IFN-λ receptor 1 expression is induced in chronic hepatitis C and correlates with the IFN-λ3 genotype and with nonresponsiveness to IFN-α therapies
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Harnessing Mechanistic Knowledge on Beneficial Versus Deleterious IFN-I Effects to Design Innovative Immunotherapies Targeting Cytokine Activity to Specific Cell TypesInterferon lambda polymorphisms associate with body iron indices and hepatic expression of interferon-responsive long non-coding RNA in chronic hepatitis C.IFN-λ4: the paradoxical new member of the interferon lambda family.Predictive value of interferon-lambda gene polymorphisms for treatment response in chronic hepatitis C.IL-28B is a key regulator of B- and T-cell vaccine responses against influenzaIFNL4 and IFNL3 associated polymorphisms strongly influence the spontaneous IFN-alpha receptor-1 expression in HCV-infected patients.Transcriptional Dysregulation of Upstream Signaling of IFN Pathway in Chronic HCV Type 4 Induced Liver Fibrosis.The Effect of Chronic Hepatitis B Virus Infection on BDCA3+ Dendritic Cell Frequency and Function.Viral genotype correlates with distinct liver gene transcription signatures in chronic hepatitis C virus infection.IFN-λ3 polymorphism indirectly influences NK cell phenotype and function during acute HCV infectionConcerted action of IFN-α and IFN-λ induces local NK cell immunity and halts cancer growth.Interferon Lambda: Modulating Immunity in Infectious Diseases.Innate and adaptive genetic pathways in HCV infection.The impact of the interferon-lambda family on the innate and adaptive immune response to viral infections.Cellular Mechanism for Impaired Hepatitis C Virus Clearance by Interferon Associated with IFNL3 Gene Polymorphisms Relates to Intrahepatic Interferon-λ Expression.IFN-λ cancer immunotherapy: new kid on the block.The Role of Type III Interferons in Hepatitis C Virus Infection and Therapy.Contribution of type III interferons to antiviral immunity: location, location, location.Evaluation of HTLV-1 HBZ and proviral load, together with host IFN λ3, in pathogenesis of HAM/TSP.SOCS1 is an inducible negative regulator of IFN lambda induced gene expression in vivo.IFNλ is a potent anti-influenza therapeutic without the inflammatory side effects of IFNα treatment.Transcriptome analysis reveals a classical interferon signature induced by IFNλ4 in human primary cells.Retinoic acid-inducible gene 1 and sensing of hepatitis B virus revisited.Hepatic expression levels of interferons and interferon-stimulated genes in patients with chronic hepatitis C: A phenotype-genotype correlation study.Does the interferon-lambda rather than the interferon-alpha pathway determine the outcome of hepatitis C virus infection?
P2860
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P2860
IFN-λ receptor 1 expression is induced in chronic hepatitis C and correlates with the IFN-λ3 genotype and with nonresponsiveness to IFN-α therapies
description
2014 nî lūn-bûn
@nan
2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年学术文章
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2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
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2014年學術文章
@yue
name
IFN-λ receptor 1 expression is ...... ponsiveness to IFN-α therapies
@ast
IFN-λ receptor 1 expression is ...... ponsiveness to IFN-α therapies
@en
IFN-λ receptor 1 expression is ...... onsiveness to IFN-α therapies.
@nl
type
label
IFN-λ receptor 1 expression is ...... ponsiveness to IFN-α therapies
@ast
IFN-λ receptor 1 expression is ...... ponsiveness to IFN-α therapies
@en
IFN-λ receptor 1 expression is ...... onsiveness to IFN-α therapies.
@nl
prefLabel
IFN-λ receptor 1 expression is ...... ponsiveness to IFN-α therapies
@ast
IFN-λ receptor 1 expression is ...... ponsiveness to IFN-α therapies
@en
IFN-λ receptor 1 expression is ...... onsiveness to IFN-α therapies.
@nl
P2093
P2860
P50
P356
P1476
IFN-λ receptor 1 expression is ...... ponsiveness to IFN-α therapies
@en
P2093
Benedetta Campana
Francois H T Duong
Gaia Trincucci
Ilona Krol
Laura Heydmann
Sarah C Durand
Thomas F Baumert
Tujana Boldanova
P2860
P304
P356
10.1084/JEM.20131557
P407
P577
2014-04-21T00:00:00Z