Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice. - Test2 - elhamkhani - TriplyDB

Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.

Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.

http://www.wikidata.org/entity/Q33606369

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The role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesis Development of next-generation respiratory virus vaccines through targeted modifications to viral immunomodulatory genes Coronavirus virulence genes with main focus on SARS-CoV envelope gene Coronavirus gene 7 counteracts host defenses and modulates virus virulence Genome-wide analysis of protein-protein interactions and involvement of viral proteins in SARS-CoV replication Structure and inhibition of the SARS coronavirus envelope protein ion channel Structure of a conserved Golgi complex-targeting signal in coronavirus envelope proteins Viral and Cellular mRNA Translation in Coronavirus-Infected Cells.Severe acute respiratory syndrome coronavirus envelope protein ion channel activity promotes virus fitness and pathogenesis Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease.SARS-CoV 9b protein diffuses into nucleus, undergoes active Crm1 mediated nucleocytoplasmic export and triggers apoptosis when retained in the nucleus.The PDZ-binding motif of severe acute respiratory syndrome coronavirus envelope protein is a determinant of viral pathogenesis.Effects of Toll-like receptor stimulation on eosinophilic infiltration in lungs of BALB/c mice immunized with UV-inactivated severe acute respiratory syndrome-related coronavirus vaccine.Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis.The SARS coronavirus E protein interacts with PALS1 and alters tight junction formation and epithelial morphogenesis.An immunosuppressed Syrian golden hamster model for SARS-CoV infection.Severe acute respiratory syndrome coronaviruses with mutations in the E protein are attenuated and promising vaccine candidates.A Coronavirus E Protein Is Present in Two Distinct Pools with Different Effects on Assembly and the Secretory Pathway.Coronavirus envelope (E) protein remains at the site of assembly Severe acute respiratory syndrome coronavirus E protein transports calcium ions and activates the NLRP3 inflammasome Coronavirus E protein forms ion channels with functionally and structurally-involved membrane lipids.Identification of the Mechanisms Causing Reversion to Virulence in an Attenuated SARS-CoV for the Design of a Genetically Stable Vaccine.Subcellular location and topology of severe acute respiratory syndrome coronavirus envelope protein Complete protection against severe acute respiratory syndrome coronavirus-mediated lethal respiratory disease in aged mice by immunization with a mouse-adapted virus lacking E protein.Analysis of SARS-CoV E protein ion channel activity by tuning the protein and lipid charge.Severe acute respiratory syndrome coronavirus protein 6 is required for optimal replication.Alphacoronavirus protein 7 modulates host innate immune response.Engineering a replication-competent, propagation-defective Middle East respiratory syndrome coronavirus as a vaccine candidate SARS vaccines: where are we?Inhibition of NF-κB-mediated inflammation in severe acute respiratory syndrome coronavirus-infected mice increases survival Animal models in virus research: their utility and limitations.Self-Replicating RNA.Role of fomites in SARS transmission during the largest hospital outbreak in Hong Kong.A mouse model for MERS coronavirus-induced acute respiratory distress syndrome.Severe Acute Respiratory Syndrome (SARS) Coronavirus ORF8 Protein Is Acquired from SARS-Related Coronavirus from Greater Horseshoe Bats through Recombination Acquisition of new protein domains by coronaviruses: analysis of overlapping genes coding for proteins N and 9b in SARS coronavirus.Molecular epidemiology and phylogenetic analysis of porcine epidemic diarrhea virus (PEDV) field isolates in Korea.Transgenic expression of human P-selectin glycoprotein ligand-1 is not sufficient for enterovirus 71 infection in mice.Role of Severe Acute Respiratory Syndrome Coronavirus Viroporins E, 3a, and 8a in Replication and Pathogenesis.

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P2860

Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.

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J.VIROL.2008.03.005

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Pathogenicity of severe acute ...... ants in hACE-2 transgenic mice

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Lecia Pewe

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Maria Teresa Rejas

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Marta L Dediego

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P2860

Severe acute respiratory syndrome coronavirus-like virus in Chinese horseshoe bats

Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein

Characterization and complete genome sequence of a novel coronavirus, coronavirus HKU1, from patients with pneumonia

Severe acute respiratory syndrome coronavirus phylogeny: toward consensus

Severe acute respiratory syndrome coronavirus 7a accessory protein is a viral structural protein

Coronavirus particle assembly: primary structure requirements of the membrane protein.

Robust hepatitis C virus infection in vitro.

Coronavirus as a possible cause of severe acute respiratory syndrome

Characterization of a novel coronavirus associated with severe acute respiratory syndrome

Bats are natural reservoirs of SARS-like coronaviruses

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