Checkpoint signaling, base excision repair, and PARP promote survival of colon cancer cells treated with 5-fluorodeoxyuridine but not 5-fluorouracil.

Checkpoint signaling, base excision repair, and PARP promote survival of colon cancer cells treated with 5-fluorodeoxyuridine but not 5-fluorouracil.

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http://www.wikidata.org/entity/Q34110442
The cancer therapeutic potential of Chk1 inhibitors: how mechanistic studies impact on clinical trial designMomordica charantia Extract Induces Apoptosis in Human Cancer Cells through Caspase- and Mitochondria-Dependent Pathways.Poly (ADP-ribose) polymerase inhibitors: recent advances and future development.Identification of DNA repair pathways that affect the survival of ovarian cancer cells treated with a poly(ADP-ribose) polymerase inhibitor in a novel drug combination.Genomically Incorporated 5-Fluorouracil that Escapes UNG-Initiated Base Excision Repair Blocks DNA Replication and Activates Homologous RecombinationTranscriptome profiling identifies genes and pathways deregulated upon floxuridine treatment in colorectal cancer cells harboring GOF mutant p53.Tumor site- and stage-specific associations between allelic variants of glutathione S-transferase and DNA-repair genes and overall survival in colorectal cancer patients receiving 5-fluorouracil-based chemotherapyHuman apurinic/apyrimidinic endonuclease 1.Standing the test of time: targeting thymidylate biosynthesis in cancer therapy.Methoxyamine Enhances 5-Fluorouracil-Induced Radiosensitization in Colon Cancer Cell Line HT29.Glycogen Synthase Kinase 3 (GSK-3)-mediated Phosphorylation of Uracil N-Glycosylase 2 (UNG2) Facilitates the Repair of Floxuridine-induced DNA Lesions and Promotes Cell Survival.PARP inhibitor attenuated colony formation can be restored by MAP kinase inhibitors in different irradiated cancer cell lines.Dietary supplementation with omega-3 fatty acid attenuates 5-fluorouracil induced mucositis in miceBase excision repair imbalance in colorectal cancer has prognostic value and modulates response to chemotherapySelective depletion of metastatic stem cells as therapy for human colorectal cancer
P2860

Checkpoint signaling, base excision repair, and PARP promote survival of colon cancer cells treated with 5-fluorodeoxyuridine but not 5-fluorouracil.

Item
http://www.wikidata.org/entity/Q34110442
description
2011 nî lūn-bûn
@nan
2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@ast
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@en
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@nl
type
label
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@ast
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@en
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@nl
prefLabel
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@ast
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@en
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P932
P356
P1433
P1476
Checkpoint signaling, base exc ...... ridine but not 5-fluorouracil.
@en
P2093
P2860
P304
P31
P356
10.1371/JOURNAL.PONE.0028862
P407
P433
P478
P577
P5875
P698
P932