lobe epilepsy is associated with enhanced Alzheimer-like neuropathology in 3×Tg-AD mice TFP5, a Peptide Inhibitor of Aberrant and Hyperactive Cdk5/p25, Attenuates Pathological Phenotypes and Restores Synaptic Function in CK-p25Tg Mice.Immunolesion-induced loss of cholinergic projection neurones promotes β-amyloidosis and tau hyperphosphorylation in the hippocampus of triple-transgenic mice.In vivo optical signatures of neuronal death in a mouse model of Alzheimer's disease.Increased hippocampal excitability in the 3xTgAD mouse model for Alzheimer's disease in vivo.Safety and pharmacological characterization of the molecular tweezer CLR01 - a broad-spectrum inhibitor of amyloid proteins' toxicity.Ablation of TNF-RI/RII expression in Alzheimer's disease mice leads to an unexpected enhancement of pathology: implications for chronic pan-TNF-α suppressive therapeutic strategies in the brain.BACE1 elevation is involved in amyloid plaque development in the triple transgenic model of Alzheimer's disease: differential Aβ antibody labeling of early-onset axon terminal pathology.Genomics of Dementia: APOE- and CYP2D6-Related Pharmacogenetics.Biochemical and morphological characterization of the AβPP/PS/tau triple transgenic mouse model and its relevance to sporadic Alzheimer's disease.Linalool reverses neuropathological and behavioral impairments in old triple transgenic Alzheimer's mice.3xTg-AD mice exhibit an activated central stress axis during early-stage pathology.Age-related loss of noradrenergic neurons in the brains of triple transgenic mice.A non-transgenic mouse model (icv-STZ mouse) of Alzheimer's disease: similarities to and differences from the transgenic model (3xTg-AD mouse).Sustained interleukin-1β overexpression exacerbates tau pathology despite reduced amyloid burden in an Alzheimer's mouse model.Transgenic models of Alzheimer's disease: better utilization of existing models through viral transgenesis.

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Detailed immunohistochemical characterization of temporal and spatial progression of Alzheimer's disease-related pathologies in male triple-transgenic mice

http://www.wikidata.org/entity/Q33359744

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2008 nî lūn-bûn

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2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Detailed immunohistochemical c ...... in male triple-transgenic mice

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Detailed immunohistochemical c ...... in male triple-transgenic mice

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Detailed immunohistochemical c ...... in male triple-transgenic mice

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Detailed immunohistochemical c ...... in male triple-transgenic mice

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Detailed immunohistochemical c ...... in male triple-transgenic mice

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Detailed immunohistochemical c ...... in male triple-transgenic mice

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BMC Neuroscience

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Detailed immunohistochemical c ...... in male triple-transgenic mice

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Michael A Mastrangelo

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William J Bowers

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P2860

A protein factor essential for microtubule assembly

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments

Global prevalence of dementia: a Delphi consensus study

Cloning and sequencing of the cDNA encoding a core protein of the paired helical filament of Alzheimer disease: identification as the microtubule-associated protein tau

Ultrastructure and biochemical composition of paired helical filaments in corticobasal degeneration

Early correlation of microglial activation with enhanced tumor necrosis factor-alpha and monocyte chemoattractant protein-1 expression specifically within the entorhinal cortex of triple transgenic Alzheimer's disease mice

Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

Early phenotypic changes in transgenic mice that overexpress different mutants of amyloid precursor protein in brain

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10.1186/1471-2202-9-81

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9

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2008-08-12T00:00:00Z

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Alzheimer's disease

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