about
A transmembrane serine protease is linked to the severe acute respiratory syndrome coronavirus receptor and activates virus entryACE2 receptor expression and severe acute respiratory syndrome coronavirus infection depend on differentiation of human airway epithelia.Middle East respiratory syndrome coronavirus (MERS-CoV): announcement of the Coronavirus Study GroupStructural and biological basis of CTL escape in coronavirus-infected micePrevention of Cytotoxic T Cell Escape Using a Heteroclitic Subdominant Viral T Cell DeterminantCrystal structure-based exploration of the important role of Arg106 in the RNA-binding domain of human coronavirus OC43 nucleocapsid proteinStructural Basis for the Identification of the N-Terminal Domain of Coronavirus Nucleocapsid Protein as an Antiviral TargetStructural and Functional Correlates of Enhanced Antiviral Immunity Generated by Heteroclitic CD8 T Cell EpitopesInfection of human airway epithelia by SARS coronavirus is associated with ACE2 expression and localizationEquine Immunoglobulin and Equine Neutralizing F(ab')₂ Protect Mice from West Nile Virus InfectionRapid generation of a mouse model for Middle East respiratory syndrome.Identification of an ideal adjuvant for receptor-binding domain-based subunit vaccines against Middle East respiratory syndrome coronavirus.A humanized neutralizing antibody against MERS-CoV targeting the receptor-binding domain of the spike protein.DNA vaccine encoding Middle East respiratory syndrome coronavirus S1 protein induces protective immune responses in mice.Congenital viral infections of the brain: lessons learned from lymphocytic choriomeningitis virus in the neonatal rat.Evasion by stealth: inefficient immune activation underlies poor T cell response and severe disease in SARS-CoV-infected mice.Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.De novo recruitment of antigen-experienced and naive T cells contributes to the long-term maintenance of antiviral T cell populations in the persistently infected central nervous systemCoronaviruses post-SARS: update on replication and pathogenesis.Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease.The N-terminal region of severe acute respiratory syndrome coronavirus protein 6 induces membrane rearrangement and enhances virus replication.Regulatory T cells inhibit T cell proliferation and decrease demyelination in mice chronically infected with a coronavirusViral expression of CCL2 is sufficient to induce demyelination in RAG1-/- mice infected with a neurotropic coronavirus.Virus-neutralizing monoclonal antibody expressed in milk of transgenic mice provides full protection against virus-induced encephalitis.High-magnitude, virus-specific CD4 T-cell response in the central nervous system of coronavirus-infected mice.Important roles for gamma interferon and NKG2D in gammadelta T-cell-induced demyelination in T-cell receptor beta-deficient mice infected with a coronavirusA severe acute respiratory syndrome-associated coronavirus-specific protein enhances virulence of an attenuated murine coronavirusVirus-specific regulatory T cells ameliorate encephalitis by repressing effector T cell functions from priming to effector stagesSevere acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis.T cell responses are required for protection from clinical disease and for virus clearance in severe acute respiratory syndrome coronavirus-infected mice.Autocrine interferon priming in macrophages but not dendritic cells results in enhanced cytokine and chemokine production after coronavirus infectionVirus-specific memory CD8 T cells provide substantial protection from lethal severe acute respiratory syndrome coronavirus infection.The cellular redox environment alters antigen presentationMouse hepatitis virus.CD4 T-cell-mediated demyelination is increased in the absence of gamma interferon in mice infected with mouse hepatitis virus.Preferential infection of mature dendritic cells by mouse hepatitis virus strain JHMMurine coronavirus ubiquitin-like domain is important for papain-like protease stability and viral pathogenesis.Coronaviruses: an overview of their replication and pathogenesis.Immunopathogenesis of coronavirus infections: implications for SARS.Highly activated cytotoxic CD8 T cells express protective IL-10 at the peak of coronavirus-induced encephalitis.
P50
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P50
description
Forscher
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chercheur
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investigador
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onderzoeker
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researcher
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հետազոտող
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研究者
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name
Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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Stanley Perlman
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P106
P1153
7102708317
P31
P496
0000-0003-4213-2354