The different functions of Stat5 and chromatin alteration through Stat5 proteins
about
Epigenetic repression of the Igk locus by STAT5-mediated recruitment of the histone methyltransferase Ezh2Oncoproteomic analysis reveals co-upregulation of RELA and STAT5 in carboplatin resistant ovarian carcinomaHow Intrinsic Molecular Dynamics Control Intramolecular Communication in Signal Transducers and Activators of Transcription Factor STAT5Stat5a serine 725 and 779 phosphorylation is a prerequisite for hematopoietic transformationAlternative ways of modulating JAK-STAT pathway: Looking beyond phosphorylation.Epidermal growth factor receptor signaling synergizes with Hedgehog/GLI in oncogenic transformation via activation of the MEK/ERK/JUN pathway.Altered gene expression patterns during the initiation and promotion stages of neonatally diethylstilbestrol-induced hyperplasia/dysplasia/neoplasia in the hamster uterus.Essential role for Stat5a/b in myeloproliferative neoplasms induced by BCR-ABL1 and JAK2(V617F) in mice.Hepatic growth hormone and glucocorticoid receptor signaling in body growth, steatosis and metabolic liver cancer development.GH/STAT5 signaling during the growth period in livers of mice overexpressing GH.The signal transducers Stat1 and Stat3 and their novel target Jmjd3 drive the expression of inflammatory genes in microglia.An Unbiased Linkage Approach Reveals That the p53 Pathway Is Coupled to NK Cell Maturation.O-GlcNAcylation of STAT5 controls tyrosine phosphorylation and oncogenic transcription in STAT5-dependent malignancies.STAT5 N-domain deleted isoforms are naturally occurring hypomorphs partially rescued in hematopoiesis by transgenic Bcl-2 expression.Stat5 is indispensable for the maintenance of bcr/abl-positive leukaemia.The inhibition of stat5 by a Peptide aptamer ligand specific for the DNA binding domain prevents target gene transactivation and the growth of breast and prostate tumor cellsStat5 Exerts Distinct, Vital Functions in the Cytoplasm and Nucleus of Bcr-Abl+ K562 and Jak2(V617F)+ HEL Leukemia Cells.Malignant Phenotypes in Metastatic Melanoma are Governed by SR-BI and its Association with Glycosylation and STAT5 Activation.Differential signaling through p190 and p210 BCR-ABL fusion proteins revealed by interactome and phosphoproteome analysis.Control of CD8 T cell proliferation and terminal differentiation by active STAT5 and CDKN2A/CDKN2B.
P2860
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P2860
The different functions of Stat5 and chromatin alteration through Stat5 proteins
description
2008 nî lūn-bûn
@nan
2008 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@ast
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@en
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@nl
type
label
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@ast
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@en
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@nl
prefLabel
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@ast
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@en
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@nl
P2093
P2860
P50
P356
P1476
The different functions of Stat5 and chromatin alteration through Stat5 proteins
@en
P2093
Andrea Hoelbl
Barbara Zankl
Ernst W Mullner
Fabrice Gouilleux
Harini Nivarti
Hartmut Beug
Jan-Wilhelm Kornfeld
Katrin Friedbichler
Marc A Kerenyi
Mathias Muller
P2860
P304
P356
10.2741/3151
P577
2008-05-01T00:00:00Z