Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
about
Engineering the transmissible gastroenteritis virus genome as an expression vector inducing lactogenic immunityThe role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesisRewiring the severe acute respiratory syndrome coronavirus (SARS-CoV) transcription circuit: engineering a recombination-resistant genomeCoronavirus virulence genes with main focus on SARS-CoV envelope geneCoronavirus reverse genetic systems: infectious clones and repliconsGeneration of a replication-competent, propagation-deficient virus vector based on the transmissible gastroenteritis coronavirus genome.Structure and inhibition of the SARS coronavirus envelope protein ion channelStructure of a conserved Golgi complex-targeting signal in coronavirus envelope proteinsThe M, E, and N structural proteins of the severe acute respiratory syndrome coronavirus are required for efficient assembly, trafficking, and release of virus-like particles.Coronavirus replicase-reporter fusions provide quantitative analysis of replication and replication complex formation.Coronavirus non-structural protein 1 is a major pathogenicity factor: implications for the rational design of coronavirus vaccines.The replicase gene of avian coronavirus infectious bronchitis virus is a determinant of pathogenicityRole of nucleotides immediately flanking the transcription-regulating sequence core in coronavirus subgenomic mRNA synthesisPorcine epidemic diarrhoea virus: a comprehensive review of molecular epidemiology, diagnosis, and vaccines.Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis.A major determinant for membrane protein interaction localizes to the carboxy-terminal domain of the mouse coronavirus nucleocapsid protein.Severe acute respiratory syndrome coronavirus group-specific open reading frames encode nonessential functions for replication in cell cultures and mice.Reverse genetic analysis of the transcription regulatory sequence of the coronavirus transmissible gastroenteritis virus.Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.Severe acute respiratory syndrome coronavirus infection of human ciliated airway epithelia: role of ciliated cells in viral spread in the conducting airways of the lungs.Systematic assembly of a full-length infectious cDNA of mouse hepatitis virus strain A59.Proteome profile of swine testicular cells infected with porcine transmissible gastroenteritis coronavirus.Evolved variants of the membrane protein can partially replace the envelope protein in murine coronavirus assembly.Identification of functionally important negatively charged residues in the carboxy end of mouse hepatitis coronavirus A59 nucleocapsid protein.The small envelope protein E is not essential for murine coronavirus replication.A severe acute respiratory syndrome coronavirus that lacks the E gene is attenuated in vitro and in vivo.Exceptional flexibility in the sequence requirements for coronavirus small envelope protein functionRole of the coronavirus E viroporin protein transmembrane domain in virus assembly.Coronavirus envelope (E) protein remains at the site of assemblyGenome organization and reverse genetic analysis of a type I feline coronavirusEnvelope protein palmitoylations are crucial for murine coronavirus assembly.Importance of conserved cysteine residues in the coronavirus envelope protein.SARS-CoV replication and pathogenesis in an in vitro model of the human conducting airway epitheliumReverse genetics with a full-length infectious cDNA of severe acute respiratory syndrome coronavirus.Vaccines to prevent severe acute respiratory syndrome coronavirus-induced disease.SARS coronavirus accessory proteins.Detection and phylogenetic analysis of porcine epidemic diarrhea virus in central China based on the ORF3 gene and the S1 gene.Genetic analysis of determinants for spike glycoprotein assembly into murine coronavirus virions: distinct roles for charge-rich and cysteine-rich regions of the endodomain.Inhibition of NF-κB-mediated inflammation in severe acute respiratory syndrome coronavirus-infected mice increases survivalThe coronavirus E protein: assembly and beyond.
P2860
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P2860
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
description
2002 nî lūn-bûn
@nan
2002 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@ast
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@en
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@nl
type
label
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@ast
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@en
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@nl
prefLabel
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@ast
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@en
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@nl
P2860
P1433
P1476
Heterologous gene expression from transmissible gastroenteritis virus replicon particles.
@en
P2093
Boyd Yount
Kristopher M Curtis
P2860
P304
P356
10.1128/JVI.76.3.1422-1434.2002
P407
P577
2002-02-01T00:00:00Z