PRL-3 promotes telomere deprotection and chromosomal instability.
about
Dysfunctional telomeres and hematological disorders.A loss-of-function genetic screening reveals synergistic targeting of AKT/mTOR and WTN/β-catenin pathways for treatment of AML with high PRL-3 phosphatase.Knockdown of PRL-3 increases mitochondrial superoxide anion production through transcriptional regulation of RAP1
P2860
PRL-3 promotes telomere deprotection and chromosomal instability.
description
2017 nî lūn-bûn
@nan
2017 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2017 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
name
PRL-3 promotes telomere deprotection and chromosomal instability.
@ast
PRL-3 promotes telomere deprotection and chromosomal instability.
@en
type
label
PRL-3 promotes telomere deprotection and chromosomal instability.
@ast
PRL-3 promotes telomere deprotection and chromosomal instability.
@en
prefLabel
PRL-3 promotes telomere deprotection and chromosomal instability.
@ast
PRL-3 promotes telomere deprotection and chromosomal instability.
@en
P2093
P2860
P356
P1476
PRL-3 promotes telomere deprotection and chromosomal instability.
@en
P2093
Caiyun Liu
Chengchao Shou
Shenyi Lian
Xiaofang Xing
Yongyong Yang
P2860
P356
10.1093/NAR/GKX392
P407
P577
2017-05-08T00:00:00Z