Cellular senescence and the senescent secretory phenotype: therapeutic opportunities. - Test2 - elhamkhani - TriplyDB

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

http://www.wikidata.org/entity/Q34035882

about

Multiple facets of p53 in senescence induction and maintenance DNA Damage: A Main Determinant of Vascular Aging "Inflammaging" as a Druggable Target: A Senescence-Associated Secretory Phenotype-Centered View of Type 2 Diabetes Finding Ponce de Leon's Pill: Challenges in Screening for Anti-Aging Molecules Molecular mechanisms of biological aging in intervertebral discs Cellular senescence impact on immune cell fate and function Cellular Senescence and the Biology of Aging, Disease, and Frailty Pathogenesis of endothelial cell dysfunction in chronic kidney disease: a retrospective and what the future may hold DNA damage response (DDR) and senescence: shuttled inflamma-miRNAs on the stage of inflamm-aging Parainflammation, chronic inflammation, and age-related macular degeneration Stromal Fibroblast in Age-Related Cancer: Role in Tumorigenesis and Potential as Novel Therapeutic Target Do telomeres adapt to physiological stress? Exploring the effect of exercise on telomere length and telomere-related proteins Macroautophagy and the oncogene-induced senescence Immune surveillance of unhealthy cells by natural killer cells Primary biliary cirrhosis is a generalized autoimmune epithelitis Therapeutic targeting of replicative immortality Dynamic modelling of pathways to cellular senescence reveals strategies for targeted interventions Extracellular microRNAs and endothelial hyperglycaemic memory: a therapeutic opportunity?Aging and adipose tissue: potential interventions for diabetes and regenerative medicine Short Telomeres in Key Tissues Initiate Local and Systemic Aging in Zebrafish Phosphofructokinase deficiency impairs ATP generation, autophagy, and redox balance in rheumatoid arthritis T cells Defining the toxicology of aging Rapamycin induces pluripotent genes associated with avoidance of replicative senescence Aging of mice is associated with p16(Ink4a)- and β-galactosidase-positive macrophage accumulation that can be induced in young mice by senescent cells Contact inhibition and high cell density deactivate the mammalian target of rapamycin pathway, thus suppressing the senescence program Oxidative DNA damage caused by inflammation may link to stress-induced non-targeted effects.Loss of Arp2/3 induces an NF-κB-dependent, nonautonomous effect on chemotactic signaling Age-associated defects in EphA2 signaling impair the migration of human cardiac progenitor cells.Primary sclerosing cholangitis and the microbiota: current knowledge and perspectives on etiopathogenesis and emerging therapies Senescence associated macrophages and "macroph-aging": are they pieces of the same puzzle?Developments in our understanding of the effects of growth hormone on white adipose tissue from mice: implications to the clinic.Identification of senescence-associated circular RNAs (SAC-RNAs) reveals senescence suppressor CircPVT1 Old Maids: Aging and Its Impact on Microglia Function.Expansion of myeloid-derived suppressor cells with aging in the bone marrow of mice through a NF-κB-dependent mechanism.Ionizing radiation reduces ADAM10 expression in brain microvascular endothelial cells undergoing stress-induced senescence.Long-lived crowded-litter mice exhibit lasting effects on insulin sensitivity and energy homeostasis.Senescent tumor cells lead the collective invasion in thyroid cancer.Revisiting the hallmarks of cancer.Molecular and phenotypic biomarkers of aging p53 Modulates the Fate of Cardiac Progenitor Cells Ex Vivo and in the Diabetic Heart In Vivo.

P2860

Q26738906-635F680A-C319-4903-B183-3ABEAD1A4DC4 Q26746962-438D6DBB-E7B9-4538-81D5-BDD6D5240A7D Q26747251-22CC8200-99D6-483B-9838-02B79F4FBE32 Q26747742-1BB421E0-4B91-46B7-BD59-C441566D08C7 Q26764941-35ED167E-ECDE-4F00-A0E7-C1A0891E2749 Q26766576-6DB50CC4-C3B7-4D14-9A47-6E0E42F9D73A Q26781231-6EF2A00D-AE2F-473F-977E-56E7796EF944 Q26781354-81138E45-4AE9-406C-8784-5C553AE8531C Q26782606-19C90EED-7114-48DE-B344-C3329ABCB5D9 Q26796268-116211D2-6CDB-4FE4-8BE7-501B4DAE7474 Q26797288-9CB6F261-476F-4679-9D74-017C5C098AF2 Q26853657-56C9A9AE-3862-4AFC-A36C-80872EF66F9E Q26863440-E09FC66D-CAA0-4BDF-95A6-7696751ABA61 Q27003079-4AA7D747-F707-4EC8-95B9-49D82F17DC7D Q27016114-185D6D84-F8C9-4507-BC0C-BB5CA416FABE Q27027457-19C26ACB-E483-45B7-A3C3-21D511EC6F8C Q27321555-68BC91A4-1054-4A33-A682-B39325061A36 Q28079163-57CE17E4-A325-4049-89D9-406E5282C727 Q28079178-DE7EFDA1-0515-4805-BF33-122E40FCC269 Q28272305-F580C334-3F43-4483-A643-B182B0DCEEA7 Q28298650-27F86008-2FE6-4EC0-AB19-15E566435778 Q28389491-A3D63590-030A-4648-AE70-BDD54435578B Q28572137-EDE0058B-FC92-41A0-ADCC-71F0390C2C14 Q28972229-90E66E21-851D-494A-BF68-9F51F47A73AB Q28972583-05D34EB7-4B10-4308-A517-E1F69355F9EA Q30394633-EF7702F6-2467-463F-B864-0EB98A659E6A Q30560511-AD11AE45-A60C-454B-AB25-EFEDB3AD6C8B Q30564050-6066079B-A138-485F-99BF-9D547F2CC0C7 Q31170968-BEE1DCA8-9FDF-43BA-8FCC-46CCBD1AE06C Q33364422-0BFE90CD-8A33-4A0E-A1CB-37C9CE6763C0 Q33580161-AFF48D0F-1E02-4156-A9CE-67F2928F7DB3 Q33580276-8A8C9506-5E7D-42CF-8805-912730892E91 Q33624540-DE925BCF-4839-492A-A865-4836FD096A11 Q33638965-A1453E2E-751B-419D-9F92-78C7CDBF621C Q33657479-18C3DCF1-E207-40C8-8798-431F8B5B6DEA Q33701511-439BF2F7-3E81-4E49-87AF-66618F44C85F Q33702243-C02A6EDE-CFE1-48E7-874A-5101087EC48A Q33732986-534DA9AC-F1DE-4F0B-872C-923A689FE57D Q33806547-8BBB3E45-3EDF-4578-A95A-3FA54C132F5F Q33809750-6310C4C5-15F6-4323-A04C-A383DF891D9D

P2860

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

http://www.wikidata.org/entity/Q34035882

description

2013 nî lūn-bûn

@nan

2013 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի մարտին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@ast

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@en

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@nl

type

label

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@ast

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@en

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@nl

prefLabel

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@ast

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@en

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@nl

P1433

Q34035882-56F97C92-4475-4D63-985D-761916ADD12A

P1476

Q34035882-A812DE59-571C-44D2-A98F-3E5D3BA815CC

P2093

Q34035882-04A23DCC-4B80-4B8A-84A0-719A181B1E1D

Q34035882-44939CFB-BFB6-4558-BBA2-0E8495180398

Q34035882-67CFF687-A285-4DAE-A1A4-0ADA013047E1

Q34035882-76749AA1-CA35-4697-A440-EE1D362190E5

Q34035882-969D8C6F-068C-4970-82FE-8A833B8EC332

P2860

Q34035882-03422587-B720-4EF0-A000-43C3C2076272

Q34035882-03F7A5F0-0DC7-48E0-BED3-D1241A543E30

Q34035882-04C13134-FA1B-4BB7-8AD1-E432683BD498

Q34035882-04D28485-12B9-45A5-A372-16653791A064

Q34035882-05BAF184-BC56-49BC-A0E9-56C99DEC0A1E

Q34035882-06A78A64-8781-42E1-B814-088AA73D6A3D

Q34035882-09271962-DBAD-41FC-9164-B33F98AE5F65

Q34035882-16689423-979F-46DF-9891-72791F7EF4F9

Q34035882-16EA2FBF-C718-407F-B75A-6965373AAE14

Q34035882-1790112C-73B0-4F84-8924-C5B8ED8D9722

P304

Q34035882-4A655410-73B4-452C-833D-720F6B6648E2

P31

Q34035882-CA3C0241-EAEB-4B23-B876-BE69DCF722D5

P356

Q34035882-C1B06C88-5198-40CA-A8DC-2A1FE9F6CF3A

P407

Q34035882-92332BAF-31D3-42FF-AB89-F25ECAB2323B

P433

Q34035882-C6C85235-AF72-49A6-A58A-874EB3BDCDC1

P478

Q34035882-B80F6079-2A42-4D2F-A49A-43A8C766AFE4

P577

Q34035882-47252581-F543-4130-8B82-2C380417D65A

P698

Q34035882-82EF357E-E8E8-4802-B1ED-17DDA820DABC

P932

Q34035882-7605A6D9-80CB-4EA9-91B3-FD5F4EA37CE7

P356

P1433

Journal of Clinical Investigation

P1476

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities.

@en

P2093

James L Kirkland

http://www.w3.org/2001/XMLSchema#string

Jan van Deursen

http://www.w3.org/2001/XMLSchema#string

Judith Campisi

http://www.w3.org/2001/XMLSchema#string

Tamara Tchkonia

http://www.w3.org/2001/XMLSchema#string

Yi Zhu

http://www.w3.org/2001/XMLSchema#string

P2860

Oncogene-Induced Senescence: Putting the Brakes on Tumor Development

Feedback between p21 and reactive oxygen production is necessary for cell senescence

The DNA damage pathway regulates innate immune system ligands of the NKG2D receptor

Cellular senescence controls fibrosis in wound healing

Senescence of activated stellate cells limits liver fibrosis

The matricellular protein CCN1 induces fibroblast senescence and restricts fibrosis in cutaneous wound healing

Nonlinear multisystem physiological dysregulation associated with frailty in older women: implications for etiology and treatment

MicroRNAs miR-146a/b negatively modulate the senescence-associated inflammatory mediators IL-6 and IL-8

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

Frailty in older adults: evidence for a phenotype

P304

966-972

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1172/JCI64098

http://www.w3.org/2001/XMLSchema#string

P407

P433

3

http://www.w3.org/2001/XMLSchema#string

P478

123

http://www.w3.org/2001/XMLSchema#string

P577

2013-03-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

23454759

http://www.w3.org/2001/XMLSchema#string

P932

3582125

http://www.w3.org/2001/XMLSchema#string