Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis. - Test2 - elhamkhani - TriplyDB

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

http://www.wikidata.org/entity/Q33367473

about

The role of epigenetics in the pathology of diabetic complications The miR-30 miRNA family regulates Xenopus pronephros development and targets the transcription factor Xlim1/Lhx1 MicroRNAs in kidney fibrosis and diabetic nephropathy: roles on EMT and EndMT MicroRNAs in the pathogenesis of cystic kidney disease Developments in renal pharmacogenomics and applications in chronic kidney disease MicroRNAs in Diabetic Kidney Disease Epigenetics of progression of chronic kidney disease: fact or fantasy?Stem cells: potential and challenges for kidney repair Genetic and epigenetic factors influencing chronic kidney disease Epigenetics of Renal Development and Disease Podocin inactivation in mature kidneys causes focal segmental glomerulosclerosis and nephrotic syndrome Mini-review: emerging roles of microRNAs in the pathophysiology of renal diseases microRNAs in kidneys: biogenesis, regulation, and pathophysiological roles Systems biology approach to identify transcriptome reprogramming and candidate microRNA targets during the progression of polycystic kidney disease miR-21 and miR-214 are consistently modulated during renal injury in rodent models TGFβ-stimulated microRNA-21 utilizes PTEN to orchestrate AKT/mTORC1 signaling for mesangial cell hypertrophy and matrix expansion MicroRNA-23b Targets Ras GTPase-Activating Protein SH3 Domain-Binding Protein 2 to Alleviate Fibrosis and Albuminuria in Diabetic Nephropathy Human SBK1 is dysregulated in multiple cancers and promotes survival of ovary cancer SK-OV-3 cells.Dicer regulates the development of nephrogenic and ureteric compartments in the mammalian kidney.The microRNA-processing enzyme dicer maintains juxtaglomerular cells miRNAs in mammalian ureteric bud development.Reductions in laminin beta2 mRNA translation are responsible for impaired IGFBP-5-mediated mesangial cell migration in the presence of high glucose.MicroRNA-30a Regulation of Epithelial-Mesenchymal Transition in Diabetic Cataracts Through Targeting SNAI1.BAMBI is expressed in endothelial cells and is regulated by lysosomal/autolysosomal degradation Intermediate filaments take the heat as stress proteins.Recurrent somatic mutation in DROSHA induces microRNA profile changes in Wilms tumour MicroRNA-17~92 is required for nephrogenesis and renal function Targeted deletion of Dicer from proximal tubules protects against renal ischemia-reperfusion injury.Modeling bias and variation in the stochastic processes of small RNA sequencing.The microRNA-processing enzyme Dicer is essential for thyroid function.MicroRNA-34a is induced via p53 during cisplatin nephrotoxicity and contributes to cell survival.Evaluation of microRNAs miR-196a, miR-30a-5P, and miR-490 as biomarkers of disease activity among patients with FSGS.A miR-1207-5p binding site polymorphism abolishes regulation of HBEGF and is associated with disease severity in CFHR5 nephropathy The role of microRNAs in diabetic nephropathy.MicroRNA profiles in allograft tissues and paired urines associate with chronic allograft dysfunction with IF/TA.Morphogenesis and molecular mechanisms involved in human kidney development.MicroRNA-324-3p promotes renal fibrosis and is a target of ACE inhibition Renal stromal miRNAs are required for normal nephrogenesis and glomerular mesangial survival.Upregulated expression of integrin α1 in mesangial cells and integrin α3 and vimentin in podocytes of Col4a3-null (Alport) mice Urinary microRNA-10a and microRNA-30d serve as novel, sensitive and specific biomarkers for kidney injury

P2860

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P2860

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

http://www.wikidata.org/entity/Q33367473

description

2008 nî lūn-bûn

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2008 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

@ast

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

@en

type

label

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

@ast

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

@en

prefLabel

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

@ast

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

@en

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Journal of the American Society of Nephrology

P1476

Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.

@en

P2093

Celine Chiu

http://www.w3.org/2001/XMLSchema#string

Greg Khitrov

http://www.w3.org/2001/XMLSchema#string

Jin Chen

http://www.w3.org/2001/XMLSchema#string

Lawrence B Holzman

http://www.w3.org/2001/XMLSchema#string

Liping Yu

http://www.w3.org/2001/XMLSchema#string

Peter Mundel

http://www.w3.org/2001/XMLSchema#string

Shaolin Shi

http://www.w3.org/2001/XMLSchema#string

Weijia Zhang

http://www.w3.org/2001/XMLSchema#string

Yezhou Sun

http://www.w3.org/2001/XMLSchema#string

P2860

Targeted inhibition of miRNA maturation with morpholinos reveals a role for miR-375 in pancreatic islet development

Bioconductor: open software development for computational biology and bioinformatics

Immediate early gene X-1 interacts with proteins that modulate apoptosis

S100B and S100A6 differentially modulate cell survival by interacting with distinct RAGE (receptor for advanced glycation end products) immunoglobulin domains

The RNaseIII enzyme Dicer is required for morphogenesis but not patterning of the vertebrate limb.

Exportin 5 is a RanGTP-dependent dsRNA-binding protein that mediates nuclear export of pre-miRNAs

MicroRNA genes are transcribed by RNA polymerase II

Significance analysis of microarrays applied to the ionizing radiation response

Targeted deletion of Dicer in the heart leads to dilated cardiomyopathy and heart failure

Requirement of bic/microRNA-155 for normal immune function

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2159-2169

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10.1681/ASN.2008030312

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11

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19

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Matthias Merkenschlager

Erwin Böttinger

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2008-09-05T00:00:00Z

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23242089

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18776119

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2573016

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