Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
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The role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesisSevere acute respiratory syndrome coronavirus 7a accessory protein is a viral structural proteinGenetic lesions within the 3a gene of SARS-CoV.Identification of Hepta- and Octo-Uridine stretches as sole signals for programmed +1 and -1 ribosomal frameshifting during translation of SARS-CoV ORF 3a variants.Severe acute respiratory syndrome coronavirus as an agent of emerging and reemerging infectionGenome-wide analysis of protein-protein interactions and involvement of viral proteins in SARS-CoV replicationAmino acids 15-28 in the ectodomain of SARS coronavirus 3a protein induces neutralizing antibodies.Analysis of intraviral protein-protein interactions of the SARS coronavirus ORFeome.Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.A single tyrosine in the severe acute respiratory syndrome coronavirus membrane protein cytoplasmic tail is important for efficient interaction with spike proteinCoronaviruses post-SARS: update on replication and pathogenesis.The N-terminal region of severe acute respiratory syndrome coronavirus protein 6 induces membrane rearrangement and enhances virus replication.A severe acute respiratory syndrome-associated coronavirus-specific protein enhances virulence of an attenuated murine coronavirusSevere acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis.Severe acute respiratory syndrome coronavirus group-specific open reading frames encode nonessential functions for replication in cell cultures and mice.Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.Severe acute respiratory syndrome coronavirus 3a protein is released in membranous structures from 3a protein-expressing cells and infected cells.Severe acute respiratory syndrome-associated coronavirus 3a protein forms an ion channel and modulates virus release.Severe acute respiratory syndrome coronavirus nsp1 protein suppresses host gene expression by promoting host mRNA degradationSevere acute respiratory syndrome coronavirus protein 6 accelerates murine coronavirus infections.A severe acute respiratory syndrome coronavirus that lacks the E gene is attenuated in vitro and in vivo.The Emerging Roles of Viroporins in ER Stress Response and Autophagy Induction during Virus InfectionSevere acute respiratory syndrome coronavirus accessory protein 6 is a virion-associated protein and is released from 6 protein-expressing cells.The ns12.9 Accessory Protein of Human Coronavirus OC43 Is a Viroporin Involved in Virion Morphogenesis and Pathogenesis.The 29-nucleotide deletion present in human but not in animal severe acute respiratory syndrome coronaviruses disrupts the functional expression of open reading frame 8.Hosting the severe acute respiratory syndrome coronavirus: specific cell factors required for infection.Proteomics analysis unravels the functional repertoire of coronavirus nonstructural protein 3.Severe acute respiratory syndrome coronavirus protein 6 accelerates murine hepatitis virus infections by more than one mechanism.Vaccines to prevent severe acute respiratory syndrome coronavirus-induced disease.The transmembrane domain of the severe acute respiratory syndrome coronavirus ORF7b protein is necessary and sufficient for its retention in the Golgi complex.SARS coronavirus accessory proteins.Humoral and cellular immune responses induced by 3a DNA vaccines against severe acute respiratory syndrome (SARS) or SARS-like coronavirus in mice.Severe acute respiratory syndrome coronavirus protein 6 is required for optimal replication.Suppression of host gene expression by nsp1 proteins of group 2 bat coronaviruses.Molecular determinants for subcellular localization of the severe acute respiratory syndrome coronavirus open reading frame 3b protein.SARS vaccines: where are we?The YXXΦ motif within the severe acute respiratory syndrome coronavirus (SARS-CoV) 3a protein is crucial for its intracellular transport.Significance of coronavirus mutants in feces and diseased tissues of cats suffering from feline infectious peritonitis.Virulence factors in porcine coronaviruses and vaccine design.Human coronavirus NL63 open reading frame 3 encodes a virion-incorporated N-glycosylated membrane protein.
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P2860
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
description
2005 nî lūn-bûn
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2005 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մարտին հրատարակված գիտական հոդված
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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name
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
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Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@en
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@nl
type
label
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@ast
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@en
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@nl
prefLabel
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@ast
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@en
Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@nl
P2093
P2860
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Severe acute respiratory syndrome coronavirus 3a protein is a viral structural protein
@en
P2093
Eric C Mossel
Krishna Narayanan
Taisuke Inoue
Vsevolod L Popov
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P304
P356
10.1128/JVI.79.5.3182-3186.2005
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2005-03-01T00:00:00Z