Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors. - Test2 - elhamkhani - TriplyDB

Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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Physical and functional interactions between STAT3 and Kaposi's sarcoma-associated herpesvirus-encoded LANA A Central Role for STAT3 in Gammaherpesvirus-Life Cycle and -Diseases Molecular signature of Epstein Barr virus-positive Burkitt lymphoma and post-transplant lymphoproliferative disorder suggest different roles for Epstein Barr virus.EBV latent membrane protein 1 activates Akt, NFkappaB, and Stat3 in B cell lymphomas.IL-21 imposes a type II EBV gene expression on type III and type I B cells by the repression of C- and activation of LMP-1-promoter.Augmented latent membrane protein 1 expression from Epstein-Barr virus episomes with minimal terminal repeats Latent membrane protein 1 regulates STAT1 through NF-kappaB-dependent interferon secretion in Epstein-Barr virus-immortalized B cells.Modulation of the cell growth regulator mTOR by Epstein-Barr virus-encoded LMP2A.Constitutive activation of Stat5a by retrovirus integration in early pre-B lymphomas of SL/Kh strain mice.Identification and characterization of CCAAT enhancer-binding protein (C/EBP) as a transcriptional activator for Epstein-Barr virus oncogene latent membrane protein 1.Burkitt's lymphoma: the Rosetta Stone deciphering Epstein-Barr virus biology.Enhanced IL-6/IL-6R signaling promotes growth and malignant properties in EBV-infected premalignant and cancerous nasopharyngeal epithelial cells.Heat shock protein 90 inhibitors repress latent membrane protein 1 (LMP1) expression and proliferation of Epstein-Barr virus-positive natural killer cell lymphoma.Gene expression profiling of Epstein-Barr virus-positive diffuse large B-cell lymphoma of the elderly reveals alterations of characteristic oncogenetic pathways The heat shock protein 90 inhibitor BIIB021 suppresses the growth of T and natural killer cell lymphomas.Recent advances in underlying pathologies provide insight into interleukin-8 expression-mediated inflammation and angiogenesis.Technology insight: Applications of emerging immunotherapeutic strategies for Epstein-Barr virus-associated malignancies.Epstein-Barr virus lytic transactivator Zta enhances chemotactic activity through induction of interleukin-8 in nasopharyngeal carcinoma cells.Constitutive activation of STAT3 and STAT5 is present in the majority of nasopharyngeal carcinoma and correlates with better prognosis Induction of Epstein-Barr Virus Oncoprotein LMP1 by Transcription Factors AP-2 and Early B Cell Factor NF-κB Signaling Regulates Expression of Epstein-Barr Virus BART MicroRNAs and Long Noncoding RNAs in Nasopharyngeal Carcinoma.Ablation of STAT3 in the B Cell Compartment Restricts Gammaherpesvirus Latency In Vivo.Signal transducer and activator of transcription 3 limits Epstein-Barr virus lytic activation in B lymphocytes.Prognostic significance of signal transducer and activator of transcription 5 and 5b expression in Epstein-Barr virus-positive patients with chronic lymphocytic leukemia B lymphocytes from patients with a hypomorphic mutation in STAT3 resist Epstein-Barr virus-driven cell proliferation Epstein-Barr virus-encoded LMP2A regulates viral and cellular gene expression by modulation of the NF-kappaB transcription factor pathway.Induction of Epstein-Barr virus latent membrane protein 1 by a lytic transactivator Rta.The role of STAT5 in the development, function, and transformation of B and T lymphocytes.Epstein-barr virus and the pathogenesis of T and NK lymphoma: a mystery unsolved.Switching of EBV cycles between latent and lytic states.Regulation of Epstein-Barr virus reactivation from latency.Nuclear factor-kappaB binds to the Epstein-Barr Virus LMP1 promoter and upregulates its expression Interleukin-6-dependent growth in a newly established plasmablastic lymphoma cell line and its therapeutic targets Epstein-Barr virus latent membrane protein 1 induces expression of the epidermal growth factor receptor through effects on Bcl-3 and STAT3.A neutralized human LMP1-IgG inhibits ENKTL growth by suppressing the JAK3/STAT3 signaling pathway.Tofacitinib induces G1 cell-cycle arrest and inhibits tumor growth in Epstein-Barr virus-associated T and natural killer cell lymphoma cells High frequency of latent membrane protein-1 30-bp deletion variant with specific single mutations in Epstein-Barr virus-associated nasopharyngeal carcinoma in Moroccan patients.LPLUNC1 suppresses IL-6-induced nasopharyngeal carcinoma cell proliferation via inhibiting the Stat3 activation.Epstein-Barr virus LMP-1 natural sequence variants differ in their potential to activate cellular signaling pathways.Epstein-Barr virus regulates STAT1 through latent membrane protein 1.

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P2860

Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

http://www.wikidata.org/entity/Q33866046

description

2001 nî lūn-bûn

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2001 թուականի Մարտին հրատարակուած գիտական յօդուած

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2001 թվականի մարտին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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JVI.75.6.2929-2937.2001

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Journal of Virology

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Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

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P2093

H Chen

http://www.w3.org/2001/XMLSchema#string

J M Lee

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M Borowitz

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M H Ng

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R F Ambinder

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S D Hayward

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Y Zong

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P2860

STAT3beta, a splice variant of transcription factor STAT3, is a dominant negative regulator of transcription

The Epstein-Barr virus oncoprotein latent membrane protein 1 engages the tumor necrosis factor receptor-associated proteins TRADD and receptor-interacting protein (RIP) but does not induce apoptosis or require RIP for NF-kappaB activation

The Epstein-Barr virus oncogene product latent membrane protein 1 engages the tumor necrosis factor receptor-associated death domain protein to mediate B lymphocyte growth transformation and activate NF-kappaB

Control of inflammation, cytokine expression, and germinal center formation by BCL-6

STATs and gene regulation

Stat3 as an oncogene

STATs in oncogenesis

Transcriptional responses to polypeptide ligands: the JAK-STAT pathway

Abrogation of interleukin-3 dependence of myeloid cells by the v-src oncogene requires SH2 and SH3 domains which specify activation of STATs

TEL-JAK2 transgenic mice develop T-cell leukemia.

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2929-2937

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11222718

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Epstein–Barr virus

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