Decreased NR1, NR2A, and SAP102 transcript expression in the hippocampus in bipolar disorder.
about
Replication of ketamine's antidepressant efficacy in bipolar depression: a randomized controlled add-on trialA randomized add-on trial of an N-methyl-D-aspartate antagonist in treatment-resistant bipolar depressionPostmortem brain: an underutilized substrate for studying severe mental illnessCross-diagnostic comparison of duration mismatch negativity and P3a in bipolar disorder and schizophrenia.Acute D-serine treatment produces antidepressant-like effects in rodentsAdvancing biomarker research: utilizing 'Big Data' approaches for the characterization and prevention of bipolar disorder.Bimodal effect of lithium plasma levels on hippocampal glutamate concentrations in bipolar II depression: a pilot study.Increased excitotoxicity and neuroinflammatory markers in postmortem frontal cortex from bipolar disorder patients.N-methyl-D-aspartate receptor expression in parvalbumin-containing inhibitory neurons in the prefrontal cortex in bipolar disorderBrain glutamatergic characteristics of pediatric offspring of parents with bipolar disorder.Glutamatergic gene expression is specifically reduced in thalamocortical projecting relay neurons in schizophrenia.Integrated neurobiology of bipolar disorder.Magnetic resonance spectroscopy studies of glutamate-related abnormalities in mood disordersAltered expression of synapse and glutamate related genes in post-mortem hippocampus of depressed subjectsGluN2B protein deficits in the left, but not the right, hippocampus in schizophrenia.Brain glutamate levels measured by magnetic resonance spectroscopy in patients with bipolar disorder: a meta-analysis.Glutamatergic modulators: the future of treating mood disorders?Differential roles of hippocampal glutamatergic receptors in neuropathic anxiety-like behavior after partial sciatic nerve ligation in rats.Lamotrigine blocks NMDA receptor-initiated arachidonic acid signalling in rat brain: implications for its efficacy in bipolar disorderThe N-methyl-D-aspartate receptor as a neurobiological intersection between bipolar disorder and alcohol use: a longitudinal mismatch negativity studyNovel glutamatergic agents for major depressive disorder and bipolar disorder.Expression profiling in neuropsychiatric disorders: emphasis on glutamate receptors in bipolar disorderSynaptic proteins in the hippocampus indicative of increased neuronal activity in CA3 in schizophrenia.Imaging brain signal transduction and metabolism via arachidonic and docosahexaenoic acid in animals and humans.Transcription factor SP4 phosphorylation is altered in the postmortem cerebellum of bipolar disorder and schizophrenia subjectsChronic carbamazepine administration reduces N-methyl-D-aspartate receptor-initiated signaling via arachidonic acid in rat brainOverview of Glutamatergic Dysregulation in Central Pathologies.NMDA hypofunction as a convergence point for progression and symptoms of schizophreniaCholinergic connectivity: it's implications for psychiatric disorders.Chronic administration of valproic acid reduces brain NMDA signaling via arachidonic acid in unanesthetized rats.Neurotransmission and bipolar disorder: a systematic family-based association study.Decreased mRNA expression of netrin-G1 and netrin-G2 in the temporal lobe in schizophrenia and bipolar disorder.Molecular evidence of synaptic pathology in the CA1 region in schizophrenia.Targeting the glutamatergic system to develop novel, improved therapeutics for mood disordersThe role of the tripartite glutamatergic synapse in the pathophysiology and therapeutics of mood disorders.Targeting glutamatergic signaling for the development of novel therapeutics for mood disordersChronic carbamazepine administration attenuates dopamine D2-like receptor-initiated signaling via arachidonic acid in rat brain.Neural correlates of rapid antidepressant response to ketamine in bipolar disorder.Depression and treatment response: dynamic interplay of signaling pathways and altered neural processes.Neuropathology of mood disorders: do we see the stigmata of inflammation?
P2860
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P2860
Decreased NR1, NR2A, and SAP102 transcript expression in the hippocampus in bipolar disorder.
description
2006 nî lūn-bûn
@nan
2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
name
Decreased NR1, NR2A, and SAP10 ...... ppocampus in bipolar disorder.
@ast
Decreased NR1, NR2A, and SAP10 ...... ppocampus in bipolar disorder.
@en
type
label
Decreased NR1, NR2A, and SAP10 ...... ppocampus in bipolar disorder.
@ast
Decreased NR1, NR2A, and SAP10 ...... ppocampus in bipolar disorder.
@en
prefLabel
Decreased NR1, NR2A, and SAP10 ...... ppocampus in bipolar disorder.
@ast
Decreased NR1, NR2A, and SAP10 ...... ppocampus in bipolar disorder.
@en
P2093
P2860
P1433
P1476
Decreased NR1, NR2A, and SAP10 ...... ppocampus in bipolar disorder.
@en
P2093
Brian Dean
Elizabeth Scarr
James H Meador-Woodruff
Lars V Kristiansen
Monica Beneyto
Robert E McCullumsmith
P2860
P304
P356
10.1016/J.BRAINRES.2006.09.011
P407
P577
2006-11-17T00:00:00Z