Progress in understanding the factors regulating reversibility of long-term potentiation.
about
Levodopa-induced plasticity: a double-edged sword in Parkinson's disease?The reemergence of long-term potentiation in aged Alzheimer's disease mouse model.Glutamate receptors in extinction and extinction-based therapies for psychiatric illnessLong-term synaptic plasticity is impaired in rats with lesions of the ventrolateral preoptic nucleus.Cellular mechanisms underlying acquired epilepsy: the calcium hypothesis of the induction and maintainance of epilepsy.Behavioral and neuroanatomical abnormalities in pleiotrophin knockout mice.Reversal of plasticity-like effects in the human motor cortexLocal stimulation of the adenosine A2B receptors induces an increased release of IL-6 in mouse striatum: an in vivo microdialysis study.Structural and molecular remodeling of dendritic spine substructures during long-term potentiation.A form of long-lasting, learning-related synaptic plasticity in the hippocampus induced by heterosynaptic low-frequency pairing.A Temporary Gating of Actin Remodeling during Synaptic Plasticity Consists of the Interplay between the Kinase and Structural Functions of CaMKIICompetitive interactions between endogenous LTD and LTP in the hippocampus underlie the storage of emotional memories and stress-induced amnesia.Differential translation and fragile X syndrome.Conformational signaling required for synaptic plasticity by the NMDA receptor complexStatus Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent MannerHippocampal neurogenesis protects against cocaine-primed relapse.Neuregulin-1 regulates LTP at CA1 hippocampal synapses through activation of dopamine D4 receptorsMechanisms of disease: basic-research-driven investigations in humans--the case of hyperkinetic disorders.A role of p38 mitogen-activated protein kinase in adenosine A₁ receptor-mediated synaptic depotentiation in area CA1 of the rat hippocampus.Mice overexpressing type 1 adenylyl cyclase show enhanced spatial memory flexibility in the absence of intact synaptic long-term depression.Cannabinoids in Parkinson's DiseaseSeparate Ionotropic and Metabotropic Glutamate Receptor Functions in Depotentiation vs. LTP: A Distinct Role for Group1 mGluR Subtypes and NMDARs.Different phosphatase-dependent mechanisms mediate long-term depression and depotentiation of long-term potentiation in mouse hippocampal CA1 area.Enhancement of Cognitive and Electrophysiological Measures of Hippocampal Functioning in Rats by a Low, But Not High, Dose of Dehydroepiandrosterone Sulfate (DHEAS)Bidirectional synaptic plasticity and spatial memory flexibility require Ca2+-stimulated adenylyl cyclases.Leptin reverses long-term potentiation at hippocampal CA1 synapses.Switching off LTP: mGlu and NMDA receptor-dependent novelty exploration-induced depotentiation in the rat hippocampus.Characterization of the mechanism underlying the reversal of long term potentiation by low frequency stimulation at hippocampal CA1 synapses.Postsynaptic application of a cAMP analogue reverses long-term potentiation in hippocampal CA1 pyramidal neurons.Role for cAMP and protein phosphatase in the presynaptic expression of mouse hippocampal mossy fibre depotentiation.Loss of bidirectional striatal synaptic plasticity in L-DOPA-induced dyskinesia.Long-term potentiation is impaired in rat hippocampal slices that produce spontaneous sharp waves.Local protein synthesis and GABAB receptors regulate the reversibility of long-term potentiation at murine hippocampal mossy fibre-CA3 synapses.NR2A-containing NMDA receptors are required for L-LTP induction and depotentiation in CA1 region of hippocampal slices.Bidirectional modulation of hippocampal synaptic plasticity by Dopaminergic D4-receptors in the CA1 area of hippocampus.Modulation by group 1 metabotropic glutamate receptors of depotentiation in the dentate gyrus of freely moving rats.Regulation of Striatal Neuron Activity by Cyclic Nucleotide Signaling and Phosphodiesterase Inhibition: Implications for the Treatment of Parkinson's Disease.Partial inhibition of PP1 alters bidirectional synaptic plasticity in the hippocampus.Spike-timing-dependent plasticity at resting and conditioned lateral perforant path synapses on granule cells in the dentate gyrus: different roles of N-methyl-D-aspartate and group I metabotropic glutamate receptors.
P2860
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P2860
Progress in understanding the factors regulating reversibility of long-term potentiation.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Progress in understanding the factors regulating reversibility of long-term potentiation.
@ast
Progress in understanding the factors regulating reversibility of long-term potentiation.
@en
type
label
Progress in understanding the factors regulating reversibility of long-term potentiation.
@ast
Progress in understanding the factors regulating reversibility of long-term potentiation.
@en
prefLabel
Progress in understanding the factors regulating reversibility of long-term potentiation.
@ast
Progress in understanding the factors regulating reversibility of long-term potentiation.
@en
P2860
P1476
Progress in understanding the factors regulating reversibility of long-term potentiation.
@en
P2860
P356
10.1515/REVNEURO.2001.12.1.51
P577
2001-01-01T00:00:00Z