Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophy
about
Cdc42: An essential Rho-type GTPase controlling eukaryotic cell polarity.The case for inhibiting p38 mitogen-activated protein kinase in heart failureMitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.Wiring the cell signaling circuitry by the NF-kappa B and JNK1 crosstalk and its applications in human diseasesTargeted inhibition of calcineurin prevents agonist-induced cardiomyocyte hypertrophySpecific role of the extracellular signal-regulated kinase pathway in angiotensin II-induced cardiac hypertrophy in vitroTissue-specific GATA factors are transcriptional effectors of the small GTPase RhoAActivation of NF-kappa B is required for hypertrophic growth of primary rat neonatal ventricular cardiomyocytesActivated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivoRole of the stress-activated protein kinases in endothelin-induced cardiomyocyte hypertrophyThe small GTP-binding protein Rac1 induces cardiac myocyte hypertrophy through the activation of apoptosis signal-regulating kinase 1 and nuclear factor-kappa BDistinct roles of mitogen-activated protein kinase pathways in GATA-4 transcription factor-mediated regulation of B-type natriuretic peptide genep38 MAP kinases in the heartGuanine nucleotide exchange factor-like factor (Rlf) induces gene expression and potentiates alpha 1-adrenergic receptor-induced transcriptional responses in neonatal rat ventricular myocytes.Phosphorylation-dependent degradation of p300 by doxorubicin-activated p38 mitogen-activated protein kinase in cardiac cells.Regulation of cardiac hypertrophy in vivo by the stress-activated protein kinases/c-Jun NH(2)-terminal kinases.Mechanical strain activates BNP gene transcription through a p38/NF-kappaB-dependent mechanism.AFos dissociates cardiac myocyte hypertrophy and expression of the pathological gene programThe role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis.Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectivesCyclic AMP inhibits p38 activation via CREB-induced dynein light chain.p38alpha mitogen-activated protein kinase plays a critical role in cardiomyocyte survival but not in cardiac hypertrophic growth in response to pressure overload.Paradoxical impairment of angiogenesis, endothelial function and circulating number of endothelial progenitor cells in DPP4-deficient rat after critical limb ischemia.The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy.Differentiation stage-specific activation of p38 mitogen-activated protein kinase isoforms in primary human erythroid cellsDentin matrix protein 1 (DMP1) signals via cell surface integrin.Genetic analysis of p38 MAP kinases in myogenesis: fundamental role of p38alpha in abrogating myoblast proliferationThe role of endothelin-1 in myocarditis and inflammatory cardiomyopathy: old lessons and new insights.Metabolic Reprogramming Is Required for Myofibroblast Contractility and Differentiation.Activation of nuclear factor-kappaB is necessary for myotrophin-induced cardiac hypertrophy.Low-dose radiation affects cardiac physiology: gene networks and molecular signaling in cardiomyocytes.Characterization of hsp27 kinases activated by elevated aortic pressure in heart.Role of mitogen-activated protein kinase pathways in multifactorial adverse cardiac remodeling associated with metabolic syndromeThe role of the paracrine/autocrine mediator endothelin-1 in regulation of cardiac contractility and growth.Calcium signaling phenomena in heart diseases: a perspective.α₁A-adrenergic receptors regulate cardiac hypertrophy in vivo through interleukin-6 secretion.Regulation of Ncx1 gene expression in the normal and hypertrophic heart.Overexpression of mitogen-activated protein kinase kinase 6 in the heart improves functional recovery from ischemia in vitro and protects against myocardial infarction in vivo.Survival pathways in hypertrophy and heart failure: the gp130-STAT axis.Role of mitogen-activated protein kinase in cardiac hypertrophy and heart failure
P2860
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P2860
Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophy
description
1998 nî lūn-bûn
@nan
1998 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Opposing effects of Jun kinase ...... s on cardiomyocyte hypertrophy
@ast
Opposing effects of Jun kinase ...... s on cardiomyocyte hypertrophy
@en
type
label
Opposing effects of Jun kinase ...... s on cardiomyocyte hypertrophy
@ast
Opposing effects of Jun kinase ...... s on cardiomyocyte hypertrophy
@en
prefLabel
Opposing effects of Jun kinase ...... s on cardiomyocyte hypertrophy
@ast
Opposing effects of Jun kinase ...... s on cardiomyocyte hypertrophy
@en
P2093
P2860
P356
P1476
Opposing effects of Jun kinase ...... s on cardiomyocyte hypertrophy
@en
P2093
P2860
P304
P356
10.1128/MCB.18.6.3518
P407
P577
1998-06-01T00:00:00Z