Mutator mutations enhance tumorigenic efficiency across fitness landscapes. - Test2 - elhamkhani - TriplyDB

Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

http://www.wikidata.org/entity/Q33466042

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The mutator phenotype in cancer: molecular mechanisms and targeting strategies The origins of cancer robustness and evolvability.Impact of genetic dynamics and single-cell heterogeneity on development of nonstandard personalized medicine strategies for cancer.The rich phase structure of a mutator model.Long range personalized cancer treatment strategies incorporating evolutionary dynamics.Translating predictive biomarkers within oncology clinical development programs.Genomic Instability in Cancer: Teetering on the Limit of Tolerance.Evolutionary dynamics and significance of multiple subclonal mutations in cancer.The effect of one additional driver mutation on tumor progression.Reply: Is There Any Genetic Instability in Human Cancer?Modelling the evolution of genetic instability during tumour progression.Can a minimal replicating construct be identified as the embodiment of cancer?

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Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

http://www.wikidata.org/entity/Q33466042

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

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Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

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Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

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Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

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Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

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Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

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Mutator mutations enhance tumorigenic efficiency across fitness landscapes.

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Robert A Beckman

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P2860

Ubiquitous somatic mutations in simple repeated sequences reveal a new mechanism for colonic carcinogenesis

The Hallmarks of Cancer

Embryonic stem cells and somatic cells differ in mutation frequency and type

A new theory on cancer-inducing mechanism

The age distribution of cancer and a multi-stage theory of carcinogenesis

Human cancers express a mutator phenotype

The consensus coding sequences of human breast and colorectal cancers

The genomic landscapes of human breast and colorectal cancers

Genetic instabilities in human cancers

Mutation at the polymerase active site of mouse DNA polymerase delta increases genomic instability and accelerates tumorigenesis

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